Heres what the cycle will be...
wk 1-14 Test E @ 750 mg/wk
wk 1-14 Masteron E @ 600mg/wk
wk 1-10 Tren E @ 600mg/wk
PCT: Clomid, Nolva, Myogenx, Cyogenx, Creatine...
Any other suggestions, opinions, comments on proper PCT procedures?
Heres what the cycle will be...
wk 1-14 Test E @ 750 mg/wk
wk 1-14 Masteron E @ 600mg/wk
wk 1-10 Tren E @ 600mg/wk
PCT: Clomid, Nolva, Myogenx, Cyogenx, Creatine...
Any other suggestions, opinions, comments on proper PCT procedures?
Moush,
That is the exact cycle I am looking at for my next cycle (mine will be slightly lower doses). I am still experimenting and researching the proper pct for me. Currently I am in week 2 of my pct consisting of nolva, raloxifen and aromasin. So far it is working great. I feel much better than I did on my last pct (aromasin, nolva and hcg).
I had kind of dismissed clomid as old fashined and outdated, but after reading x_moes's posts, I might add it in with my next pct. So if I were to run your cycle my pct would look something like this:
Week 1-6 Nolvadex 20mg ed
Week 1-6 Clomid 50mg ed
Week 1-6 Aromasin 25mg ed
I might even run hcg during cycle and up to pct to maintain testicular form and function, but I wouldn't run it during pct.
thanks njord...what happened to running clomid 300mg for 1 day, 100mg day 2-15 and 50 mg from then on?
aromasin?? is that l-dex?
I know some people like to run their clomid at higher doses in the beginning, but I have ran it at 50mg ed and way fine on it. I'm wondering if people weren't experiencing all those side effects because of the higher doses. You have to figure it out for yourself how you want to run it.Originally Posted by moush
Aromasin (Exemestane) is a steroidal aromatase inhibitor, it is an AI like aramidex(l-dex) but works on a different mode of action.
Here is a quote from dailymed:
"Exemestane is an irreversible, steroidal aromatase inactivator, structurally related to the natural substrate androstenedione. It acts as a false substrate for the aromatase enzyme, and is processed to an intermediate that binds irreversibly to the active site of the enzyme causing its inactivation, an effect also known as "suicide inhibition"
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