I`m going to do do a cycle consisting of Trenbolone acetate 300mg/w and Test enh. 500mg/w.
Here on the forum most people say that you should continue the test a couple of weeks afterwards, but i thought the acetate ester left the body rather quick?
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I`m going to do do a cycle consisting of Trenbolone acetate 300mg/w and Test enh. 500mg/w.
Here on the forum most people say that you should continue the test a couple of weeks afterwards, but i thought the acetate ester left the body rather quick?
It's does clear quickly.The theory is it aid in recovery by running Test a few weeks past the Fina.Quote:
Originally Posted by Asgard
It's really nice to still be running something after dropping the tren ace. Your body will not like loosing that hormone, keeping the test in the cycle is kinda like a compromise.
I'd bump the tren to at least 50mg ed...Are you shooting ED? If not you should be. I've experienced less sides from tren when running it ED as opposed to EOD. Also, if the sides aren't too much, you might want to bump it to 75mg ED, but you'd have to recalculate your test dosage because you'd be shooting 525mg of tren and only 500mg of test. I'd go with 600mg test and 525mg tren, but that's me.Quote:
Originally Posted by Asgard
sweet...Quote:
Originally Posted by ODC0717
Trenbolone leaves some very potent and suppressive metabolites behind for a few weeks.
big k....you have any links for that?I'd be interested in reading them,since I haven't really read/seen much on Trenbolone.Quote:
Originally Posted by big k.l.g
Yeh, but the tren a ester is the lightest in this respect.Quote:
Originally Posted by big k.l.g
>Ace<
I'll look to see if a have the studies saved from the time i wrote the tren ace profile. A lot of us don't realize how sick tren is, hell even one of tren's metabolites is as anabolic as test and binds better to the PgR than progesterone itself.Quote:
Originally Posted by Pinnacle
wait a stinkn minute here, the profiles were written by hooker...umm...wait are you hooker aka A.R. ? am I stupid?Quote:
Originally Posted by big k.l.g
No...he had profiles up before Hooker wrote/posted all his.Quote:
Originally Posted by smiler
Nah you're just stupid :1laugh:Quote:
Originally Posted by smiler
:D kidding......... I have 6 profiles in that section, due to the fact that people can't tell the diff between mine and Hooker’s is a huge complement, well, kinda.
ok, I'm stupid...nice job though, I've read most of emQuote:
Originally Posted by big k.l.g
Actually, that isn't so.Quote:
Originally Posted by Pinnacle
His profiles are still up.
Hooker as Steroid.com Writer/Editor edited 'em.
Under Contract they are the property of Steroid.com
"lightest"?Quote:
Originally Posted by MastaAce
The metabolite is the metabolite of the Hormone... not the ester.
Thus...How can the Tren-A's metabolites be 'lighter'?
The metabolites will be the same.
aye maybe he means it LEAVES 11days sooner than trenE?Quote:
Originally Posted by Narkissos
I knew Tony edited some and gave credit to big k for the articles.Quote:
Originally Posted by Narkissos
But I could have sworn that I saw big k's name on some BEFORE the profile forum made a dramatic change.But what the hell do I know...lol...
:DQuote:
Originally Posted by Pinnacle
Both counts are correct.
His name is on some posted prior to them being assimilated into the profile forum.
Scroll down to the bottom of the page in this thread and you'll see a quick link to 'related threads'
The Tren profile is there :thumbsup:
LOL! I noticed that as well. The profiles are the same though.Quote:
Originally Posted by Pinnacle
i would do more tren and run it ed
ok, ok alright already I said I'm stupidQuote:
Originally Posted by big k.l.g
Got it spot on brutha! Perhaps lightest wasn't the best word, but you knew what I meant nark!Quote:
Originally Posted by taiboxa
Relax man :aaGreen22 We were not even talking about what you said!Quote:
Originally Posted by smiler
I know bro, I was kiddingQuote:
Originally Posted by big k.l.g
Effects of the androgenic growth promoter 17-beta-trenbolone on fecundity and reproductive endocrinology of the fathead minnow.
Ankley GT, Jensen KM, Makynen EA, Kahl MD, Korte JJ, Hornung MW, Henry TR, Denny JS, Leino RL, Wilson VS, Cardon MC, Hartig PC, Gray LE.
U.S. Environmental Protection Agency, National Health and Environmental Effects Research Laboratory, Mid-Continent Ecology Division, 6201 Congdon Boulevard, Duluth, Minnesota 55804, USA. [email protected]
Trenbolone acetate is a synthetic steroid that is extensively used in the United States as a growth promoter in beef cattle. The acetate is administered to livestock via slow-release implants; some is converted by the animal to 17-beta-trenbolone, a relatively potent androgen receptor agonist in mammalian systems. Recent studies indicate that excreted 17-beta-trenbolone is comparatively stable in animal waste, suggesting the potential for exposure to aquatic animals via direct discharge, runoff, or both. However, little is known concerning the toxicity of trenbolone to fish. Our goal was to assess the effects of 17-beta-trenbolone on reproductive endocrinology of the fathead minnow (Pimephales promelas). An in vitro competitive binding study with the fathead minnow androgen receptor demonstrated that 17-beta-trenbolone had a higher affinity for the receptor than that of the endogenous ligand, testosterone. Male and female fish were exposed for 21 d to nominal (target) concentrations of 17-beta-trenbolone ranging from 0.005 to 50 microg/L. Fecundity of the fish was significantly reduced by exposure to measured test concentrations > or = 0.027 microg/ L. The 17-beta-trenbolone was clearly androgenic in vivo at these concentrations, as evidenced by the de novo production in females of dorsal (nuptial) tubercles, structures normally present only on the heads of mature males. Plasma steroid (testosterone and beta-estradiol) and vitellogenin concentrations in the females all were significantly reduced by exposure to 17-beta-trenbolone. The 17-beta-trenbolone also altered reproductive physiology of male fathead minnows, albeit at concentrations much higher than those producing effects in females. Males exposed to 17-beta-trenbolone at 41 microg/L (measured) exhibited decreased plasma concentrations of 11-ketotestosterone and increased concentrations of beta-estradiol and vitellogenin. Overall, our studies indicate that 17-beta-trenbolone is a potent androgen and reproductive toxicant in fish. Given the widespread use of trenbolone acetate as a growth promoter, and relative stability of its metabolites in animal wastes, further studies are warranted to assess potential ecological risk.
PMID: 12785594 [PubMed - indexed for MEDLINE]