rage problem

[MilitiaGuy]

i am on a deca /test/dbol cycle and i am having a serious problem with rage yesterday i was about to hit my dad and i insulted my teacher what should i do ?? plz help

[sd11]

IMO roid rage is complete bullshit, I think it's probably just in your head

[MilitiaGuy]

Originally posted by sd11
IMO roid rage is complete bullshit, I think it's probably just in your head

hmmm i hope so...10x

[Warrior]

What should you do?

1. Don't even think AAS causes roid rage . You will then only enable yourself to be a jerk. "It's just the testosterone - I can be out of control for now."

2. Learn to control yourself. A basic that you have to learn to deal with. Impulsive moves make horrible descions. Think before you act.

[tt333]

Use all of that rage in the gym.

[m16a2]

Originally posted by newbieguy
i am on a deca/test/dbol cycle and i am having a serious problem with rage yesterday i was about to hit my dad and i insulted my teacher what should i do ?? plz help

If your on that cycle then you must be disciplined in the gym if you hope to get good gains from it. Discipline extends outside the gym bro! Everyone gets a little rage, but you just gotta control it, otherwise you'll get yourself into a mess. When you find yourself getting heated, just take a step back, relax, and reflect on the causes for your anger. This usually occupies enough time to calm down, and I usually realize that its not worth getting mad about.

[Rickson]

Be a man and except responsibility for your actions. If you can't control yourself better then that you will be better off not taking AAS.

[psleez65]

Can only speak from personal experience, when its happened to me it seemed to be during times of elevated blood presure during cycle .Might want to get it checked out.

[mammoth]

Rickson and Warrior both made very good points. If you allow yourself to get caught up in the "I'm on roids so therefore I have an excuse" BS, you will end up creating too many distractions for yourself. People are going to question your behavior and start accusing you of being "on something". Even though you are, you don't want everybody around you to know about it. Your concentration in the gym will go to shit as your ego hits the ceiling. One of the biggest obstacles in training is humbling yourself. Don't let your attitude and actions disrupt your cycle. When you are on a cycle you should concentrate on milking it for all of its benefits. I know how it feels to get that instantaneous rush of adrenaline that makes you want to rip someones head off. Like Rickson said, if you can't handle it then don't do it.

[Pheedno]

I think roid rage is bullshit as well, a dick on is a dick off. Not calling you a dick , but I think your giving yourself a reason to be angry.

Do what I do, it sounds really strange but it works. If I'm getting really frustrated and getting pissed(on or off), I stop, go to a sink and wash my forearms.

Don't knock it, it works like a charm

[palme]

Originally posted by Pheedno
Do what I do, it sounds really strange but it works. If I'm getting really frustrated and getting pissed(on or off), I stop, go to a sink and wash my forearms.

Don't knock it, it works like a charm

I dont knock it at all bro. I leave the room and go have a glas of water to calm myself down.
Water works in mysterius ways

[Saucefit]

Roid rage seems to be a myth. You must recognize it and you'll be fine. My first cycle I found my patience to be very short, but I controlled it. Most of what I've seen is guys (idiots) who end up getting a "big head" because they are on juice. Granted you feel great, probably look great, but some people will just end up thinking they are king of the world. They get arrogant and think they can kick everyone's ass, god they look stupid. I work at a gym so I see this a lot. When I'm on a cycle I am the happiest guy in the world. Don't let it get to your head. Those idiots out there are the ones who give steriods a bad name. To close: you should never end up out of control.

[gettingbigger816]

Originally posted by Warrior
What should you do?

1. Don't even think AAS causes roid rage . You will then only enable yourself to be a jerk. "It's just the testosterone - I can be out of control for now."

2. Learn to control yourself. A basic that you have to learn to deal with. Impulsive moves make horrible descions. Think before you act.

Great advice. I think it is just mind over matter. Dont believe any of that bullshit that juice causes rage. Its all in ur head.

[humpymuscle]

kill them. kill them all.

