Bridging with Eq

[BlackWidow]

Hey bro,

Now, 3 weeks left out of my cycle and I was thinking of bridging.

Yeah, I know what some are going to say.. No no no bad bad bad... If you look with more wide prespective, you'll see all we discuss here is about bad steroids but we are trying to get the benefits with less or no price to pay...

And that's what I am trying to do here....

Let's review what I thought about....

First, I have done free/total test. before any steroids so, I have a base!

This is my last week of Test enanthate. I'll be waiting 2 weeks to start my PCT. So, makes a total of 3 weeks left till first PCT day.

I'll start EQ this week @ 200 mg EW.
Then start my PCT which consists of Cloid, Nolva and Prov. for 8 weeks

I will run the eq for 8 weeks so the last week of EQ will be PCT week 5 leaving 3 weeks till the end of PCT AND also till EQ got completely out of my system...

Then BLOOD WORK

If base reached, CONGRATULATIONS
If not, Another 4 weeks of Clomid @ 100 mg ED and Nolva @ 40 mg ED then another BLOOD WORK

I think it'll be OK

Then wait another 4 weeks ...... yeah bros, BLOOD WORK AGAIN

Then new cycle!!

So, it will be like this:

Last week of cycle: Eq @ 200 mg
Wait-week 1: Eq
Wait-week 2: Eq
PCT
Week 1: Clomid 100 - nolva 40 - prov 50 - eq
Week 2: Same
Week 3: same
Week 4: same
Week 5: same
Week 6: Clomid 50 - Nolva 20 - prov. 50 - NO EQ
Week 7: same
Week 8 same

Sorry for making such long post but that's what I'm thinking about

Looking for a good discussion and any help will be appreciated!

Thanks

[crawdaddy]

Simply not a good idea. Give your body the proper amount of time to return to normal and you will "reap" the benefits in the long run. As much as I often look for the same quick fix, its just not smart. Good luck.

[Bigmax]

bro 200mgs of EQ a week is almost a waste....not to mention it will take up to 5 weeks to actually kick in...and you want to stop in week 8??...quite honestly dont think you'll see to much or get to much out of it in cycle or while doing your PCT..

[Mazzive_T]

^^^^ what they said. Dont do it.

a) its affecting your pct
b) its not worth it.

finish your cycle, do your pct, then use the equation time on + pct = time off, before you start again.

T.

[BG]

Bridge with GH/IGF or Slin or all.

[BlackWidow]

Bridge with GH/IGF or Slin or all.

Believe me, Slin is dangerous!! Its side effects are not fuly understood on these boards...

[BlackWidow]

bro 200mgs of EQ a week is almost a waste....not to mention it will take up to 5 weeks to actually kick in...and you want to stop in week 8??...quite honestly dont think you'll see to much or get to much out of it in cycle or while doing your PCT..

You're right... But remeber, kickin is very different from just action.
Test. enanthate kicks in after 5 or 6 weeks but it dose start action within 2 days of the first inj. getting eleased from its ester....

Kick in time is the time that the conc. of the substance reach a level that can produce noticable difference....

But me, I don't want to notice a thin... I just want a small amount of an anabolic substance while trying to get my natty levels back to normal.. That's it!

[hugovsilva]

You will be running pct while having equipoise hanging around. What is the purpose of trying to recover while administering suppressive androgens?

[magic32]

But me, I don't want to notice a thin... I just want a small amount of an anabolic substance while trying to get my natty levels back to normal.. That's it!

I don't follow the logic here...

You will be running pct while having equipoise hanging around. What is the purpose of trying to recover while administering suppressive androgens?

Superb question!

...or here

[Njord]

You will be running pct while having equipoise hanging around. What is the purpose of trying to recover while administering suppressive androgens?

My guess is he's thinking that the EQ is not suppressive.

[football2007]

Believe me, Slin is dangerous!! Its side effects are not fuly understood on these boards...

please explain...

[BlackWidow]

My guess is he's thinking that the EQ is not suppressive.

No, I know Eq is suppressive... But less suppressive than the test I was using for sure.

There's a common concept in treating any kind of drug depemdence... You have a substance that you cannot withdraw, so you replace it with a weaker one of a similar action with easier withdrawal. This method take more time but is of the same effectivenss.

[BlackWidow]

please explain...

Antibodies against insulin

[taiboxa]

Antibodies against insulin

way to elaborate... anyways every study i have seen seems to indicate that the antibodies are not a response to the use of exogenous insulin but instead are a genetic defect.

