d-bol questions

[Irishguy22]

hey hows it goin guys i am starting a new cycle soon
wk1-5 50 mg d-bol ed
wk1-13 750mg test e/wk
wks1-10 500mg eq/wk

i have l-dex, nolva, proviron what substances and at waht dose should i take them in order to help combat some of the d-bol bloat( i know d-bol retains a lot of water but i would like to lessen it a bit if i can thanks a lot

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Nolva & proviron is my fav combo to fight estrogen & water retention 20mg nolva & 50mg proviron will do the job, keep the l-dex on hand & btw here is a cycle advise extend the EQ to 12wks never do EQ less than 12wks because its so slow & you wont get much out of it in just 10wks because it start to kick in between weeks 7 to 9 if you can extend the test to 14wks & then Eq to 13 you will be much happier with the results.

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[Swifto]

hey hows it goin guys i am starting a new cycle soon
wk1-5 50 mg d-bol ed
wk1-13 750mg test e/wk
wks1-10 500mg eq/wk

i have l-dex, nolva, proviron what substances and at waht dose should i take them in order to help combat some of the d-bol bloat( i know d-bol retains a lot of water but i would like to lessen it a bit if i can thanks a lot

Firstly, the Test and EQ need to be extended. Run the Test till week 14 and EQ to week 13. PCT starts 14 days after last Enan shot and 21 days after last EQ shot.

Second, I'd use the L-Dex whilst "on" and Proviron during PCT. 0.5mg/ED of L-Dex should suffice. Thats unless you have enough Proviron to run whilst "on" and during PCT? L-Dex will be more effective at reducing water retention than Proviron, but, Proviron has the added benifit of reducing levels of SHBG.

[hosam4ever]

proviron shouldnt b run during pct at all

[Swifto]

proviron shouldnt b run during pct at all

Please elaborate?

It raises serum levels of LH/FSH, masks a decrease in sex drive and reduces levels of SHBG.

[hosam4ever]

its androgin & acts like oral masteron without its anabolic effect so it will not help in bringing back the sex drive

[Swifto]

its androgin & acts like oral masteron without its anabolic effect so it will not help in bringing back the sex drive

Its not used primarily to restore the HPTA, but more of an aid. Thats where HCG and Nolva come in. The compunds I use are HCG/Nolva/Proviron. Proviron will also not affect the HPTA at high doses.

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased.

Varma TR, Patel RH.

Department of Obstetrics & Gynaecology, St. George's Hospital Medical School London, U.K.

Two hundred fifty subfertile men with idiopathic oligospermia (count less than 20 million/ml) were treated with mesterolone (100-150 mg/day) for 12 months. Seminal analysis were assayed 3 times and serum follicle stimulating hormone (FSH) luteinizing hormone (LH) and plasma testosterone were assayed once before treatment and repeated at 3, 6, 9 and 12 months after the initiation of treatment. One hundred ten patients (44%) had normal serum FSH, LH and plasma testosterone, 85 patients (34%) had low serum FSH, LH and low plasma testosterone. One hundred seventy-five patients (70%) had moderate oligospermia (count 5 to less than 20 million/ml) and 75 patients (30%) had severe oligospermia (count less than 5 million/ml). Seventy-five moderately oligospermic patients showed significant improvement in the sperm density, total sperm count and motility following mesterolone therapy whereas only 12% showed improvement in the severe oligospermic group. Mesterolone had no depressing effect on low or normal serum FSH and LH levels but had depressing effect on 25% if the levels were elevated. There was no significant adverse effect on testosterone levels or on liver function. One hundred fifteen (46%) pregnancies resulted following the treatment, 9 of 115 (7.8%) aborted and 2 (1.7%) had ectopic pregnancy. Mesterolone was found to be more useful in patients with a sperm count ranging between 5 and 20 million/ml. Those with severe oligospermia (count less than 5 million) do not seem to benefit from this therapy.