[darmadoc]

The fact that you are aware of the rage before you act on it is good, it means you are miondful of the situation. When you start to find yourself in a situation that is causing you trouble, stop to think about what is going on and why you are angry. If that doesn't work, just put some distance between you and the situation.

I am on the same cycle and have had no such problem. In fact, I feel happy all the time. I have to go with the roid rage is overrated group. But anger is anger no matter where it comes from. You need to manage it or you'll get in trouble legally or physically (there's always some one tougher or better armed than you).

If you can't control it-you may need to get off the gear. AS has enough false bad press as it is. Good luck.

[jammergsxr]

All Roid Rage is,is a simple fact of ones maturity level.

[MBaraso]

If your an asshole before you start using AS....Chances are you'll be an even bigger one when using. It's still no excuse though....

[Got Gear?]

I'm another person who doesn't believe in roid rage .

It may make me more agressive, but it doesn't effect anger or my ability to keep a level head. Like others have said, I think it's a mental thing in your case.

And I've got a question for you... What's your age newbieguy?

[monstercojones]

i think that any androgen will only increase aggression. which only makes violent impulses harder to control, it doesnt make you more a violent person per se- it simply requires more self-control and maturity to let things go and not let things bother you so much.


chillllll out man.

go hit a bong or jerk off or something.

[tt333]

If you cant control yourself then you shouldnt be on AS.

[New GearGuy]

Two Words Bro.............. COP OUT!! Whats next the neighbor's dog barks all the time so you blow its brains out?? Where do you stop????!!!!!!!!

We all have urges ,we just have to control them.

Thats like saying you only screwed the fat ugly girl cause ' you were drunk.

Glad to see not too many people buy into this B.S.

Hope you learned your lesson man.

Just my opinon.

[MR BICEPS]

Thers alot of good points to be made. I particularly liked SAUCEFIT'S opinions!
Yes you feel great and pumped up with energy at times making it easy to make impulsive decisions. Most of it is a mentality position. I'm getting stronger and I'm not gonna take any crap from anyone.
If you have a temper you must try very hard to control it. Roids dont make you mad but when you do get mad they can makes you a little madder.
I think one becomes braver with all the added muscle,strength and energy. This can contributes to a rage but should make one feel better for the most part.
I was in a very good mood while on my first cycle but I did get mad once and crush my cell phone with my bare hands. Again you take the next step of anger if your pushed.
Ive also heard of male PMS! This is when males experience high levels of estrogen! Thats right estrogen!

[PUMP]

Originally posted by sd11
IMO roid rage is complete bullshit, I think it's probably just in your head

I agree. Unless you are a hot tempered guy to begin with.

[mmaximus25]

***Your blood pressure is already high. When in a stressful situation, confrontation, or when angry your pressure will go through the roof... sending a pump through your muscles....
***You know… there are mentally weak people that unconsciously associate a good feeling when becoming angry, due to this pump.
*** And consciously making an effort to associate being strong when angry...weak minds can not survive in our little world of biochemiclly enhancing ourselves.... and the more you stress your machine the faster you will break.
So take it out on the weights bro... they will never go anywhere no matter how much you move them...
MMAX

[platinum]

I'm not even on the juice and I have days like that. I use it in the gym. When I do go on, if it gets worse (which I doubt), I'll control it. You must decide ahead of time what you will do when it "boils over" Believe me...I know

[orto]

What researchers think:
Anabolic /Androgenic Steroid Use and Aggression I: A Review of the Evidence
by Jack Darkes, PhD
Assistant Professor,
Department of Psychology
Director of Interventions,
Alcohol and Substance Use Research Institute
University of South Florida


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Introduction
The association between anabolic/androgenic steroid (AAS) use and aggression ("’roid rage ") has been widely accepted in the culture in general, the mainstream media, and the resistance training subculture. This view has been bolstered by the use of AAS "induced" rage as a legal defense (Pope & Katz, 1990). And, although AAS use is not limited to those who perform resistance exercise, the evidence suggests that lifters using AAS are likely to use much higher doses than are those engaging in other athletic endeavors. Therefore, aggression has been both expected and reported to be more prevalent among weight trainers and this phenomenon has become part of the culture of bodybuilding, as well. More recently, naturally occurring androgen precursors have also entered the discussion (Ueki & Okano, 1999; Yesalis, 1999).