[magic32]

Sounds a lot like Darwinism.

No, I know Eq is suppressive... But less suppressive than the test I was using for sure.
THIS IS A GOOD THEORY, BUT WITH IDENTICAL ANABOLIC RATINGS, WHY WOULD YOU BELIEVE IT?

There's a common concept in treating any kind of drug depemdence... You have a substance that you cannot withdraw, so you replace it with a weaker one of a similar action with easier withdrawal. This method take more time but is of the same effectivenss.
THIS IS TRUE ONLY IF THE PERSON IS MERELY SUPPRESSED AND NOT SHUT DOWN. DURATION WOULD BE THE INDEPENDENT VARIABLE, AND IF YOU WERE ON ENTH FOR A SIGNIFICANT AMOUNT OF TIME, THEN SWITCHING TO EQ (EVEN AT LOW DOSES) WON'T BEGIN NATTY RESTORATION. YOU'RE BODY WILL RECOGNIZE SUPRAPHYSIOLOGICAL DOSES AND "NEVER" RESTART PRODUCTION...I.E. CRUISING.

AGAIN THE QUINTESSENTIAL QUESTION IS, "WAS I SUPPRESSED OR SHUT DOWN?"

[BlackWidow]

Sounds a lot like Darwinism.

As for identical steroids ratings, it also says, Eq anabolic ratio is 100 so the same as testosterone. Is this true?! Can you tell me that gains on 500 mg Eq equals those on 500 mg Test? NEVER. So, these ratings are deviated from what happens in the real life.
Eq is way less suppressive than testosterone. I have friends that use Eq as a supplement without any exogenous test with and without PCT and they're just fine. I don't mean it's not suppressive but low suppressive is the word....

How can you tell that you're suppressed or shut down?! I dunno!

Let's just say, I am shutdown. ok?! Running EQ alone will never give a chnce to my enogenous hormones to recover, but REMEMBER, I am running it with an aggressive PCT. So, wouldn't that atleast change me from the shut down satate to the being only suppressed? Then from this suppressed state I got to, it will be much more easier to regain my haemostatsis...

That's my idea!

[BlackWidow]

way to elaborate... anyways every study i have seen seems to indicate that the antibodies are not a response to the use of exogenous insulin but instead are a genetic defect.

Yes, antibodies against insulin can be a genetic defect. But, this's only seen with type I DM.

When you're using exogenous insulin, your body MAY recognize it as an antigenis substance and your immune system responds by starting producing antibodies against it...

That's a way to differntiate between a patient with insulin-producing tuour and exogenous insulin intake. With exogenous insulin intake, you MAY find antibodies.

Notice the word MAY carefully!

[hunter7]

i dont have any first hand knowledge but i was always curious about what you are considering. In Jose Canseco's book juiced, he talked about how he would cycle off test but run var and winny year round and he claimed to know everything about steroids. I would imagine if you could recover to 75% while still running EQ alone your gains would be easier maintained. but the question is how much can you recover while still running EQ? i would love to hear your results and plus if your doing bloodwork i dont think you will have to much of a problem. another thing is everyone parrots 12 week cycle then pct which is usually what i do but people without this knowledge do all kinds of stuff and many claim to have no problems. people use hcg on way more suppressive cycles so if you took that for pct and continued EQ theoretically that should work. Any criticisms of this?

[BlackWidow]

i dont have any first hand knowledge but i was always curious about what you are considering. In Jose Canseco's book juiced, he talked about how he would cycle off test but run var and winny year round and he claimed to know everything about steroids. I would imagine if you could recover to 75% while still running EQ alone your gains would be easier maintained. but the question is how much can you recover while still running EQ? i would love to hear your results and plus if your doing bloodwork i dont think you will have to much of a problem. another thing is everyone parrots 12 week cycle then pct which is usually what i do but people without this knowledge do all kinds of stuff and many claim to have no problems. people use hcg on way more suppressive cycles so if you took that for pct and continued EQ theoretically that should work. Any criticisms of this?

I'll keep you posted bout the results.

As for the HCG thing, I am not a fan! You're taking steroids inhibiting your LH and FSH leading to decreased endogenous Testosterone. So, the only functioning step in your Hypthalamic pitutary axis is GnRH (LHRH & FSH)... Taking HCG will suppress them too leaving you without a scratch to start from!!

That's my opnion about HCG.

