whats up with that??....im on 2 mg of arimidex a day and lately ive been noticing that i don't want to have sex as often.....and when i do i go soft sometimes......ive heard that this is an effect of having no estrogen in ur body.....HELP!!
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whats up with that??....im on 2 mg of arimidex a day and lately ive been noticing that i don't want to have sex as often.....and when i do i go soft sometimes......ive heard that this is an effect of having no estrogen in ur body.....HELP!!
Why are you on 2 mg/day???? What does the rest of your cycle like.....Im using .25mg/day and no bloat or loss of sex drive.....
well im very very prone to estrogen related sides....and ive tried 1mg a day but wasn't enough....the rest of my cycle goes like this....Quote:
Originally Posted by Daniel S.
week 1-12 sust 250 every 3 days
week 4-14 EQ 400 mg a week
WHATS GOING ON????
Hey bro.......Why are you running your EQ past your test? When do you plan on starting clomid therapy?...You should stop the EQ one week before the test so the half lifes will match....
Either put your EQ at 12, or your Sust at 14. Start clomid 3wks post.
2mg of a-dex is a lot bro. I do not see how that is neccessary. Back it down to 1mg. If 1mg is possitivley not enough. Go up in .25mg incriments.
Your lipids are probably screwed right now. Get on some policosanol(OTC supp derived from sugar cane) at 20-40mg a day. 10mg of Nolva ED wouldn't be a bad idea either
i was going to run the EQ past the test b/c i wanted to harden up the gaines alittle and get the harsh androgens out of my system before starting clomid therapy.....and too tell you the truth im already on 10 mgs of Nolva a day along with the 2 mgs of a-dex.....i was thinking of just gettin rid of the arimidex all together and gettin myself a whole lot of Nolva instead.....i have HCG would this help at all???Quote:
Originally Posted by Pheedno
Help.....bump!! :(
Pheedno,Quote:
Originally Posted by Pheedno
Why do you think his lipids are screwed? Does arimidex adversely affect lipids? Please elaborate.
Quote:
Originally Posted by BASK8KACE
Inhibitors such as a-dex and femara effect lipids(primarily HDL) because estrogen greatly contributes in the stabilization of cholesterol. If you inhibit the production of estrogen, the lipid environment can become "unstable"
Now personally, a-dex/l-dex has not had an effect on my HDL(has the potential), but I've never used more than .5mg. Femara has dropped my HDL into the teens.
Nolva being a SERM, helps in eleminating blood estrogen by binding to the receptor, but doesn't prevent conversion; in addition it mimicks liver and bone estrogen which help in creating a healthy heart environment.
pheedno....help me out on what i should do....the only thing im concerned about with estrogen is gyno....thanks
he already told you, what are you missing?Quote:
Originally Posted by Pheedno
Thank you, Pheedno.Quote:
Originally Posted by Pheedno
Does proviron, which is an armomatase inbitor like arimidex, have a similar adverse effect on lipids?
Well, I've never seen any bloodwork while provrion is being taken, and I've never heard anyone mention it before. BUT, any med that prevents estrogen production has the protential to effect the lipids. Inactivators won't(aromasin), and SERMs won't
Thanks Pheedno.
I'm going through old posts and found this one. Wanted to add the following definition for others who reference this since you refered to SERM in your post:
SERM
SELECTIVE ESTROGEN RECEPTOR MODULATORS
The group of drugs classified as SERM selectively acts on estrogen receptors present in different tissues and organs: breast, uterus, bones. Their agonistic action on bone and lipid metabolism has been documented in clinical trials. Positive influence on bones appears as the inhibition of bone resorption (confirmed with bone markers) and estrogen-like increase in bone mineral density, and in consequence decrease in the risk of osteoporotic bone fractures. Their agonistic action on lipids is shown as the decrease in serum total cholesterol, LDL-cholesterol, without significant influence on HDL-cholesterol and TG.
SERM do not stimulate the uterus and breast, contrary to estrogens which increase the risk of neoplasms. Their unfavorable influence on the activation markers of hemostasis and fibrinolysis was not found.
Bump.
xxample
Quote:
Originally Posted by Pheedno
I know it hasn't clearly/definitively been shown yet, but I think aromasin will have just as significant of an effect on HDL as do the inhibitors. Whether it be competive inhibition (AIs) or complete/permanent inactivation of aromatase (specifically, each aromatase enzyme to which aromasin binds), you're still getting the same net qualitative effect.....greatly reduced estrogen conversion.
I know it's not a hugely important issue, since one would likely be using a SERM (nolva) along side of it anyway.
I agree that 2mg/day of Adex is excessive....even 1mg/day is a lot. Maybe try 1mg/day with 20mg/day of nolva, if 10mg/day doesn't cut it....that's a far better option than going crazy on the AI dosing, as you've now found out.
2mg/day is too much. 0.25mg/day of Adex and 10mg/day of Nolva should be enaught