This series will examine the support for and potential strength of the causal link between AAS use and aggression and discuss putative processes associated with it. In this installment, representative research on the AAS use and aggression relationship in humans is briefly reviewed, including limited coverage of research on endogenous testosterone levels and aggressive behavior, in order to highlight prevalent themes in the literature. For a more in-depth analysis, recent reviews by Bahrke, Yesalis, & Wright (1996) and Sharp and Collins (1998) are suggested. Further installments will evaluate the evidence for a direct causal relationship between AAS use and aggressive behavior in humans, and a model in which aggression in AAS users is moderated by distal antecedent factors, and partially mediated though proximal psychological variables, will be proposed.

This series will not discuss the pharmacology of the potential AAS/aggression relationship or potential undesirable physical effects of AAS use. Such issues are addressed in many peer-reviewed and popular periodicals, including Meso-Rx. This series is not intended to suggest a lack of potential psychiatric or medical risk involved in AAS use, nor to endorse or condemn AAS use.

In general, although there has been tacit acceptance of the direct relationship between AAS use and aggression in most quarters, a review of the literature finds that support for this relationship is equivocal. In fact, in studies that controlled for extraneous factors through rigorous inclusion criteria and random assignment, there is little evidence to suggest that moderate AAS use leads to aggressive behavior. However, experimental research addressing real-world patterns and levels of use is needed.


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Testosterone , aggression, and dominance
The association of endogenous testosterone (T) with dominance, aggression, or aggressive behavior has a long history in the literature (see Bahrke, Yesalis, & Wright, 1990; 1996 for a full review). The role of T in dominant behavior among males is largely uncontested. However, the notion that dominance and aggression are the same phenomenon is not universally accepted (see Mazur & Booth, 1998). For instance, similar endogenous T levels have been found in both socially dominant but nonaggressive prisoners and their aggressive counterparts (Ehrenkranz, Bliss, & Sheard, 1974). In fact, most studies supporting an endogenous T and aggression link might also be interpreted as suggesting a T – dominance link (Mazur, 1976).

Studies unequivocally supporting a direct relationship between endogenous T and aggression have largely been accomplished with animals. This hypothesis is more rarely supported in humans. Some studies accomplished with "pathological" populations, such as prison inmates, have found that higher T relates to higher probabilities of committing violent crime, being viewed as dominant, and increased rule breaking while incarcerated (Dabbs, 1996). However, this could also reflect a link between T and dominance. Should studies support such a link, a major interpretive hurdle remains; incarcerated individuals are likely to differ from the general populace in many ways that might relate to aggressive behavior, T levels, or both. The generality of such findings is limited, providing little information about T and aggression in the general populace. Indeed, Dabbs (1996) noted that "Relatively few people out of the entire population engage in criminal behavior, regardless of their testosterone levels (p. 180)" suggesting crucial differences between incarcerated subjects and the general population that are not exclusively related to or a result of endogenous T. Such studies highlight the difficulty in generalizing from index cases (such as prisoners or individual "pathological" cases) to the general population.

Also of interest is the fact that the relationship between dominance and endogenous T is not uni-directional. Endogenous T levels not only predict dominant behavior, but are also predicted by it. Winning (the act of dominating) has been associated with an increase in T from pre to post-competition (see Elias, 1981; Gladue, Boehler, & McCaul, 1989; Mazur & Booth, 1998). Hence, increased levels of T in dominant samples might be a result rather than a cause, although this finding has not been universally supported (see Suay et al., 1999, for instance). In addition, some researchers have reported pre-contest rises in T, suggesting an anticipation of future need. This anticipatory rise in endogenous T suggests a system whereby a classically conditioned expectation exerts its influence, a system with implications for psychological theories of the AAS/aggression relationship.

