Thread: Milk Thistle on Cycle
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09-22-2006, 09:07 PM #1
Milk Thistle on Cycle
Im still a noob trying to gather info so go easy on me. Obviously it is necessary to take Milk Thistle after your cycle, but is it a good idea to take it during a cycle? Will it in any way minimize or limit the full potential of being on gear? Or should it be taken everyday along with multivitamins and other supps/vits?
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09-22-2006, 10:46 PM #2
if you are taking an oral then yes you have to take it during your cycle. you dont have to but if you want to save your liver than take it
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09-23-2006, 12:17 AM #3
I start taking supps 1 week before cycle, like a loading phase, then through cycle, and through PCT.
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09-23-2006, 12:19 AM #4~ Vet~ I like Thai Girls
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Originally Posted by kyjelly
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09-23-2006, 09:01 AM #5
When you say supplements you mean, milk thistle and all the other vitamins correct? Kale your avis are the f@cking greatest!!!
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09-23-2006, 09:59 AM #6Originally Posted by SuperLift
yes, as in red yeast rice, hawthorn berry, flush free naicin, NAC, milk thistle, CoQ10, etc.
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09-23-2006, 11:06 AM #7Originally Posted by kyjelly
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09-23-2006, 11:28 AM #8Originally Posted by SuperLift
yeah, make sure you got everything down before doing any cycle. And PCT!
but yeah, milk thistle is for liver, the others r for blood pressure and chloestroel and such.
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09-23-2006, 11:55 AM #9Originally Posted by kyjelly
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09-23-2006, 11:59 AM #10Originally Posted by SuperLift
lol, yeah, they sure do. on my cycle of superdrol, I got a nose bleed cuz of blood pressure.
all those supps are a must in my book, and Im pretty sure in almost ev1's here.
You need to know correct PCT also, if you dont that is.
look at the sticky at the top by byran. it pretty much hits most supps. IMO, you dont need all of them, but the top1 or 2 in each category I think is needed.Last edited by kyjelly; 09-23-2006 at 12:02 PM.
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09-23-2006, 12:08 PM #11
read UpStateTank's SD log.
Upstates SD Log
he plans out supps and PCT. Note that it is for an SD cycle, and not test or anything, but it is basically the same.
I think with illeagal roids, like test, you throw in nolva throughout the cycle. and milk thistle isnt needed for it isnt an oral and doesnt mess up liver.
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09-23-2006, 09:40 PM #12Banned
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You should be on milk thistle 24/7 on or off cycle. protect the liver as much as possible
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09-26-2006, 06:59 PM #13
Don't forget saw palmetto on cycle (prostate enlargement)
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09-29-2006, 09:46 PM #14Originally Posted by SVTMuscle
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09-29-2006, 09:47 PM #15
I know people mean well with reccomending the 'liver protection supplements', but it's pretty much a huge racket to make people money.
These products (liver protectors) were designed to offset the symptoms
of bacterial or viral hepatitis and fatty liver disease/cirrhosis. Yes, in
those conditions, LFT's may be elevated quite a bit but the underlying
mechanism causing the increase in LFT's is vastly different then the
*potential* rise in LFT's related to C-17a alkylated steroids .
So these "liver supps" probably won't do squat WRT to "protecting
your liver" against this mysterious quasi-phantom "liver problem"
everyone thinks orals cause.
Your liver is a very resilient organ - you would have to be down to
less than 10% liver function before you would die as an FYI
Liver toxicity is overblown (greatly) and has only been documented
with the methylated stuff and only with a few of them anyhow.
And, liver enzyme levels will usually, but not always, go back to normal
within 90 days of someone stopping use all on their own.
Just because oral steroids "allegedly" cause transaminase levels to rise
a bit DOES NOT MEAN that the supplements you buy and use are going
to reverse this (even if a bunch of good bros here who mean well are
repeating unsubstantiated 'advice' they heard here or read over 'there'.
There is no proof...even of the really "stretched and embellished" proof
that these liver "protectors" will protect against a rise in LFT's related
to methyl anabolic oral use.
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09-29-2006, 09:48 PM #16
Anabolic steroid-induced hepatotoxicity: is it overstated?
Researchers: Dickerman RD, Pertusi RM, Zachariah NY, Dufour DR, McConathy WJ
The Department of Biomedical Science, University of North Texas Health Science Center, Fort Worth 76107-2699, USA.
Source: Clin J Sport Med 1999 Jan;9(1):34-9
Summary:
Subjects: The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and non-exercising medical students (592) were used as controls.
Measurements: The focus of the blood chemistry profiles was on aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels. (All indicators of liver function.)
Results: In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT.
OBJECTIVE: There have been numerous reports of hepatic dysfunction secondary to anabolic steroid use based on elevated levels of serum aminotransferases. This study was conducted to distinguish between serum aminotransaminase elevations secondary to intense resistance training and anabolic steroid -induced hepatotoxicity in elite bodybuilders. DESIGN: This was a case-control study of serum chemistry profiles from bodybuilders using and not using anabolic steroids with comparisons to a cohort of medical students and patients with hepatitis. PARTICIPANTS: The participants were bodybuilders taking self-directed regimens of anabolic steroids (n = 15) and bodybuilders not taking steroids (n = 10). Blood chemistry profiles from patients with viral hepatitis (n = 49) and exercising and nonexercising medical students (592) were used as controls. MAIN OUTCOME MEASURES: The focus in blood chemistry profiles was aspartate aminotransferase (AST), alanine aminotransferase (ALT), gamma-glutamyltranspeptidase (GGT), and creatine kinase (CK) levels. RESULTS: In both groups of bodybuilders, CK, AST, and ALT were elevated, whereas GGT remained in the normal range. In contrast, patients with hepatitis had elevations of all three enzymes: ALT, AST, and GGT. Creatine kinase (CK) was elevated in all exercising groups. Patients with hepatitis were the only group in which a correlation was found between aminotransferases and GGT. CONCLUSION: Prior reports of anabolic steroid-induced hepatotoxicity based on elevated aminotransferase levels may have been overstated, because no exercising subjects, including steroid users, demonstrated hepatic dysfunction based on GGT levels. Such reports may have misled the medical community to emphasize steroid-induced hepatotoxicity when interpreting elevated aminotransferase levels and disregard muscle damage. For these reasons, when evaluating hepatic function in cases of anabolic steroid therapy or abuse, CK and GGT levels should be considered in addition to ALT and AST levels as essential elements of the assessment.
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11-04-2006, 10:39 PM #17New Member
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11-08-2006, 06:05 PM #18Junior Member
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whats a good mg to take daily?... 500? 1000?
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