Prolactin and Recovery

[qscgugcsq]

I posted a similar question about Estrogen about 2 weeks ago.

First of all, I must say that 19nor really intrigate me and I didn't read enough to understand it very well, So If I'm wrong I want to be inform. I'm here to learn.

Normally, the recovery of a 19nor cycle is harder than other estrogen related cycle..

If I'm right it's because the prolactin and progesterone affect negatively the recovery of the pituitary. Prolactin/progesterone = low LH/FSH = Low test (a really basic way to see this...)

So...

I often see advice like: Keeping low estrogen should limit the progestrone sides. (low estrogen will limit the production or prolactin and progesterone right?)

But still, there is some progesterone and prolactin even if the estrogen is under control, only there is not enough to feel side effect.

Normally, the cabergolline/pramil must be kept on hand.

BUT, Doing a burst of cabergoline/pramil just before the pct(exemple: the last 2 weeks .25 cabergoline EOD).

So, Even if the estrogen is under control, the cabergoline will drop the progesterone/prolactin enough to make the recovery easier.

I've been told that with estrogen, keeping it at a normal quantity will be more efficient at restoring the pituitary than drop it completly, but men are supose to have very few progesterone/prolactin, so if the goal is to be at normal to recover it makes sence to lower as much as possible these hormones...

What do you think of all this?

[qscgugcsq]

Bump!!!!!!!

[redz]

I always run caber up to pct with Tren. I think it helps a ton in the recovery department.

[kelkel]

Your body needs estrogen. Keep it at a normal level and that in and of itself will control prolactin in most cases. Having caber on hand is always a great idea as we are all different and you're not getting BW every week. No need to lower them as much as possible. Just keep them normal. BTW it does not take much caber to crush your prolactin level. Maybe .25 x 2 per week would probably suffice for most people, imo. EOD is overkill.

[redz]

Maybe .25 x 2 per week would probably suffice for most people

Interesting I have been running too much by the sound of it. I have done 0.5 x2 per week

[OnTheSauce]

I run .5 2x a week

[qscgugcsq]

Up to now it seems that I am right about a burst of cabergoline sould be great but still I'va only got opinion up to now.

Does someone actually tried it and saw a difference between recovery or have any link about research who has been done???

It would be really apreciated.

[kelkel]

Have not heard anyone speak directly to that, and back it up scientifically...

[Bonaparte]

19-nors themselves are extremely suppressive to the HPTA. More so than estrogen or anything else.

[Perseverance1]

I run .5 caber e4d.....it works well for me as far as my dick working goes.

[qscgugcsq]

19-nors themselves are extremely suppressive to the HPTA. More so than estrogen or anything else.

Yea, but does the fast shut down make it hard to recover or it's the prolactin and progesterone left in the body or maybe both...??

[slumchop]

i am a huge nor19 fan, run deca every cycle. Im old, helps the joints and the gains are great. running nor19 can be tricky, you run deca and tren complete opposite of each. deca you run other. deca you run 50% less than your test your running and tren you run higher than the test. i always stop nor19 at least two weeks prior to finish of cycle and finish last two weeks on test alone than go to PCT. shut down is hard that why i stop nor19 early. always run an ai, i run aromsin and prami

[Bonaparte]

Yea, but does the fast shut down make it hard to recover or it's the prolactin and progesterone left in the body or maybe both...??

Well, Swifto was the expert on this suff, but my understanding is that there is no actual increase in progesterone. 19-nors themselves are progestins and can activate the progesterone receptor as well as the AR. And the prolactin thing is a a fairly rare occurence and can be eliminated by using an AI (since PRL and PRG require E2 to work), or directly with a dopamine agonist. So the suppression is from the drug itself (and its long-lingering metabolites), not these other hormones.