
Originally Posted by
meathead320
Hi all,
Heard of a method from SWIM, (who the f*ck is SWIM) on controling E2 and Prolactin, on cycle, with heavy doses of AAS that leads to lots of prolactin, and the dreaded "deca/tren-dick", as well as gyno and such.
AAS do not directly increase PRL, even 19-Nor's.
As we all know, we do need some Estradiol, but not too much. Too low and we don't make gains as well, and too low is also a big culprit in ED. That and prolactin being the big culprit in ED from low libido as well.
Too high/low an estrogen level will cause ED.
To high a prolactin level will cause ED.
Stress, nervousness, alcohol consumtion and other psychological issues will cause ED, not all hormonal.
These are the kinds of boner killing that PDE-5 drugs cannot help us with as if E2 is too low (actually they do, take PCT for example), or prolacting too high it kills desire, no getting the mood, not a blood flow issue.
Anastrozole is good for controling E2, but does not do much for controlling prolactin, although it does tend to stay ok so long as E2 levels are kept in check.
Not true.
Prolactin is regulated as estrogen is regulated and synthesised. Its called the Long Feedback Mechanism. Get your boy SWIM to read up on it.
Some guys over time, even on test alone, can get high Prolactin anyway.
Thats because anything that interacts with the estrogen receptor (ER), can increase prolactin. Any steroid that interects with the ER can push PRL off the chart.
This obviously more common with 19-nor steroids however.
Nonsense.
Show me a study that shows 19-Nor's increase prolactin more than other steroids that meerly interect with the ER... You wont find one. Because its parrotted garbage on these boards. Its CAN happen, but not where near as you would think and guru's and research chem sites have you believe.
Nandrolone and Trenbolone with interact with the progesterone receptor (Nandrolone is a full agonist), but that doesnt mean prolactin will shoot off the charts.
Well, Letrozole does bring down Prolactin (Not directly, because it controlls estrogen), as well as harshly dropping E2. Often guys have trouble controlling it and drive E2 down too low.
Well SWIM claims to have experimented with this, and found a very odd solution. He takes a high enough dose of letro to completely destroy E2 formation all together. Bring down prolactin as well.
Right on the money. But I bet he didnt know what I have just explained above because his theory's dont add up.
Then he take 1/4th the normal dose of an Eythl Estradiol female birth control pill ED.
What for??
I don't know what the dose is, mg or mcg or whatever, but the thought of killing off all E2 and Prolacting with letro, then replacing the E2 itself with a tiny dose of EthnylEstradiol sounds crazy, yet there is logic to it.
None at all. Why not control estrogen, not destroy it with another AI (Aromasin 10mg/ED) then take a dopamine agonist to control prolactin IF it increases. Talk about stupid...
Anyone here ever tried this? results? was it able to control the prolactin and keep the E2 normal/level on cycle?