Hallo guys,
I like that you would read this article because I find it quite interesting:
https://www.******.com/forum/steroid...-hpta-shutdown
Can you give me your personal opinion about ?
thanks
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Hallo guys,
I like that you would read this article because I find it quite interesting:
https://www.******.com/forum/steroid...-hpta-shutdown
Can you give me your personal opinion about ?
thanks
ok I post the article directly:
Using Anabolic Steroids Without HPTA SHUTDOWN!
RESEARCH SHOWS THAT NOT ALL STEROIDS CAUSE SHUTDOWN!
*NO MORE POST-CYCLE CRASH!
*You can now USE CERTAIN STEROIDS DURING PCT! *(Pre-PCT)
*You can now formulate a cycle that will NOT CAUSE SHUTDOWN
Some steroids only REDUCE TESTOSTERONE PRODUCTION(to varying degrees), whereas other steroids will SHUTDOWN the HPTA resulting in a complete cessation of androgen production.
NOT ALL ANDROGENS CAUSE SHUTDOWN
"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.
SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Turinabol, Anavar, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan)
Very Androgenic/Progestenic/Estrogenic steroids(Trenbolone, Nandrolone, Anadrol, Testosterone) cause a COMPLETE shutdown of endogenous hormone production.
The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier recovery!
The Following steroids will NOT SHUTDOWN THE HPTA:
Turinabol, Anavar, Proviron, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan, Clostebol, and 4-ADiol.
Pre-PCT: PRE-PCT allows the HPTA to begin LH/FSH output, while still receiving additional anabolic support. This is the peroid of time where we utilize a NON-inhibitory steroid while the endogenous testosterone level begins to recover. This occurs PRIOR TO FULL PCT, so that by the time we begin full PCT the HPTA has already began recovering.
Active RECOVERY: The HPTA BEGINS to restore endogenous testosterone production once it detects the body's androgen level beginning to decline(end of cycle).
Therefore, HPTA CAN BEGIN TO RECOVER WHILE STILL IN AN ANABOLIC STATE!
The following drugs can be used during Active Recovery:
Anavar/Proviron= 40mgs/25mgs
Anavar/Masteron= 40mgs/300mgs
Primobolan/Masteron= 300mgs/300mgs
Turinabol/Proviron= 40mgs/25mgs
Turinabol/Masteron= 40mgs/300mgs
Winstrol/Masteron= 50mgs/300mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/300mgs
Examples...
In a SHORT CYCLE:
Weeks 1-4: Testosterone Propionate, 100mgs ED
Weeks 1-4: Dianabol, 50mgs ED
Weeks 1-4: NPP, 400mgs
Weeks 4-8: PRE-PCT(ACTIVE RECOVERY)
Weeks 8-?: POST CYCLE THERAPY
A Standard Cycle:
Weeks 1-6: Dianabol, 30mgs ED
Weeks 1-10: Testosterone Enanthate, 500mgs
Weeks 8-12: Winstrol, 100mgs ED
Weeks 12-16: PRE-PCT(ACTIVE RECOVERY) **
Weeks 16-26: **POST CYCLE THERAPY
DO NOT end your cycle ABRUPTLY! Don't just END your cycle cold-turkey! If you are SHUTDOWN, full restoration can take weeks and even MONTHS. Therefore, one should REMAIN ON minimally-inhibitive STEROIDS(HPTA) in an attempt to MAINTAIN the gains they made while ON CYCLE, while STILL BEGINNING TO RECOVER TESTOSTERONE PRODUCTION. On top of that, one still continues to progess from the mild additional anabolic support.
NOT only does it mean that you can run a COMPLETE CYCLE with NO SHUTDOWN whatsoever(as long as the right compounds, dosages, and durations are used), it also means that if you ARE SHUTDOWN from your cycle, you do NOT HAVE TO COME RIGHT OFF CYCLE! Actually, it is BETTER TO STAY ON CYCLE WHILE YOUR ENDOGENOUS TESTOSTERONE LEVEL BEGINS TO INCREASE!
