LOL, as any Hitchhiker's Guide knowledgeable person knows the answer is "42".
All kidding aside this is a very serious question with some significant ramifications, peripheral to which happens to be the answer to MS’s (I always liked this guy) present & extremely apt question:
There really is a very rational and comprehensible answer to both of our questions which has to do the two metabolites of the steroid hormones progesterone and deoxycorticosterone, 3 alpha-hydroxy-5 alpha-dihydroprogesterone and 3 alpha, 5 alpha-tetrahydrodeoxycorticosterone, are potent barbiturate-like ligands of the gamma-aminobutyric acid (GABA) receptor-chloride ion channel complex. At concentrations between 10(-7) and 10(-5)M both steroids inhibited binding of the convulsant t-butylbicyclophosphorothionate to the GABA-receptor complex and increased the binding of the benzodiazepine flunitrazepam; they also stimulated chloride uptake (as measured by uptake of 36Cl-) into isolated brain vesicles, and potentiated the inhibitory actions of GABA in cultured rat hippocampal and spinal cord neurons. These data may explain the ability of certain steroid hormones (and other contraindicative hormone related drugs like SERMs and AIs) to rapidly alter neuronal excitability and may provide a mechanism for the inhibiting actions of numerous naturally occurring bodiliy chemicals as well as, both synthetic anesthetic & "YES" believe it or not, similarly reactive anabolic steroid synthesis.
Merc,
Should you be so inclined, here are some of the central resources...
http://www.pubmedcentral.nih.gov/art...?artid=2042897
http://www.ncbi.nlm.nih.gov/pubmed/15282269
http://www.ncbi.nlm.nih.gov/pubmed/16702996
http://www.ncbi.nlm.nih.gov/pubmed/2598927
http://www.ncbi.nlm.nih.gov/pubmed/2888123
http://www.ncbi.nlm.nih.gov/pubmed/2445967