How bad is bridging for receptors? And
at what point does it become detrimental to bridge (ie too long of a bridge)??
the proposed bridge would include primo, eq and possibly proviron..
How bad is bridging for receptors? And
at what point does it become detrimental to bridge (ie too long of a bridge)??
the proposed bridge would include primo, eq and possibly proviron..
bump
Bridging is bad IMO, time off is the key to long term health and success. I understand why people do it but ive seen the detrimental long term results in friends, im talking serious suppression and premature forced HRT.
-XL
jing jai
Xtralarg
a follow-up question then...if there are time constraints, most people stated it would be better to stay on and bridge then do a cycle, recover--then jump back on another cycle...? thoughts?
im sure your well up on gear so ill just put my 2 peneth in.
id have thought that recovery after a long bridging/cycling period is going to be difficult and theres a possible loss of gains the longer it takes.i also think that the compound used for bridging,lets say test,the doses would have to be pushed higher and higher to get any kind of result from that compound.
i think this is when short cycling really comes into play,in and out with quicker recovery rates,makes sense to me.Originally Posted by CaptainDominate
I would never use test to bridge...my plan was using primobolan and possibly eq...i also think that the compound used for bridging,lets say test,the doses would have to be pushed higher and higher to get any kind of result from that compound.
If I were to bridge then it would be 2 short cycles(4-6 weeks) with a 4-6 week bridge, I would then have a minimum of 100 days off to recover. Im just cautious thats all.Originally Posted by CaptainDominate
-XL
jing jai
Agreed, and it works, ive done it many times!Originally Posted by helium3
-XL
jing jai
What if long esters are involved ie. primobolan? thanks for the replies too
Better for what?Originally Posted by CaptainDominate
Better for keeping gains: yes, if you are over your natrual limit.
Better for recovering your HPTA: NO!
Personally, I would never have bridged unless I knew I had self-described HRT ready when coming off. Facing chronic supression is a risk when bridging, ive seen it happen more than once...
Same principles apply IMO.Originally Posted by CaptainDominate
-XL
jing jai
wow...some guys said it Would be better for the hpta rather than going on cycle, going off, then going right back on...?Better for what?
XtralargSame principles apply IMO.
how would you get the full effect of a long ester during a shorter cycle tho? i never understood that, things like deca-durabolin, eq, primo...
They have to be run a quite a high dosage, check out Marcus300 thread on shprt cycles!Originally Posted by CaptainDominate
-XL
jing jai
I've read the thread before...but even with that high-dose (frontloading effect) how does it kick in that fast? how would you get the benefit of an eq (i know there are shorter estered versions) if its recommended to be run 12 wks min???
I dont think so. Why?Originally Posted by CaptainDominate
Comming clean off a cycle the hypotalmus/Pituirary will restore its normal functions. Letting your system getting back to normal every time with shorter intervals in between cycles, will defently be easier to recover from. Being on(bridging), the hpta will stay aspleep, and the longer you are supressed/shut down, the longer it will take for your system to get back to normal imho.
Yea i gotcha Vitor...i think those guys were assuming that i couldnt fuller recover within 8 wks...I dont think so. Why?
All I can say is that long esters taken at the right dosage work in short cycles for me and people I know. Try it for yourself, it may suit you also!Originally Posted by CaptainDominate
-XL
jing jai
Why not just bridge with GH,IGF,SLIN and clen. No suppression yet you will still be creating a very anabolic enviroment for the body.
agreed.Originally Posted by perfectbeast2001
although i did read a study that long term aas use actually upregulates the receptors...
A bit of info....
One of the most common beliefs concerning anabolic/androgenic steroid (AAS) usage is that the androgen receptor (AR) downregulates as a result of such usage. This has been claimed repeatedly in many books and articles, and it is claimed constantly on bulletin boards and the like. If I’ve heard it once, I’ve heard it a thousand times. If it were just being stated as an abstruse hypothesis, with no practical implications, with no decisions being based on it, that might be of little importance.
Unfortunately, this claim is used to support all kinds of arguments and bad advice concerning practical steroid usage. Thus, the error is no small one.
We will look at this matter fairly closely in this article. However, in brief the conclusions may be summed up as follows:
• There is no scientific evidence whatsoever that AR downregulation occurs in human muscle, or in any tissue, in response to above normal (supraphysiological) levels of AAS.
• Where AR downregulation in response to AAS has been seen in cell culture, these results do not apply because the downregulation is either not relative to normal androgen levels but to zero androgen, or estrogen may have been the causative factor, or assay methods inaccurate for this purpose were used, or often a combination of these problems make the results inapplicable to the issue of supraphysiological use of androgens by athletes.
• AR upregulation in response to supraphysiological levels of androgen in cell culture has repeatedly been observed in experiments using accurate assay methods and devoid of the above problems.
• AR downregulation in response to AAS does not agree with real world results obtained by bodybuilders, whereas upregulation does agree with real world results. (A neutral position, where levels in human muscle might be thought not to change in response to high levels of androgen, is not disproven however.)
• The "theoretical" arguments advanced by proponents of AR downregulation are invariably without merit.
The belief that androgen receptors downregulate in response to androgen is one of the most unfounded and absurd concepts in bodybuilding.
Article by Bill Roberts
Agreed bigguns..
CD, I suggested to bridge with a less suppressive compound because of your timeframe you were working with before the next show. List your schedule for these other guys to see rather than just the question..
Obviously it's more beneficial for someone to come off and recover rather than stay suppressed, but you have to keep your situation in mind and the timeline you're trying to work off of.. Do a show at a later date if this is a problem?
I cant use GH or IGF because of a back tumor...(bad luck)...Why not just bridge with GH,IGF,SLIN and clen. No suppression yet you will still be creating a very anabolic enviroment for the body.
my contest is june 2007...diet and contest cycle begins march 1....
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