this whole thing with PBs cycles seem to have many flaws in its design. I was hoping that people on this board would be able to clear this up with me. For an example i will use the Cycle that PB made for the mr.America
its- 2 50ml bottles
bottle A
20ml deca
20ml primodepot
10ml test propt
bottle B-
20ml Test enth
20ml sust
10ml prop
day 1-5 10ml bottle A
6-12 5ml bottle A and B
13-18-bottle B each day
2mg armidex ed
30mg nolva ed
first off the placement of these aas doesnt make sense why would you place highly anabolic low androgenic compounds first, it would make more sense to use highly androgenic compounds such as bottle B first to utilize the size and strength before the body reacts with its catabolic countermeasures and to quickly peak plasma levels and then switch to the more anabolic compounds to solidify the mass gain.
second problem in this is that it is supposed to be a 28 day cycle but technically some of the compounds stay active until day 38. For instance the sustanon is stopped at day 18 and it has a 20 day active life meaning it will remain active until day 38.
third problem- there seems to be Fluxating plasma levels due to different estered steroids. at first plasma levels are sustained By the primo/test/Deca and then and then the tests blend replaces the Test/Deca/Primo plasma levels. But each Test has a different half life and active life so from day 18-38 (38 due to sustanons active life) we will experiance a fluxation in blood plasma levels.
For example
the Test prop has and active life of 72 hours and thus by day 21, it will no longer have an effect on the body and plasma levels will decrease.
The test enth has an 8 day active life so at day 26 it will no longer be active in the blood and thus have no effect and blood plasma level will decrease further
now only the sustanon is active in the body, and every day the plasma concentration decreases up until day 38 meaning less growth.
Because of these different esters we now have unstable plasma levels and which is not a suitable environement for muscle growth.
It would make much more sense to run short estered compounds along with the long estered tests to make sure plasma levels remain stable until all long acting esters are no longer active in the body. For example- run NPP,Prop, Winstrol/masteron/tren ace/bold eq( fast acting) in some combination to up plasma levels.
4th problem- placement of aas in cycle.
The timing seems way off in this cycle. For example placing primo/deca at the first part of the cycle. For Primo has a active life of of 10 days so why stop it at day 12. it could be run up to day 18 and allow for more anabolic activity.
the placement of Test prop is also way off, Why on earth whould you ever stop it at day 18 when it could be run up untill day 25 successfully and allow for less fluxation in plasma levels . As i stated above due to improper employment of long estered test we have fluxations in blood plasma levels, that problem could be fixed if test prop was use to replace the long estered tests plasma levels as they slowly stopped being active in the body. If this technique was used blood levels would remain stable throughout the cycle and give better growth.
also the placement of sustanon is off as it defeats the whole point of these cycles which is to alter the bodies homeostatis and the PTOR quickly before catabolic countermeasuers set in. But due to sustanon being stopped at day 18 it will remain active until day 38. The sustanon should have been stopped at day 10 and have had its plasma levels been replaced by a faster acting compound
5th problem- PB totes the use of armidex 2mg ed and Nolvadex at 30mg ed. This eliminates the estrogen in the body and we want to control it not eliminate it as estrogen enhances GH/IGF-1 production and increase muscle glycogen synthesis and increase androgen receptor sensisitivity which we want. It would thus make more sense to use an antagonsists so we can still have estrogen circulating in the bloodstream just not being taken up into the cell.
also Nolva is bad as it reduces gh and igf-1 production in the liver.
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Originally Posted by big k.l.g