[hugovsilva]

Not if taken with nolvadex and an AI (Aromasin would be the choice here)

[BlackWidow]

Not if taken with nolvadex and an AI (Aromasin would be the choice here)

It remains suppressive with whatever substance you're taking with.

[BG]

^^^I have look that one up, I dont think thats totally true, allthough I do agree, hcg would be one of worst bridging ideas. I still stick with my first recommendations, I know many guys that use slin, NONE have had problems and some are oldschool guys, been doing it along time.

[taiboxa]

i used slin for a year straight and had no problems whats so ever.

[magic32]

I don't think I've ever seen anyone with so much misinformation. You know a whole lot, you really do, but your application of said knowledge is severely misappropriated.

As for identical steroids ratings, it also says, Eq anabolic ratio is 100 so the same as testosterone. Is this true?! Can you tell me that gains on 500 mg Eq equals those on 500 mg Test? NEVER. So, these ratings are deviated from what happens in the real life.
Eq is way less suppressive than testosterone.
THE ANABOLIC RATING ISN'T MEANT TO TRANSLATE INTO MUSCLE BUILDING, ELSE TREN WOULD GIVE YOU 5 TIMES MORE MUSCLE THAN TEST, AND HALO 19 TIMES MORE. YOU SHOULD LEARN WHAT THE ANABOLIC & ANDROGENIC RATINGS ACTUALLY MEAN.

I have friends that use Eq as a supplement without any exogenous test with and without PCT...
BRILLIANT USE OF AN ANABOLIC STEROID.
THEY SHOULD BE COMMENDED, AND CONTINUE TO RECEIVE ALL THE PLAUDITS YOU'RE PRESENTLY BESTOWING UPON THEM.

...and they're just fine.
AS DETERMINED BY WHAT ENDOCRINOLOGIST? PLEASE LIST HIS/HER NAME AND PLACE OF EMPLOYMENT, OR DO YOU MEAN BY THEIR AND YOUR DEFINITION OF "JUST FINE"?

How can you tell that you're suppressed or shut down?! I dunno!
THIS IS SOMETHING ELSE YOU REALLY NEED TO LEARN, ESPECIALLY IF YOU'D LIKE TO ENGAGE IN RATIONAL DEBATE ON THE MATTER.

Let's just say, I am shutdown. ok?! Running EQ alone will never give a chnce to my enogenous hormones to recover, but REMEMBER, I am running it with an aggressive PCT.
THERE ARE CERTAIN FUNDAMENTAL HORMONAL PRINCIPLES THAT THE BODY ADHERES TO, AND PCT ISN'T CAPABLE OF SUPERCEDING OR EVEN SUSPENDING THEM.
PEOPLE CONSISTENTLY AND ERRONEOUSLY ATTRIBUTE PROPERTIES TO PCT, AS YOU'RE NOW DOING, THAT SIMPLY DON'T EXIST.

So, wouldn't that atleast change me from the shut down satate to the being only suppressed?
THIS IS A VERY GOOD EXAMPLE OF THE AFOREMENTIONED PROPERTIES.

Then from this suppressed state I got to, it will be much more easier to regain my haemostatsis...

That's my idea!

Let’s delve into this one.
After, and the key word is "after" cycle bb'ers use PCT for two primary reasons. First and most importantly, it bridges the time between the cessation of aas and the restoration of natty test, by using the body's own preference for synthetic estrogen to fill receptors, thus preventing actual estrogen from attaching at the receptor level. This prevents the Test/Estro ratio imbalance, which greatly favors estro at this time, from influencing and therefore promoting undesirable estrogenic responses. This is the primary intention and effect of pct. It also possesses the secondary effect of prodding the body to increase natty test production.

SERMs taken during cycle or in conjunction with aas, CANNOT be considered PCT as it is an obvious contradiction to both terminology and function. POST-cycle-therapy simply CANNOT occur DURING cycle…that would make it "cycle-therapy" for which there are valid applications, but not the one you’re intending. This scenario would still provide the primary effect listed above, but not the secondary one. Once shut-down occurs, the testes cannot even begin natty restoration until supraphysiological doses are no longer sensed by the negative feedback loop, and even after aas clearance there is still a waiting period...the aforementioned “bridge” time. So PCT could never have the effect you’re proposing while even low doses of EQ are present, IF that is, they are taken succeeding a full Test cycle. Now were we just talking about low dose suppression cycle of aas, then sure, one could taper or phase out a drug by continually reducing the dosage.

