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  1. #1
    PropAndTren is offline Junior Member
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    If Nolva and AI's are used to increase height in short people. (theoretical)

    Would they stop growth plates from closing on cycle?
    Maybe even an increase because of the testosterone effects?

    Say

    500mg Test E: 10 weeks
    Letro: 2.5mg EOD
    HCG : 750iu

    Nolva: 40/40/20/20/20

    This is beyond an issue for me as i'm 24 and just started my first cycle of prop.
    Last edited by PropAndTren; 05-17-2012 at 08:20 AM.

  2. #2
    jasc's Avatar
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    Growth plates closing stops your bone structure from growing. This has nothing to do with muscle building

    AI's can be given to teens to help them to continue to grow by limiting estrogen production. Estrogen plays a big role in closing growth plates.
    Last edited by jasc; 05-17-2012 at 08:15 AM.

  3. #3
    PropAndTren is offline Junior Member
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    Quote Originally Posted by jasc
    Growth plates closing stops your bone structure from growing. This has nothing to do with muscle building

    AI's can be given to teens to help them to continue to grow by limiting estrogen production. Estrogen plays a big role in closing growth plates.
    But would they stop plates from closing on cycle?

    Ie. Letro on cycle and nolva for PCT.

    As both are used for increasing height in short adolescents?

  4. #4
    jasc's Avatar
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    Quote Originally Posted by PropAndTren

    But would they stop plates from closing on cycle?

    Ie. Letro on cycle and nolva for PCT.

    As both are used for increasing height in short adolescents?
    I believe so, if the plates hadn't already closed.

    In order for someone to be on cycle with growth plates that hadn't already closed they'd have to be cycling at a pretty young age.

    Ideally the Letro n Nolva would keep them open, but I'm not sure how massive amounts of exogenous test would affect the growth.

    Maybe that's how Lou Ferigno got to be 6'4

  5. #5
    PropAndTren is offline Junior Member
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    Quote Originally Posted by jasc

    I believe so, if the plates hadn't already closed.

    In order for someone to be on cycle with growth plates that hadn't already closed they'd have to be cycling at a pretty young age.

    Ideally the Letro n Nolva would keep them open, but I'm not sure how massive amounts of exogenous test would affect the growth.

    Maybe that's how Lou Ferigno got to be 6'4
    Do you know anything about clomid in terms of this?

    Yeah i'm 24 and I read the plates in the shoulder clavicle can grow up to 25 but i'm guessing thats the highest estimate.
    Too late anyway I already started my prop 3 weeks ago.

    It would be an interesting experiment to see if cycling like that ie keeping estro very low to keep the growth plates open would actually work.

  6. #6
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    JohnnyVegas is offline Knowledgeable Member- Recognized Member Winner - $100
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    Quote Originally Posted by PropAndTren View Post
    It would be an interesting experiment to see if cycling like that ie keeping estro very low to keep the growth plates open would actually work.
    Growth plates are not the only problems facing a youth using AAS.

  7. #7
    PropAndTren is offline Junior Member
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    Quote Originally Posted by JohnnyVegas

    Growth plates are not the only problems facing a youth using AAS.
    I know i'm just trying to see if it would work or not.

  8. #8
    jasc's Avatar
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    Quote Originally Posted by PropAndTren

    Do you know anything about clomid in terms of this?

    Yeah i'm 24 and I read the plates in the shoulder clavicle can grow up to 25 but i'm guessing thats the highest estimate.
    Too late anyway I already started my prop 3 weeks ago.

    It would be an interesting experiment to see if cycling like that ie keeping estro very low to keep the growth plates open would actually work.
    I'd assume Clomid wouldn't work because it does not control the aromatization of Test to estrogen because it is a SERM and cannot directly lower E2 levels, but that is just a guess.

    My knowledge on this is fairly limited as I have only read a few studies on AI's n growth plates and a couple threads on here. There was one a while back where a kid was using AI's for this under the supervision of his dr. Pretty interesting read, may try to search for it.

  9. #9
    PropAndTren is offline Junior Member
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    Bump

  10. #10
    PropAndTren is offline Junior Member
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    Quote Originally Posted by jasc

    I'd assume Clomid wouldn't work because it does not control the aromatization of Test to estrogen because it is a SERM and cannot directly lower E2 levels, but that is just a guess.

    My knowledge on this is fairly limited as I have only read a few studies on AI's n growth plates and a couple threads on here. There was one a while back where a kid was using AI's for this under the supervision of his dr. Pretty interesting read, may try to search for it.
    From my reasoning I think it may work, hopefully a vet chimes in.

  11. #11
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    Quote Originally Posted by PropAndTren

    From my reasoning I think it may work, hopefully a vet chimes in.
    Serms probably wont work. They dont block all estro receptors. They actually block some and stimulate some. Simple way to research, read their wiki page.

  12. #12
    PropAndTren is offline Junior Member
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    Serms like nolva have been used to block estro and therefore keep growth plates open for longer in short boys though?

  13. #13
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    Quote Originally Posted by PropAndTren View Post
    Serms like nolva have been used to block estro and therefore keep growth plates open for longer in short boys though?
    i am not sure of the specific medical use, or which country or which hospital centre use it that way.

    but a quick look at nolva's wiki page, i found this..

    under Tamoxifen /Side Effects

    Bone

    A beneficial side effect of tamoxifen is that it prevents bone loss by acting as an estrogen receptor agonist (i.e., mimicking the effects of estrogen) in this cell type. Therefore, by inhibiting osteoclasts, it prevents osteoporosis.[38][39] When tamoxifen was launched as a drug, it was thought that tamoxifen would act as an estrogen receptor antagonist in all tissue, including bone, and therefore it was feared that it would contribute to osteoporosis. It was therefore very surprising that the opposite effect was observed clinically. Hence tamoxifen's tissue selective action directly led to the formulation of the concept of selective estrogen receptor modulators (SERMs).[40] In contrast tamoxifen appears to be associated with bone loss in premenopausal women who continue to menstruate after adjuvant chemotherapy.[41]


    Premature growth plate fusion

    While tamoxifen has been shown to antagonize the actions of estrogen in tissues such as the breast, its effects in other tissues such as bones has not been documented fully. There have been studies done in mice showing tamoxifen mimic the effects of estrogen on bone metabolism and skeletal growth. Thus increasing the possibility of pre-mature bone fusion. This effect would be less of a concern in adults who have stopped growing.[55]




    not a real medical paper, i know, but its good for a quick reference.

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