In summary, the relationship between endogenous T and aggression is complex. As with most relationships between physiology and complex behavior, it reflects a "biopsychosocial" process, involving an interaction between the biological substrate of hormonal action, the psychology of the individual, and the social environment in which behavior occurs. Additionally, inconsistent definitions and operationalizations (e.g., discriminating dominance from aggression), the bi-directional effects of T and dominance/aggression, and the lack of longitudinal studies of the T/aggression link in large representative samples, are a few of the factors that complicate the examination of this relationship.


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AAS and aggression in humans
Even a cursory search of the psychological and psychiatric literature finds it replete with empirical reports and case studies suggesting that AAS users score more highly than the norm on personality scales measuring hostility. Regardless of this seeming consensus, it has recently been acknowledged that, although AAS use and aggression are correlated, the full extent and nature of the relationship remains unexplained and a clear inference of causality cannot be drawn (Beel, Maycock, & McLean, 1998). For instance, Riem and Hursey (1995) presaged Dabbs’ (1996) sentiments regarding T and aggression, but in relation to AAS use, commenting that "In sum, not all AAS users exhibit aggressive behavior, even though all experience increases in sex steroids (p. 250)." Although AAS use is reportedly widespread (see Brower, 1992), relatively few AAS users exhibit overtly aggressive behavior (rage). Factors that might underlie this variability will be discussed later in this series.

The literature on endogenous T and aggression/hostility provides little assistance in clarifying the potential AAS/aggression relationship in humans for a number of reasons. First, in contrast to endogenous T, AAS use is a behavioral choice. Hence, it is not randomly distributed within the population and AAS users are likely to differ from nonusers. Secondly, AAS ingestion and injection are not simply physical or chemical events, but also behavioral events, part of a sub-culture and a ritual.

The literature on AAS use and aggression encompasses a range of research methods. As with most drug use literature, it is heavily laden with descriptive statistics. For example, lifetime prevalence of AAS use has been reported as 9.1% for males in Great Britain (Korkia & Stimson, 1997). Between 4% and 11% of males in the U.S. have tried AAS (Brower, 1992). And 6.3% of high school football players in Indiana are current or former AAS users (Stilger & Yesalis, (1999). [For a full review of the epidemiology of AAS use see Yesalis, Kennedy, Kopstein, & Bahrke (1993).] An abundance of anecdotal "personal stories" appear in the popular bodybuilding press (e.g., Lefavi, 1998) and case studies are also frequent in the scientific literature (e.g., Corrigan, 1996; Pope & Katz, 1990; Schulte, Hall, & Boyer, 1993; Wilson-Fearon & Parrott, 1999). These data represent naturalistic evidence of this relationship. Evidence from such reports, while rich in individual detail, contributes little to an understanding of the relationship between AAS use and aggression in the larger population. They are biased in that any number of characteristics might differentiate such individuals from the general population besides their use of AAS, again highlighting the difficulty in attempting to speculate about "normal" processes, pharmacological or psychological, in "abnormal" cases. Nonetheless, such cases constitute the majority of the evidence to which the populace is exposed.

More rigorous studies involve the observation of the concurrent correlation between variables within large groups (empirical research) or comparisons between existing groups on concurrent measures (cross-sectional research). Changes in relationships may be evaluated over time, either within or between existing groups (longitudinal or prospective studies). Lastly, treatments (i.e., the administration of AAS/placebo) may be applied to either pre-existing groups (quasi-experimental designs) or to groups of randomly assigned subjects (true experimental designs) who are then evaluated over time.

Empirical and Case Studies.