Based on your Q's & A's in this thread, I think you should take some time to study some of the finer points of aas. But I must admit, you are a thinker...and I like that.

[hugovsilva]

It remains suppressive with whatever substance you're taking with.

I hear so many people saying that HCG should never be run during pct without elaborating. Let me elaborate why I think it can.

According to Anthony Roberts:

“Unfortunately, while HCG increases Testosterone, it increases estrogen as well(12). As you probably know, estrogen acts directly on the Leydig cells to effect changes in the activities of enzymes important for testosterone synthesis (13) and may actually be considered an important part of that negative feedback loop I mentioned earlier. In addition, an increase in circulating levels of LH have been shown to induce down-regulation of LH-receptors in both rodent studies (14), as well as in human studies (15); since HCG mimics LH, you can expect it to do the same. This LH downregulation can cause an increase in steroidogenic cholesterol (the cholesterol earmarked by your body for conversion into testosterone). (16). Thus, after the initial HCG induced surge in testosterone is over, if you have used enough to downregulate your LH-receptors and increase estrogen too much, then more steroidogenic cholesterol is available. This is telling me that less is being converted to testosterone. In fact, rodent models suggest that if you take a dose large enough to cause a sharp increase of plasma testosterone, you will actually desensitize your Leydig cells to your next shot, and will possibly not experience any rise in testosterone from the second dose at all, or may only experience a very slight one at best (17.). Since this is due to LH-Receptor downregulation, and that occurs in human models too, it is pretty fair to assume that if your first dose of HCG is too large, your second won’t be very effective. Unfortunately, this lack of an increase in testosterone doesn’t necessarily mean that the HCG may be unable to increase circulating levels of Estrogen (18) And remember that increase in Estrogen will (most likely) cause your body ultimately to produce less testosterone. Low LH post-cycle is not the primary cause of slow recovery, because LH generally rises to levels above baseline after a cycle much sooner than testosterone production does. This is probably because the pituitary is working very hard to get your atrophied Leydig cells to start producing testosterone again. HCG should also bring back testicular volume; I feel the need to mention this because it’s important to me and I suspect most men as well.
It would also appear that HCG works very well when it’s used on men who have low levels of LH to begin with (as you would be after a cycle), as many studies on pre-pubertal boys and Hypogonadotropic Hypogonadal men would suggest (19)”


This one of the possibilities for suppression.

“This suggests that a pre-exposure to normal LH levels or gonadatropins in general is necessary for HCG-induced Leydig Cell desensitization. This, of course is not a problem for us, as we’ll be using it when LH/Gonadatropin levels are very low anyway …we just need to stop using it before we regain normal function, or it will work against us eventually. (19) (20). Luckily, the temporary Anabolic steroid induced hypogonadism that is experienced after a cycle basically allows us to respond to HCG like anyone with low LH levels (21), and thus, as I told you, a lot of the possible inhibitory effect of HCG is not going to be relevant because there was no prior “priming” by circulating gonadotrophins. This is great news for us, because we are going to be using HCG during PCT, when we need to get back some HPTA function, and not when we have levels of gonadatropins high enough to cause HCG-induced desensitization.”

This is what rebounds it.

“But are we still risking some inhibition and possibly delaying our recovery by using HCG? Probably not…you see, some studies in humans have shown that HCG does not actually have a direct effect on inhibiting LH release in men (22)(23), but rather (probably) works to inhibit LH secretion indirectly, simply by stimulating the production of testosterone (thus activating the negative feedback loop). Another factor involved is the induction of testicular aromatase, which raises estrogen levels, again causing inhibition. Unfortunately, yet another process, the downregulation of the Leydig Cell LH receptor itself, seems to also play a role in high dose HCG testicular desensitization. This is also done by HCG actually blocking the conversion of 17 alpha-hydroxyprogesterone (17 OHP) to testosterone (24)”

Here is another way by which HCG could cause suppression

“Nolvadex actually stops this blocking-action of HCG from taking place (25). Most likely, because of Nolvadex’s direct antiestrogenic effect and LH-upregulating effect on the Pituitary, suppression of gonadotropins via HCG is (25) almost totally stopped with concurrent administration of Nolvadex! So if we Use Nolvadex and we are only using HCG when we are low in gonadatropins, we won’t be inhibited by it at all! Right?”

Again, the counter measure.

