This is just brainstorming on my part. The need for this thought was born from the recognition that there are individuals like myself that take extremely long to recover from cycles. We're not talking long-cycles either... The cycles in question are the very standard cycle-lengths i.e. 8-12 weeks in duration.
The above-mentioned incidence of mal-recovery is the governing reason why individuals like myself and Mr. Sparkle stay away from exogenous adrogens...as the shut-down in unbearable...and recovery difficult and sketchy. The fear exists that at some point...after a cycle one may NEVER recover. That being said...here's my idea for perpetual cycling.
The cycle is two-part...a combination of two short cycles really. The difference being...the emphasis isn't on JUST androgen-mediated growth. Rather, growth is sought in both the 'ON' phase and the 'PCT'.
Compounds used while on will be taken from this list:
**NB: this is not an exhaustive list... the concept is to use a short-acting compound...and to some extent any short-acting, averagely-suppressive steroid could be added to the test-base...Yes...Testosterone IS the base. I'll explain further down.
Injectibles: Testosterone-Propionate, Trenbolone Acetate, Nandrolone PhenylPropionate...etc.
Orals: t-bol, winstrol, anavar...etc.
D-bol can be added if an Aromatase Inhibitor is used. The point of leaving it out of the list is because it aromatises easily. An increase in estrogen will increase SHBG...and also further suppress the HPTA...That's a double negative.
Aromatase Inhibitors: anastrazole (Arimidex® exemestane (Aromasin®
; letrozole (Femara®
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Prolactin Modulator: Bromocriptine... Quasi-essential if Trenbolone is used.
Insulin Sensitivity Modulating Compounds: Metformin; Avandia
Yes...by now you're seeing where i'm going with this aren't you?![]()
Compounds while 'off':
Standard 'PCT' meds: Nolva with preference...and Clomid (I'd personally go with Nolva..because Clomid can act as a mild estrogen and hypothetically could be HPTA-suppressive in some instances...tho this is subject to debate.I think i should add i no longer use Clomid because of said effects noticed in myself.)
Insulin: Humalog only... Exogenous insulin stimulates both LH and FSH..making it a great addition during PCT.
Nootropics: Nootropics are used because they increase Nerve-Growth Factor(NGF) in the HPTA. NGF increases the recovery rate of the glands in the brain, specifically the hypothalamus and pituitary. They include Hydergine, Piracetem, and Selegiline ...among others. (Google: "Nootropic+Substances +increase+testosterone"...for more info)
The Cycle Structure...
Test-prop: Weeks 1-6
Tren-A: weeks 1-6
Bromocriptine: Weeks 3-6 (just an outline... The bromo doesn't need to be started this late. It would be if no sides are evident earlier. The point in starting it later, in the absence, is to speed recovery at the end of the cycle... as Bromo acts to decrease the amount of prolactin that the pituitary releases. It keeps prolactin in check while stimulating sperm production and erectile function. Three weeks because... If used too frequently or for too long, it can lead to poor appetite and decreased receptor sensitivity.)
Metformin: weeks 1-6
Arimidex: weeks 1-6 (Conventional thought suggests Letro administration is necessary 2 weeks prior to the commencement of the cycle to get serum levels up to an effective level at the commencement of the cycle... so really If Letro is used over arimidex...the protocol will change slightly to facilitate this two week period)
Weeks 7-12
Nolvadex: Weeks 7-10 (Standard PCT doses) Weeks 11-12 (Maintenance doses: extended PCT. Doses will be similar to those used during a normal cycle at this point (e.g. 10-20 mg ED))
Insulin: Humalog Post-workout...Weeks 7-12 (Yes Six weeks on slin)
Nootropics: Pirecetam/ALCAR Weeks
The train of thought...
Instead of looking at cycling in the standard veiw..with testosterone manipulation being the base of the veiw. We'll look at it from the point of veiw of manipulating insulin more effectively.
The first half of the cycle is for increasing insulin sensitivity...tru androgen/anxillarty application. (Testosterone application increases insulin sensitivity.)
The second half of the cycle is for capitalising on that environment of increased insulin sensitivity through the administration of exogenous insulin. Exogenous insulin administration increases testosterone production through the increase in Luteinising Hormone (LH) and Follicle Stimulating Hormone (FSH)... This, coupled with the synergistic administration of SERMs and Nootropics creates an ideal environment for Post-Cycle Testosterone recovery.
Why i think this approach is both advisable and hypothetically applicable.
1. Because both halves of the cycle are growth-oriented...Recovery is faster on short cycles... and seeing that the 'cycle' isn't one-sided (i.e. One half is suppressive...while one is recovery-oriented..but BOTH are growth-oriented)...it isn't disimilar to a 12-weeker... Just easier to recover from.
2. Time on = time on
3. Because of it's structure..it is plausible that it could be repeated nearly indefinately. There is no need to take 12 weeks off...because theorectically, weeks 7-12 are recovery-oriented.
4. Exogenous Humalog administration has been shown to reduce insulin resistance...when used post-prandially as illustrated here. So insulin resistance isn't a limiting factor here either.
Again..this is just brain-storming...a hypothesis...not an actual article
Thoughts?
~Narkissos