A substantial amount of empirical research supports the AAS/aggression relationship. For instance, AAS users report higher levels of anger-arousal and hostile outlook than a group that never used AAS (Lefavi, Reeve, & Newland, 1990). Interestingly, data collected from former AAS users was not reported, so it is uncertain if they differed reliably from either group. AAS users exhibit increased instances of mood disorder (Pope & Katz, 1994), higher scores on aggression scales on personality measures (Galligani, Renck, & Hansen, 1996; Yates, Perry, & Murray, 1992) and measures of mood (Bond, Choi, & Pope, 1995). Nonetheless, as with the T/aggression relationship, findings of reliable differences in psychometrically assessed psychological characteristics between AAS users and non-users are not universal (e.g., Malone, Dimeff, Lombardo, & Sample, 1995; Swanson, 1989).

Several case studies (e.g., Pope & Katz, 1990) and retrospective evaluations of forensic records (e.g., Thilbin, Kristiansson, & Rajs, 1997) have also reported associations between AAS and aggression or other psychopathology. However, as noted previously, generalizing from case study data or criminal index cases to the larger population is, at best, a tenuous proposition.

The majority of the empirical and case studies suffer from methodological flaws, such as inconsistent operationalizations of aggression and differing psychometric measures (Bahrke, Yesalis, & Wright, 1996), making comparisons across studies difficult. Most rely exclusively on self-report measures of aggression, a method susceptible to several sources of bias. And, as mentioned earlier, inferring causation using such data is problematic in that AAS use is not randomly distributed in the population. The choice to use AAS, potentially at high doses, is likely to be confounded with a number of predisposing individual differences. For example, current or past AAS users might value aggression and consider aggressive responding a desirable outcome.

Ultimately, the data are largely inconsistent and inconclusive (Uzych, 1992) and a causal relationship between AAS use and aggression has not been established (Isacsson & Bergman, 1993).

Prospective and Longitudinal Studies

Choi, Parrott, & Cowan (1990) followed current AAS users and a non-using control group over a period of several months in a prospective and to some extent quasi-experimental design. The AAS group was evaluated both when using and not using AAS (an ABBA design) and non-users where evaluated at the same times, but never used AAS. A significant group (user/non-user) by drug phase (on/off) interaction for aggression, assessed by the Buss-Durkee Hostility Inventory (BDHI) resulted. Subsequent tests found no reliable effect for drug phase or user status. On the other hand, although there was no significant interaction for hostility (BDHI), there was a reliable effect for group: AAS users were more hostile than non-users, regardless of drug phase. This longitudinal (prospective) quasi-experimental (self-selected and administered treatments - used or did not use) study suggests that those who chose to use AAS were more hostile over time, whether using or not. The assessment of hostility prior to first ever drug use (difficult to accomplish given the low base rate of AAS use) would be more illuminating.

This study was quasi-experimental; there was no random assignment to conditions. Users self-selected drug use and had a prior history of use, and the controls chose not to use AAS and were lifetime nonusers. AAS users and nonusers have, in other empirical studies, differed in their mean scores on a variety of self-report and psychometric measures of personality and aggression (e.g., Galligani, Renck, & Hansen, 1996; Moss, Panzak, & Tarter, 1992). Therefore, any between group effects (as compared to "cycling on or off" differences) merely replicate the cross-sectional findings and might represent dispositional factors related to self-selection, rather than AAS use.

In a within subject, double-blind, prospective design, Su et al., (1993) examined four within subject drug phases: placebo baseline, low dose (40 mg/day) and high dose (240 mg/day) Methyltestosterone and placebo withdrawal. Each phase lasted 3 days. Significant increases in positive mood, negative mood, and cognitive impairment during high dose administration resulted. One out of twenty-nine (approximately 3.4%) participants exhibited a hypomanic episode (an atypical, but non-severe elevation of mood). Although changes in hostility across time showed a dose response relationship, the only reliable differences were between placebo and high dose time periods. These authors note that "The increased symptoms we noted during anabolic steroid administration, while significant, were subtle, reflecting several factors. First, the response to anabolic steroids across members of the subject group was highly variable, ranking from negligible to dramatic (p. 2763)." They acknowledged that marked increases in a small number of subjects were sufficient to create significant differences across time periods and, perhaps most interestingly, noted that "Symptomatic differences did not, however, reflect differences in plasma anabolic steroid levels (p. 2763)." It must be noted that this dosing pattern, a single AAS used at relatively low doses for a very short period of time, does not generalize to typical use in a naturalistic setting. In fact, as the quote above suggests, any behavioral or psychological response in this sample had less to do with blood levels of AAS than with other apparently unmeasured variables.