“Well…maybe…but there’s still the issue of estrogen caused by that HCG-stimulated surge in testosterone. Well…we can use low doses (300iu or so) to avoid some of that major spike in estrogen, and thus cause far less inhibition from the HCG (26). Of course, I’d want to use a bit more HCG per injection (500iu), if I could, to get my body functioning fully more quickly, and lose less of my gains. Maybe we can get away with taking some Vitamin E with our HCG, since it increases the responsiveness of plasma testosterone levels to HCG, making them significantly higher during vitamin E administration than without it (27). So we can get a better
response with our HCG by taking Vitamin E (I recommend 1,000iu/day), but that doesn’t get rid of the problem that we have, which is the estrogen increase the HCG will cause.”

But Estrogen can still be a problem.

“Lets solve that pesky estrogen problem now….

Lets add in an Aromatase Inhibitor! Which one, though? Well, since we are already using Nolvadex, we can’t use Letrozole or Arimidex, as the Nolvadex will actually greatly decrease the blood plasma levels of them (28)!

So we have to use Aromasin (exemestane) as our AI, because it’s an aromatase inactivator, meaning it makes estrogen receptors useless, and instead of just inhibiting production (as an anti-aromatase would do) it cuts off production totally. Aromasin can also cause androgenic sides (29)(30)(31), which may help to elevate your mood while you are on PCT. This final drug in my recommended PCT can effectively remove up to about 85%+ of estrogen from your body (32). Most importantly, using Aromasin together with Nolvadex doesn’t reduce exemestane’s effectiveness (33). So now, I think the problem of ANY inhibition possible with HCG is solved, and we can use that 500iu/day dose that I wanted to use previously.”

Estrogen problem solved.

I have elaborated why I think (Following AR's work) HCG can be used in PCT (I actually checked the studies mentioned in the original thread). Would you mind elaborating why you think it can’t?

Of course it will have no use if you continue to run androgens along with it.

[BlackWidow]

I don't think I've ever seen anyone with so much misinformation. You know a whole lot, you really do, but your application of said knowledge is severely misappropriated.



Let’s delve into this one.
After, and the key word is "after" cycle bb'ers use PCT for two primary reasons. First and most importantly, it bridges the time between the cessation of aas and the restoration of natty test, by using the body's own preference for synthetic estrogen to fill receptors, thus preventing actual estrogen from attaching at the receptor level. This prevents the Test/Estro ratio imbalance, which greatly favors estro at this time, from influencing and therefore promoting undesirable estrogenic responses. This is the primary intention and effect of pct. It also possesses the secondary effect of prodding the body to increase natty test production.

SERMs taken during cycle or in conjunction with aas, CANNOT be considered PCT as it is an obvious contradiction to both terminology and function. POST-cycle-therapy simply CANNOT occur DURING cycle…that would make it "cycle-therapy" for which there are valid applications, but not the one you’re intending. This scenario would still provide the primary effect listed above, but not the secondary one. Once shut-down occurs, the testes cannot even begin natty restoration until supraphysiological doses are no longer sensed by the negative feedback loop, and even after aas clearance there is still a waiting period...the aforementioned “bridge” time. So PCT could never have the effect you’re proposing while even low doses of EQ are present, IF that is, they are taken succeeding a full Test cycle. Now were we just talking about low dose suppression cycle of aas, then sure, one could taper or phase out a drug by continually reducing the dosage.

Based on your Q's & A's in this thread, I think you should take some time to study some of the finer points of aas. But I must admit, you are a thinker...and I like that.

You have a many points there, bro. I really would like to have a converation or a debate as you called it with you... So, let's just get started....

[BlackWidow]

Anabolic ratings:

I think anabolic:androgenis ratings are obtained by comaring the actions of various hormones on the levator ani muscle(causing hypertrophy and this's anabolic) and on prostate size(causing enlargment and thi's androgenic) in RATS..

So, it's more of a recommendation than a fact....

So, if you look to our members, you can see HUMANS giving thier experience so, their experiences are more reliable than ratings...

If you didn't hear an experience of that - and I am sure you heard - anyone took an eq only cycle can tell you that it's way less suppressive than test.

[BlackWidow]

They're just fine...

You DONNOT need an endocrinologist to teel you that you're fine. Nowdays, the medicine is on the way to be completely INVESTIGATION-BASED MEDICINE... So, when you got graduated from your medical school, you'll be handed a big chart showing the diagnostic approach to every single disease. The patien will just come and say, I feel ...... and you get your chart out and tell him ( go to the lab to do .... and we'll get the result soon ) then treat him, BUT, were you listening?!! The patient will come and say I FEEL......

