I have been around a few years and had the opportunity to watch some internet myths develop. Very interesting to say the least. But they're just silly. Remember, this is for education AND discussion. And I'm not bashing anybody for stating what they feel they have "learned". Most of it is just a wording issue, or a lack of understanding.
These are just a few that came to mind as good for discussion. DO NOT take offense to it.
- Clomid sides are so bad nobody should use it.
Not really at all. I've seen hundreds of users and roughly 10% (estimated) have sides that require discontinuation of use. Remember, clomiphene is stronger at stimulating LH release, which is what we want during PCT. And comparing clomiphene to tamoxifen simply on a mg for mg basis has no bearing on this. If it takes 100000000mg of one substance to work better than 40mg of the other, I'm taking it because it's my health at risk and I want what works the best.
- You MUST have even hormone blood serum levels.
Nope. There are differing sides on this view, but one thing is for sure: it is NOT a requirement. I hear people say sustanon HAS to be injected EoD. No it doesn't. I remember I used to copy/paste that write-up on this like I was some kind of brainless lemming. Yes, EoD will take full advantage of the short esters, and will allow even hormone concentrations. But the idea of sides from uneven levels is exaggerated. In fact, some would argue that because of the human body's desire to maintain homeostasis, you would want fluctuating levels of hormones, as in a tide cycle. Even blood levels have not shown any effect on gains, mostly because the vast majority of AAS users are using multiple times more hormones than required for the level of growth that their body is undergoing.
- LR3 IGF-1 causes injection site growth.
Oh, this one gets the theorists going. Too bad theory doesn't pan out in the real world as often as the theorists would like. Of the hundreds of LR3 users I've known, most still say they're not convinced of injection site growth. The theory is that the growth is because they are injecting into the muscle they just worked, but IGF also works hardest on the muscle that is damaged from the workout, so regardless of where they injected it was still gonna work most on the muscle worked. Now I know some theory boy with 6 months of experience will come on and tell me how wrong I am because he read a write-up on it. Good for him. Go eat, kiddo! Come back when you SEE enough experiments to draw your own conclusion.
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Originally Posted by N4cer