Gradually increasing doses of testosterone cypionate (150, 300 and 600 mg/week) or placebo were injected, in blocks of two weeks, into eight normal male volunteers, including both prior AAS users and nonusers (Kouri, Lukas, Pope, & Oliva, 1995). Aggression was operationalized as the number of button pushes chosen in order to subtract points from a fictitious opponent. The fictitious opponents’ subtraction of points from participants represented provocation. Two participants failed to believe the sham opponent deception and were dropped, leaving six participants for subsequent within subject comparisons. Increased "aggressive responding" in response to provocation, as compared to both placebo administration and baseline measures, followed testosterone administration. Higher scores were also reported on the Aggression Questionnaire at post testosterone as compared to baseline, largely due to increases in the Physical Aggression score. Whether the participants included (five lifters and 3 non-lifters: 3 with a prior history of AAS use) and the measure of aggression used provide much insight into the AAS/aggression relationship is uncertain. It was not clear which participants were excluded or, in light of the exclusions, how to interpret the statement "Since many of the subjects could not discriminate the testosterone treatment from the placebo treatment… (pp. 77-78)" in view of the small number of participants included in the analyses.

Quasi-experimental studies

Swanson (1989) examined concurrent differences between current AAS users, non-AAS using athletes, and non-using non-athletes on aggressive behavior. Group membership was verified by urinalysis. A sham reaction time competition was used and the participants’ choice of a noise level to which their "opponent" was exposed if the opponent were slower on the task constituted the measure of aggression. Participants also completed the BDHI. No between group differences were found in behavioral or self-report indices. This study is subject to the previous caveats regarding self-selection when using pre-existing groups, as well as issues related to the operationalization of aggression. Even so, while certain correlations were significant within the AAS using group, there were no differences reported between AAS users, non-using athletes, and non-using non-athletes.

Experimental Studies

Several true experimental studies, incorporating random assignment of non-using participants to AAS or placebo treatments, have recently appeared. Although the ability of such studies to generalize to self-initiated and self-maintained AAS use can be limited, they address a number of the problems associated with the cross-sectional, prospective, and quasi-experimental designs reviewed above. They constitute a true test of the AAS/aggression relationship while controlling for biases associated with self-selection and the existence of predisposing characteristics.

Bjorkqvist, Nygren, Bjorklund, and Bjorkqvist (1994) randomly assigned twenty-seven male participants to receive no-treatment control, placebo, or 40 mg/day orally administered testosterone (Panteston) over a seven day period. Both self-reported and observer-rated mood showed no effect of drug treatment. In fact, the only reliable differences reported, for self-reported anger, irritation, frustration, and impulsivity and for observer ratings of frustration, indicated that the placebo group scored higher than the no-treatment or testosterone treated groups. While, as in earlier studies, the low dose level certainly impacts the applicability of these results to real world AAS use, it is clear that anticipation and expectation played a part in participants’ observer rated behavior and self-report. However, as these authors point out "What is surprising and calls for an explanation, is the absence of a placebo effect in the group receiving testosterone (p. 24)."

