Tricks for GH Bloat?

**FranKieC** · 07-06-2007, 09:13 AM

Any ideas?

I started at 212 and now am up to 220-222. My pants size is the same if not a little more loose. Is there anything I can do to drop this water?

**taiboxa** · 07-06-2007, 10:26 AM

uhh.. dont be a pu$$y? lol

**stocky121** · 07-06-2007, 10:28 AM

what dose are you running and how did you ramp up ?

**FranKieC** · 07-06-2007, 12:12 PM

Originally Posted by taiboxa

uhh.. dont be a pu$$y? lol

lolool

**FranKieC** · 07-06-2007, 12:13 PM

Originally Posted by stocky121

what dose are you running and how did you ramp up ?

5iu's serostim

Started at 3.3ius then ramped up ever two weeks

Should I come back down to 3iu's? Or should I get some Dandiline Root?

**fred9** · 07-09-2007, 04:06 PM

Blockade of the renin-angiotensin-aldosterone system prevents growth hormone -induced fluid retention in humans
J. Moller, N. Moller, E. Frandsen, T. Wolthers, J. O. Jorgensen and J. S. Christiansen
Medical Department M (Endocrinology and Diabetes), University Hospital of Aarhus, Denmark.

To test if the renin-angiotensin-aldosterone system (RAAS) is involved in growth hormone (GH)-associated fluid retention, we examined the effect of GH administration in the presence or absence of RAAS blockade at different levels on body fluid homeostasis. Eight subjects were examined in a controlled, randomized double-blinded trial. During four 6-day periods they received subcutaneous GH (6 IU-m-2) or placebo injections and tablets as follows: 1) placebo and placebo, 2) GH and placebo, 3) GH and captopril, and 4) GH and spironolactone. GH increased extracellular volume (liters; placebo 18.87 +/- 0.85; GH + placebo 20.43 +/- 1.01) but this effect was abolished by captopril (GH + captopril 18.82 +/- 0.67) and spironolactone (GH + spironolactone 18.99 +/- 0.85). Correspondingly, the GH-induced reduction in bioimpedance was blocked by captopril and spironolactone. Plasma renin and angiotensin II concentrations increased during all three GH treatment regimens, whereas plasma aldosterone was increased only after GH plus spironolactone. The data demonstrate that GH activates the RAAS and that blockade of the RAAS by two separate mechanisms prevents fluid retention normally encountered after GH exposure. These observations suggest that the RAAS plays a key role in GH-induced regulation of fluid homeostasis.