Without an individual presentation, you can do nothing. When somebody is telling you that he has no symptoms of anything, you can't just drag him and put medications in his body.

Yeah, we are taking bad steroids and that's a risk factor but unless there's a symptom. we're fine. Individual presentation is way more important than risk factors...

Just to illustrate, a 70 years old male comes to your clinic with a history of uncontrooled diabetes, hypertension, very high LDL, smokin 3 packs of cigarette a day and family histpory of angina, his whole family died at the age of 70 from angina.. and he's complaining from abdominal pain.... Does he have an angina? No
He has aaaalll the risk factors BUT his presentation doesn't say so, so he doesn't have it.

They have no symptoms, so they're fine.... very simple!!

[BlackWidow]

Suppressed or shut down...

You said I've to learn it and I really want to.
Now I am on test. I have erections I have an ejaculatory volume less than normal but it's OK.... Am I suppressed or shut down?!!

[BlackWidow]

There's smoething I wanna talk about...
Why everbody's hinking that 1 mg of test is the same as 1 mg of deca as 1 mg of eq as 1mg of whatever substance. This is a 1 mg of AAS. This is wrong...

The body normally produce 4-9 mgs of test ED so makes a total of 28-63 mgs a week... let's just say 50 mg EW.
So, are you telling me that if I took 50 mg EQ that will be a total replacement to my tets and my LH will be zero coz I don't need any test?!! Got the point.

The normal replacement dose is 125-200 mg EW of TEST. to replace the natty 50 mg.. Doesn't this give you an idea that body reacts differently with the exogenous test rather than it does with endogenous?? And MORE IMPORTANT test to different substance....

These 200 mgs of eq may be not supra physiological as you think and that's what makes pct working during a bridge....

[BlackWidow]

bump

[Bigmax]

There's smoething I wanna talk about...
Why everbody's hinking that 1 mg of test is the same as 1 mg of deca as 1 mg of eq as 1mg of whatever substance. This is a 1 mg of AAS. This is wrong...

The body normally produce 4-9 mgs of test ED so makes a total of 28-63 mgs a week... let's just say 50 mg EW.
So, are you telling me that if I took 50 mg EQ that will be a total replacement to my tets and my LH will be zero coz I don't need any test?!! Got the point.

The normal replacement dose is 125-200 mg EW of TEST. to replace the natty 50 mg.. Doesn't this give you an idea that body reacts differently with the exogenous test rather than it does with endogenous?? And MORE IMPORTANT test to different substance....

These 200 mgs of eq may be not supra physiological as you think and that's what makes pct working during a bridge....

Bro,all due respect...alot of guys have told you what they think...you obviously are dead set on doing EQ during PCT for a bridge...By all means go for it...some will agree some will not...I know I dont...but its your body and your situation to deal with...good luck....tell us how it goes...i'm curious.

[BlackWidow]

Well Bigmax,

I'll do a bllod work before starting PCT and another one after finishing it with the bridge.. And we'll see!!

Thanks, bro.

[Bigmax]

Good bro..keep us posted...

[Elexecution]

bump, i'm interested in these blood work results..

[ChuckLee]

Believe me, Slin is dangerous!! Its side effects are not fuly understood on these boards...

There are many people that understand that, don't speak generally "on this boards"...I understand slin, if used correctly with proper diet it's fully under control.

CL

[BlackWidow]

There are many people that understand that, don't speak generally "on this boards"...I understand slin, if used correctly with proper diet it's fully under control.

CL

Very informative post!! Thta's the only thing you wanna say... that u understand slin?!! Congratulations...

[ChuckLee]

Very informative post!! Thta's the only thing you wanna say... that u understand slin?!! Congratulations...

You are nobody to say that in this board people don't fully understand side effects of slin. Many users use it after an accurate research and find fine with it without any side. What is your experience with it, as an expert I suppose?

[TallMan]

yeah BlackWidow, you said in a previous post that without symptoms you're fine

[BlackWidow]

yeah BlackWidow, you said in a previous post that without symptoms you're fine

Thta's right!! Without symptoms, you're fine BUT you can't deny you're taking a risk....
Do you know that the most common morbidity cause is idiopathic?? in another word.. abnormal response. It's not abnormal, it's just didn't figured out yet.

With any AAS you use, you can do just fine and then BOOM testicular tumour... it's rare!

As for slin, it's more documented that body can produce antibodies..