i was wondering if longers esters are stronger. since they stay in the blood for a long time ,the build up must be a lot greater than the shorter one.for exemple it is preferable to go with undecanoate or with enanthate ?
i was wondering if longers esters are stronger. since they stay in the blood for a long time ,the build up must be a lot greater than the shorter one.for exemple it is preferable to go with undecanoate or with enanthate ?
Test is test, regardless of the ester. All the ester does is delay it's entry into the blood stream. The longer the ester, the slower the release.Originally Posted by TRLS63
Enanthate is stronger by a few mg maybe. The undeconate ester weighs more so it takes up more of the total mass.
This is if you are comparing equal amounts.
thanks bro. as for the results should i go with enanthate or undecanoate??
Ever considered taking prop?
I read a paper last night on this, I got the paper for a procedure in it. TEST IS NOT TEST. Different esters have different durations and intensity. Some just outright suck, and that's why you see only certain esters on the market. Go with the enanthate bro, undecanoate has a long duration but the intensity is lower. Here's a couple examples from that paper from the guys at Shering-Plough back in 1957. In this chart we'll just look at the ester and the duration and intensity columns. The numbers in those columns are based on a comparison to test prop. For instance heptanoate, (which is another name for enanthate), has 3,3 in the duration and intensity columns. This means that enanth is about 3x as intense and it's half life is about 3x as long as prop. It has 2 numbers because they used 2 methods to test it; one number was on mice the other on rats.
Dur. Int.
2,2-Dimethylpropionate 0 0
4-methylpentanoate 4 3
enanthate 3,3 3,3
Octanoate 2 1/2
nonanoate 3,3 3,3
decanoate 2 1
10-undecenoate 4,4 2
hexadecanoate 0 0
4-t-butylphenoxyacetate 4,4 3,3
cypionate 3,3 3,3
phenylproprionate 3,3 3,3
cyclohexylacetate 3 1
i think you're confusing test with the esther, it's like saying that tren enanthate is stronger than tren acetate, it's the exact same chemical, with different esthers, you just get a tiny bit more chemical with the acetate because the enanthate esther is heavier, so it takes up more room in the total weight of the compound
If the test never gets the ester cleaved it's biologically inactive, at that point you may as well be taking prohormonesThe ester serves 2 functions as far as I can tell: 1. It makes it fat soluable so it stays in the depot longer. 2. It effects the blood concentration by how easy the ester is enzymatically cleaved once it reaches the bloodstream. Here's another quote from the same paper:
"The original hypothesis on which this study was
based stemmed from the maximal activity of the
lowest melting members of the straight chain acid
series of esters, e.g., heptanoate and nonanate.
This suggested that the more oil-soluble esters are
most useful; therefore, a group of esters of branched
chain acids was prepared (see Table I). The results
show that, while none surpasses the heptanoate
and nonanoate, several esters containing four to
eleven carbons approached them in activity. The
variation of the activity from good to poor of intermediate-
length esters, all of them low melting
solids or oils readily soluble in hydrocarbon solvents,
shows the invalidity of the hypothesis. For
example, the decreased activity of the 3,3-dimethylpentanoate
compared to the 3-methylpentanoate
is in disagreement.
It is, however, possible to make sonie generalizations.
( 1) A11 of the esters with dialkyl-substitutetf
chains have poor activity. (2) The 3-alkyl-substituted
esters (except isobutyrate) have a low order
of activity. These statements suggest that steric
hindrance plays an important role in decreasing thr.
activity. Thus, fatty acids, such as trimethylacetic
and diethylacetic, which are known to esterify
at less than one-tenth the rate of acetic, an&
therefore, also to hydrolyze with difficulty, due to
hindrance caused by branching,6 form testosteronc
esters whose intensity is so reduced that they arc
therapeutically useless."
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