I think you over evaluate what we actually have proved by studies on AAS.
When they began developing synthetic versions of testosterone and saw they behaved differently than testosterone in various ways, chemist developed 100s of steroids, most of which were never studied in humans.
AR affinity in itself has also proved to be more difficult to measure when studies revealed that agents with low affinity for the androgen receptor still caused a significant increase in AR mediated gene regulation.
There's also a lot more to AAS and AR mediated myogenesis.
Like systemic IGF1 secretion, local IGF1 turnover and on EPO mediated effects, etc.
When different AAS were developed they tried them in several different conditions. Yes Deca can be used in burn victims, so can many other AAS. Deca is also used for osteoporosis in women.
Stuff like TBOL were an exception, as it was actually designed (or taken into use) for athletic improvement. But this was under a regime that had a state sponsored doping plan. Other AAS has also only been used for athletic performance, but this is either because they were AAS that didn't have any medical use and has advantages when it came to testing.
THG is perhaps the only steroid that we know was developed with the intention for performance use and beating drug tests.
Anyways, intresting take you have, but even if you just look at the muscle tissue and says deca is enough, you're neglecting all the other physiological effects T has in the body.
We should make a new thread.
This was intresting.
Originally Posted by MuscleScience
Yeah, I had some free time and free rain of the library when I was in grad school so I would jump onto the chem and medical journal databases and look up a lot of info on all the different compounds. All these drugs were designed for a purpose. T-bol for athletes, deca for burn victims and bone mineral loss, sustason for muscle wasting ext.
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