Which AS do NOT shut your natural test down

[Rushzilla]

Hi
I'm Running a short 5 week OT only cycle.

Question What can I use with it to geep it from shutting my natural test down?

Or is there any comparable AS that wont shut me down?

P.S Not interested in using TEST

Thanks RUSH

[Auto54]

Abuse reported

[Pinnacle]

Suppression is a better word to use.Not all anabolics totally suppress your natural testosterone output


~Pinnacle~

[Hellmaskbanned]

Hi
I'm Running a short 5 week OT only cycle.

Question What can I use with it to geep it from shutting my natural test down?

Or is there any comparable AS that wont shut me down?

P.S Not interested in using TEST

Thanks RUSH

Your natural test will be surpressed, but not shutdown.
You need to run a pct with OT , because again you will be supressed pretty bad.
Don't expect to have the gains from tbol to be like the ones from a test cycle. 5-10lbs of lean muscle would be good.

What dosage are you running it at?

[Milky87]

As previously stated, it will only suppress your test production, not shut it down completely

[TheMudMan]

Any hormone you use will suppress normal hormone production which youwill need to run {CT to get HPTA functions back to normal.

[fLgAtOr]

Ross?

[***Ross***]

*NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol , masteron , proviron , halo, primo)

Very Androgenic /Progestenic/Estrogenic steroids (Tren , Deca , Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
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Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone , estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS)

[Maetenloch]

Proviron is only the AS where there are studies showing that it has little to no suppression at moderate doses. Unfortunately it's pretty much useless for building muscle, however it does have nice prosexual effects and it will raise your free test levels.

[TheMudMan]

*NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol , masteron , proviron , halo, primo)

Very Androgenic /Progestenic/Estrogenic steroids (Tren , Deca , Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
-------------------------------------------------------------------------

Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS)

Even when HPTA is "only suppressed" the user will still need to run some sort of PCTY to get functions back to normal. The only difference IMO is how hard recovery will be from the cycle that was ran.

[**Ross**]

PCT is always required when running an AAS.

However, some steroids require LESS of a PCT protocol(no HCG or serm), as supression is NOT the same as SHUTDOWN.

For the record, one can use proviron as a supplement while "OFF" steroids. NO PCT required.


[R]