Results 1 to 5 of 5

Thread: Blood Pressure

  1. #1
    madmanz is offline Junior Member
    Join Date
    Feb 2003
    Location
    clearwater fla
    Posts
    94

    Blood Pressure

    although docotr visit are important i am curious how much your blood pressure goes up during a cycle. mine was 124/74 before and went as high as 155/76 during mine due to the increase in water retention. how high is it ok to go up to before you would go off a cycle

  2. #2
    sweet's Avatar
    sweet is offline Associate Member
    Join Date
    Aug 2002
    Location
    Ottawa, Ontario
    Posts
    341
    High blood pressure is not really a problem in the short term. It becomes a problem when it is high (140/90 or above) over an extended period of time, like 10 years. There is a slight chance that you could have a stroke if your systolic pressure gets above 180, but like I said it is only slight. 155/76 is a little high but nothing to worry about.

  3. #3
    m16a2 is offline Senior Member
    Join Date
    Aug 2002
    Location
    LA
    Posts
    1,523
    My BP got up to 170 during my cycle. I doubt its due to water retention though. Do a search for blood pressure, tons and tons of bros have listed their BP increases, including myself on multiple threads.

    Right now, I'm around 140

  4. #4
    StoneColdNTO is offline New Member
    Join Date
    May 2002
    Location
    Canada
    Posts
    4
    Originally posted by sweet
    High blood pressure is not really a problem in the short term. It becomes a problem when it is high (140/90 or above) over an extended period of time, like 10 years.
    I don't usually post here, but when I see misinformation like this, I have to say something;

    High blood pressure is the main cause of heart enlargment of the left ventricle. It can happen very quickly, in a matter of months, trust me on this one, I am living proof.

  5. #5
    StoneColdNTO is offline New Member
    Join Date
    May 2002
    Location
    Canada
    Posts
    4
    Changes to Heart Muscle

    The changes to heart muscle caused by anabolic steroids are attributed to their anabolic properties in muscle tissue. Left ventricular hypertrophy is characterized by thickening of the left ventricular wall secondary to cardiac fiber enlargement. Left ventricular hypertrophy (LVH) is normally caused by a chronic increase in systemic blood pressure. It may also be seen with sudden or rapid weight gain. The thickening of the ventricular wall due to increased after-load from elevated vascular resistance can be viewed as adaptive protection up to a point. Beyond minor wall thickening, pathological LVH is a strong predictor of serious cardiovascular risk.

    It is important for physicians to realize that LVH can occur in strength athletes and bodybuilders even in the absence of anabolic steroids use. It was previously believed that the intermittent increase in blood pressure that is caused by heavy lifting was not sufficient to elicit concentric left ventricular hypertrophy (CLVH). Any evidence of CLVH in strength athletes or bodybuilders was seen as a sign of anabolic steroid use .

    During heavy lifting, systemic blood pressure is increased from what is called the valsalva maneuver. It is simply the act of forceful expiration with the mouth and nose closed producing a "bearing down" on the abdomen. Most people do this during heavy lifts such as squats or deadlifts. Pressure also increases due to blood vessels being occluded by contracting muscles. It should be noted that the LVH seen in bodybuilders and power lifters is called "concentric left ventricular hypertrophy", meaning that it is the result of contracting against acute increased systemic pressure, and is not considered pathological (i.e. unhealthy). "Eccentric" LVH is caused by constant increases of blood pressure, not as a result of the valsalva maneuver but instead due to clinical hypertension that forces the ventricle to expand against resistance.

    AAS further exacerbate the effects of lifting on the heart. AASs cause anabolism in heart muscle, at times increasing left ventricular wall thickness to 16mm (11mm is considered normal).4 However, LVH caused by resistance training either alone or in conjunction with AAS has yet to result in diastolic dysfunction, or in other words, there is yet no evidence that this thickening of the ventricular wall is pathologic.

    Treatment options

    Upon cessation of high intensity resistance exercise and obviously AAS use, ventricular wall thickness returns to within normal ranges as long as hypertension unrelated to lifting is not present. There are no treatment suggestions for LVH caused by resistance training with or without the use of AAS.


    __________________

Thread Information

Users Browsing this Thread

There are currently 1 users browsing this thread. (0 members and 1 guests)

Posting Permissions

  • You may not post new threads
  • You may not post replies
  • You may not post attachments
  • You may not edit your posts
  •