PGCL--not hype

[axiomatic6]

guys this stuff is amazing

you must try it, and i have no problem shooting in to belly fat

after 2 shots around each nipple, the fatty ***osit was basically gone!!!

[kfrost06]

What is it? What does PGCL mean?

[cookiemonstR]

ya i heard about that, just waitin for some reviews, melts the fat away does it?

[axiomatic6]

hell yes, anywhere you inject it it dissovles the fat.

it also causes local muscle growth, and i have heard it will dissolve gyno...

[Cocklyaev]

dissolves gyno? this must be a joke

[reppedout1]

everything i've read about it sounds great except for the fact that you have to run to the bathroom right after shootin it...supposedly has a nasty diuretic effect

[Cocklyaev]

if the claim that it dissolves gyno is true, i would sit on that toilet for weeks with a smile ear to ear...rather than getting surgery.

[frignugs]

if the claim that it dissolves gyno is true, i would sit on that toilet for weeks with a smile ear to ear...rather than getting surgery.

HAHAHAHHAHAHAHAH, funny shit!!!! haha

[spywizard]

Treatment of 3T3-L1 preadipocytes with arachidonic acid resulted in a dose-***endent inhibition of adipocyte differentiation. The cells failed to accumulate fat droplets and did not express stearoyl-CoA desaturase 1 mRNA, a marker for late-stage differentiation. The inhibition of differentiation was reversed by the addition of cyclooxygenase inhibitors ibuprofen or indomethacin. Inhibitors of the lipoxygenase and cytochrome P-450 epoxygenase pathways were unable to reverse the effect of arachidonic acid. Dexamethasone, one of the adipogenic agents normally used to induce differentiation, could be replaced with cyclooxygenase inhibitors in the differentiation cocktail. This implicated dexamethasone as a modulator of prostaglandin synthesis in culture. Prostaglandins F[2α] (ED[50] = 0.4 nM), E[2], and D[2] prevented differentiation, each with a specific, dose-***endent affinity. Prostaglandin F[2α] was the most potent inhibitor of differentiation, suggesting that a prostanoid FP[2] receptor (FP receptor) mediates the prostaglandin action. Fluprostenol (ED[50] = 0.3 nM), a selective FP receptor agonist, prevented differentiation, confirming the involvement of an FP receptor in the inhibition of 3T3-L1 preadipocyte differentiation. Stimulation of the FP receptor for 1 h during the first day of differentiation was sufficient to cause substantial inhibition. Endogenous PGF[2α] production was lower in differentiating cells compared to unstimulated preadipocytes. These data suggest that PGF[2α] production by preadipocytes plays a role in maintaining the undifferentiated state.

[Cocklyaev]

wow...where is this from?