You may also run a cycle that COMPLETELY AVOIDS SHUTDOWN:
Weeks 1-6: Dianabol, 40mgs ED
Weeks 1-10: Anavar, 50mgs ED
Weeks 1-10: Masteron, 100mgs EOD
Or
Weeks 1-6: Dianabol, 40mgs ED
Weeks 1-10: Primobolan, 500mgs
Weeks 6-14: Turinabol, 60mgs ED
And Many many more! There are tons of NON-inhibitory cycles that you can devise using my my list above for your guideline. Your days of HPTA suffering are over!
By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
The Hypothalamus has Androgen, Estrogen, and Progesterone receptors.
Each and EVERY anabolic steroid affects these receptors DIFFERENTLY.
Some steroids affect ALL receptors, while some only affect ONE type of receptor, while others have very little effect on ANY of these receptors.
UNDERSTANDING WHICH steroids affect which receptors, and to WHAT DEGREE, will FULLY enable the steroid user to COMPLETELY and systematically AVOID HPTA SHUTDOWN!
By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
Steroids that cause an OVERSATURATION(too many receptors activated) of these various hormone receptors, WILL CAUSE SHUTDOWN.
Steroids that DO NOT CAUSE an OVERSATURATION of ANY of these various hormone receptors, will NOT cause SHUTDOWN!
The Following drugs either DIRECTLY or INDIRECTLY activate ESTROGEN receptors, to varying degrees:
Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
The Following drugs either DIRECTLY or INDIRECTLY activate PROGESTERONE receptors, to varying degrees:
Nandrolone
Trenbolone
Oxymetholone
The Following drugs activate Androgen receptors, to varying degrees:
Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
Trenbolone
Halotestin
Oxandrolone
Stanzolol
Chlorodehydromethltestosterone
Methyltestosterone
Methenolone...
(ALL AAS*)
As we can see, the steroids that cause HPTA SHUTDOWN either OVERSATURATE ONE SPECIFIC receptor, or they activate too many TOTAL receptors(Androgen/Estrogen/Progesterone)
For instance, Trenbolone causes HPTA SHUTDOWN because it OVERSATURATES BOTH, the ANDROGEN and the PROGESTERONE receptors.
Testosterone causes SHUTDOWN because it converts to ESTROGEN and DHT, therefore, it oversaturates the Androgen/Estrogen receptors.
As we can ALSO SEE, the steroids that DO NOT cause SHUTDOWN of the HPTA, do NOT oversaturate ANY of the different hormone receptors, and thus, do NOT cause SHUTDOWN.
Methenolone(Primobolan) does not possess ANY Estrogenic or Progestational ACTIVITY WHATSOEVER. It does, by virtue of being an anabolic steroid, posses a SMALL Androgenic component. Because it lacks ANY ESTROGENIC/PROGESTATIONAL component, and it lacks a strong Androgenic component, it WILL NOT CAUSE SHUTDOWN!
Oxandrolone(Anavar) posseses NO Estrogenic/Progestational component either. AND, it also lacks a strong androgenic component. Thus, Anavar will NOT cause shutdown.
By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
*It must also be noted, that ANY steroid in LARGE enough DOSAGES for long enough DURATIONS, can cause SHUTDOWN of the HPTA.
NOT ALL ANDROGENS CAUSE SHUTDOWN*
"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.
SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)
Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.
The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
Interesting article. Would be curious to see some Vet's thoughts.
The whole text can be summarized by this^^^ last sentence.Quote:
Originally Posted by TestoSuper
Breaking it down:
We know this is not so true, plenty of examples of guys taking anavar only cycle and having tons of problems, as equally many guys taking full deca cycles recover easily and fast. Too many variables and unknowns in play to be able make this claim, as we know it is many times, a lucky draw.Quote:
The distinction between SUPRESSION and SHUTDOWN is utterly important...
Retention of gains is mostly decided by HPTA recovery, proper training and nutrition IMO.Quote:
...as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle...
Why quicker PCT? How would we decide to make the PCT quicker. Easier IMO depends, like said before, on a number of factors which are impossible to predict.Quote:
....and a quicker, easier PCT.
Its a lot more complicated than the text sugests, and the idea of this active recovery is nonsense to me.