Tricker et al. (1996), reported on mood and behavioral changes in a sample in which physical performance changes were reported separately by Bhasin et al., (1996). Testosterone administration (600 mg/week testosterone enanthate in 3 ml. sesame oil or a placebo of 3 ml. of sesame oil, IM) and exercise (strength training v. no exercise) were completely crossed to create four treatment cells. Forty-three males were randomly assigned to the four conditions and evaluated over a 30-week period in the following order – 4-week control period, 10-week treatment period, and 16-week recovery period. Forty participants completed the study. Attrition was unrelated to adverse drug effects. No between group difference in mood or behavior assessed via psychometric instrument, self-report, or observer (significant other) ratings were reported. As before, both dose and the use of a single drug may not accurately reflect naturalistic practice. Nonetheless, the administration of a supraphysiological dose of AAS over a 10-week period to randomly assigned participants found no reliable differences in aggression between those receiving AAS and those receiving placebo.

A recent study (Yates, Perry, MacIndoe, Holman, & Ellingrod, 1999) reported similar results. Of 42 participants randomly assigned to receive either 100, 250, or 500 mg/week of testosterone cypionate , 31 completed the study. The design included a 2-week period of placebo injections for all participants, followed by 14 weeks of injections at their assigned dose. Attrition was largely related to failure to attend weekly visits, although two 100 mg. dose dropouts were excluded due to psychological exclusions (personality disorder and high BDHI prior to treatment). One 250 mg. participant dropped out due to gynecomastia and one was lost to follow-up. One 500 mg. subject dropped out due to worsening acne and another withdrew due to adverse psychological effects (increased irritability, sleep-onset insomnia, and concentration problems – but no aggressive behavior). Analyses indicated no significant differences in attrition across the groups and no effect of non-completion on the results found with those who completed the study.

No reliable effects of any dose were found for measures of aggression, whether self-report or collateral ratings. Several quotes from these authors are noteworthy. First, they noted "…testosterone cypionate at doses of up to 500 mg/week is associated with minimal psychological effects for the majority of subjects in the study (p. 258)." However, "…the entry criteria were extremely rigorous. More than half of the potential subjects were excluded because of evidence of Axis I or II disorders or elevated psychometric measures of aggression (p. 259)." Again, the use of a single AAS and the range of doses administered do not reflect real world use, but neither do the exclusion criteria. Nonetheless, through the use of random assignment and rigorous exclusionary criteria, most potentially confounding variables (self-selection and pre-existing psychological factors) were controlled for in this study. The results suggest that, when such factors are controlled for, there is relatively little evidence to link AAS use with aggression at the doses used.


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Summary
This brief review of the literature finds no clear, consistent, and unequivocal support for the hypothesis that AAS use causes aggression. Does this refute the anecdotal reports and case studies that depict heavy steroid users as aggressive? No. Not only can such idiographic results not be generalized to the larger population, but also the normative data cannot account for all individual cases. In addition, ethical concerns regarding the use of higher dose levels and multiple AAS in experimental studies, confounds the pattern of use with the method of data collection (naturalistic, empirical, or experimental). It certainly does not refute the existing evidence for the modulation of neurotransmitter systems associated with aggression by androgens (e.g., Cologer-Clifford, Simon, Richer, Smoluk, & Lu, 1999). Does the inconsistency call into question the reflexive and widespread assumption that the use of AAS inevitably leads to aggressive behavior in humans or that such behavior is a result of purely pharmacological events? It would seem so, at least to some extent, and within the limits set by issues of dose and simultaneous use of multiple AAS. Certainly the null hypothesis, that AAS use is not necessarily causally related to aggression, cannot be rejected.

In short, as Beel et al. (1996) suggested, the literature reveals a rather complex relationship between AAS use and aggressive behavior. Perhaps this complexity has been over-simplified for mass distribution, an occurrence that is common in such instances. If so, there may be several reasons for it. The complexities of the relationship may be distilled down to imprecise bits of information for dissemination to a populace that deals best and most comfortably with short, easily digestible answers. People often desire easy to grasp dichotomies, preferring simple and clear-cut conclusions even when faced with decidedly complicated and uncertain realities. Perhaps this simplification reflects the desire to curtail the potential abuse of AAS. Such statements, that a certain drug causes undesirable behavior, often become an integral part of "scare tactic" approaches, presenting extreme or worse case scenarios to enhance negative expectations. Unfortunately, such messages mean little to ongoing users, whose experiences might disconfirm the assertion. And, conversely, such statements may heighten the drug’s appeal, should the outcome (e.g., increased aggression) be desirable to the individual contemplating use or lead to negative outcomes, when the taking of the drug facilitates the expected outcome.

