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  1. #1
    FCECC2 is offline Anabolic Member
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    Question enanthate VS undecanoate which one is stronger?

    i was wondering if longers esters are stronger. since they stay in the blood for a long time ,the build up must be a lot greater than the shorter one.for exemple it is preferable to go with undecanoate or with enanthate ?

  2. #2
    toolman is offline Banned
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    Quote Originally Posted by TRLS63
    i was wondering if longers esters are stronger. since they stay in the blood for a long time ,the build up must be a lot greater than the shorter one.for exemple it is preferable to go with undecanoate or with enanthate ?
    Test is test, regardless of the ester. All the ester does is delay it's entry into the blood stream. The longer the ester, the slower the release.

  3. #3
    Billy_Bathgate's Avatar
    Billy_Bathgate is offline AR Vet / Retired
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    Enanthate is stronger by a few mg maybe. The undeconate ester weighs more so it takes up more of the total mass.

    This is if you are comparing equal amounts.

  4. #4
    FCECC2 is offline Anabolic Member
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    thanks bro. as for the results should i go with enanthate or undecanoate??

  5. #5
    JDMSilviaSpecR's Avatar
    JDMSilviaSpecR is offline Vicious With Malicious Intent
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    Ever considered taking prop?

  6. #6
    Pwrlfter is offline Junior Member
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    I read a paper last night on this, I got the paper for a procedure in it. TEST IS NOT TEST. Different esters have different durations and intensity. Some just outright suck, and that's why you see only certain esters on the market. Go with the enanthate bro, undecanoate has a long duration but the intensity is lower. Here's a couple examples from that paper from the guys at Shering-Plough back in 1957. In this chart we'll just look at the ester and the duration and intensity columns. The numbers in those columns are based on a comparison to test prop. For instance heptanoate, (which is another name for enanthate), has 3,3 in the duration and intensity columns. This means that enanth is about 3x as intense and it's half life is about 3x as long as prop. It has 2 numbers because they used 2 methods to test it; one number was on mice the other on rats.

    Dur. Int.
    2,2-Dimethylpropionate 0 0
    4-methylpentanoate 4 3
    enanthate 3,3 3,3
    Octanoate 2 1/2
    nonanoate 3,3 3,3
    decanoate 2 1
    10-undecenoate 4,4 2
    hexadecanoate 0 0
    4-t-butylphenoxyacetate 4,4 3,3
    cypionate 3,3 3,3
    phenylproprionate 3,3 3,3
    cyclohexylacetate 3 1

  7. #7
    scottninpo's Avatar
    scottninpo is offline Senior Member
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    i think you're confusing test with the esther, it's like saying that tren enanthate is stronger than tren acetate, it's the exact same chemical, with different esthers, you just get a tiny bit more chemical with the acetate because the enanthate esther is heavier, so it takes up more room in the total weight of the compound

  8. #8
    Pwrlfter is offline Junior Member
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    If the test never gets the ester cleaved it's biologically inactive, at that point you may as well be taking prohormones The ester serves 2 functions as far as I can tell: 1. It makes it fat soluable so it stays in the depot longer. 2. It effects the blood concentration by how easy the ester is enzymatically cleaved once it reaches the bloodstream. Here's another quote from the same paper:

    "The original hypothesis on which this study was
    based stemmed from the maximal activity of the
    lowest melting members of the straight chain acid
    series of esters, e.g., heptanoate and nonanate.
    This suggested that the more oil-soluble esters are
    most useful; therefore, a group of esters of branched
    chain acids was prepared (see Table I). The results
    show that, while none surpasses the heptanoate
    and nonanoate, several esters containing four to
    eleven carbons approached them in activity. The
    variation of the activity from good to poor of intermediate-
    length esters, all of them low melting
    solids or oils readily soluble in hydrocarbon solvents,
    shows the invalidity of the hypothesis. For
    example, the decreased activity of the 3,3-dimethylpentanoate
    compared to the 3-methylpentanoate
    is in disagreement.
    It is, however, possible to make sonie generalizations.
    ( 1) A11 of the esters with dialkyl-substitutetf
    chains have poor activity. (2) The 3-alkyl-substituted
    esters (except isobutyrate) have a low order
    of activity. These statements suggest that steric
    hindrance plays an important role in decreasing thr.
    activity. Thus, fatty acids, such as trimethylacetic
    and diethylacetic, which are known to esterify
    at less than one-tenth the rate of acetic, an&
    therefore, also to hydrolyze with difficulty, due to
    hindrance caused by branching,6 form testosteronc
    esters whose intensity is so reduced that they arc
    therapeutically useless."

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