[DADDYDBOL]

i am very happy while on the juice. its when im not on anything and i cant get the "pump" anymore when i get alittle short. for real its all in your head- control your emotions.

[motoxxxguy]

The question about newbie's age was never answered. I realize that this is an older post, but I wouldn't be surprised if that was his high school teacher he was pissed off at!

moto

[Got Gear?]

Originally posted by motoxxxguy
The question about newbie's age was never answered. I realize that this is an older post, but I wouldn't be surprised if that was his high school teacher he was pissed off at!

moto

Yeah, that's what I'm afraid of. This kid is probably a teenager who throw's a fit every time someone "Looks at him wrong".

[thegame01]

Take it easy bro .Roid rage is bullshit ,it's just an excuse.
Besides what the fuck are you thinkin ,hitting your dad , have a little respect.


GZ

[Leroy Brown]

These are the PC answers, but IMO, roid rage is real. What the hell are you guys on? Try shooting a gram of test in your ass and then talk to me about roid rage.

[Solrock]

Orto, excellent post.

[Solrock]

In response to the original question "what should I do?", I would say that you need, NEED, to find something that calms you. Try Ricksons suggestion of washing your forearms. Or try drinking a glass of water.

You need to find something. You could ruin it all for yourself if you let your emotions rule you.

"Control your emotion, or it will control you. The angry man loses in battle as well as in life."

Focus.

[Spyder]

The rage is all in your head man. It's controllable if you want to control it. Granted, its easier to fly off the handle when on roids, but you decide how you are going to handle things. IMO, roid rage is BS for the FDA to throw another negative in there. When I had my first cycle, I didnt have one occurance of rage, and my fellow buddies didnt either. ... except for my supplier who then stiffed me. I wanted to burn down his house...but I controlled that too .....

[Honest Abe]

yes age may be a factor if your a teen, don't have experience handling certain situations.

But more importantly, is anyone else concerned? this guys name is malitia and what the hell is that avator, a gun and a sword and arabic writing?

Bro i think you may have had some issues before you started taking AS.
Something to think about!

[Pheedno]

This thread is 13 months old

[BUYLONGTERM]

This thread is 13 months old

IT IS!!! But, I have to say, I've noticed that when I'm on TEST, I actually am more calm and more relaxed! Even my x-wife notices that Im much nicer.. You got to love TEST!

[NoLimits]

This thread is 13 months old

I was going to say that same thing... why bring back an old post to respond to his question now!!!! Unless it is an informative thread, leave it!!!!

[THA GONZ]

I agree roid rage is a myth, just an excuse for people who can't control their temper.
I have seen first hand that most people who say they got roid rage were a**holes before they started and the confidence and muscle gain from roids made them even bigger a**holes.
It all comes down to maturity.
I may get a little snappy, but am well under control and won't fly off the handle and smash some ones head in with a hammer because they cut me off or something. LOL

[johnsomebody]

Old thread or not I'd like to add something.

Test doesn't seem to make me more aggressive at all, even at relatively high doses BUT what ALWAYS makes me go ballistic is when my blood sugar gets low. I can go completely raving berserko over nothing at all. I'm less likely to do that on a cycle, in fact, cuz I make sure I'm eating well ahead of getting hungry.

I wonder if what happens to some people is because they're working out more, their body's uptaking calories more rapidly, whatever, that the real problem is low blood sugar. It's just never hit them as hard when they're not on gear.

In my case all I need is some quick carbs and I'm immediately transformed into Mr. Nice.