New Member
This is my last post on this website so plz read and answer thanks
i have posted a thread be4
i have 1 last question
if my growth plates are closed cuz of takin steriods at age 16
and my frame aint wide if i carried on takin steriods and trainin
would my body frame my shoulders etc. like wider frame? still expand? like widen not grow in height
thanks
Educate B4 You Medicate~HOF~RIP Our Brother~
We can't tell you that.
Only the endo doc will be able to answer that
you can get wider if you get FAT
some of us (myself included) are members at our sister site
www.probodybuilding.com
you can register there and still get your questions answered, but there is NO steroid talk on that board
Banned
its been good old friend. best of luck in your future endeavors.
AR-Elite Hall of Famer (RETIRED)
AR's Hot British Pimp Daddy ~HOF~
Yep, see ya..
Banned
see you later (in the lounge).Originally Posted by MAD MATT
Senior Member
I hope it is.
Anabolic Member
No, most likely you will have stunted full growth in all plates including the ones in your shoulder.
That is because your plates in the knee are directly porportional to your shoulder width frame.
For example, if you stretch your arms out as wide as possible and measure it from fingertip to fingertip you will get your height.
So if your growth plates have fused, they will have fused all over your body.
Last edited by Immortal Soldier; 04-04-2009 at 07:10 PM.
"3 stars and a sun"
bye!
Associate Member
Just go and see a doctor kid.
Tell him what you have been doing and find out for sure.
Best of luck
Anabolic Member
Your not supposed to use steroids till you are 17!
"T-MOS WILL LIVE THROUGH US FOREVER"
Bye
"3 stars and a sun"
New Member
Damn dude...i have posted a thread be4
i have 1 last question
if my growth plates are closed cuz of takin steriods at age 16
and my frame aint wide if i carried on takin steriods and trainin
would my body frame my shoulders etc. like wider frame? still expand? like widen not grow in height
thanks
Junior Member
dont they have D.A.R.E. at your school..........
you should have just said NO!
Junior Member
way too young...
Junior Member
besides, at age 16 you can get great results without steroids. if i only knew that then i would have been more serious.
Knowledgeable Member
Gee, do you think Mike111 should read this? I dont think it would matter though.
Associate Member
Indeed ... dont think that would make him change his ideas ... immature fior life ...Gee, do you think Mike111 should read this? I dont think it would matter though.
Anabolic Member
No, epiphyseal fusion occurs in long bones, even if you hadn't taken steroids you will not get wider bone structure. IE there aren't growth plates in you're shoulders..
Copyright © 2001, The National Academy of Sciences
Medical Sciences
Effects of estrogen on growth plate senescence and epiphyseal fusion
In mammals, longitudinal bone growth occurs at the growth plate by endochondral bone formation. The growth plate consists of three principal zones: resting, proliferative, and hypertrophic. The resting zone lies adjacent to the epiphyseal bone and contains infrequently dividing chondrocytes. The proliferative zone contains replicating chondrocytes arranged in columns parallel to the long axis of the bone. The proliferative chondrocytes located farthest from the resting zone stop replicating and enlarge to become hypertrophic chondrocytes. These terminally differentiated cells maintain a columnar alignment in the hypertrophic zone. The processes of chondrocyte proliferation, hypertrophy, and cartilage matrix secretion result in chondrogenesis. Simultaneously, the metaphyseal border of the growth plate is invaded by blood vessels and bone cell precursors that remodel the newly formed cartilage into bone. The synchronized processes of chondrogenesis and cartilage ossification lead to longitudinal bone growth.
With increasing age, the growth plate undergoes structural and functional changes. The rate of longitudinal bone growth decreases, in large part, because of a decline in chondrocyte proliferation. These functional senescent changes are accompanied by structural senescent changes. There is a gradual decline in the overall growth plate height, proliferative zone height, hypertrophic zone height, size of hypertrophic chondrocytes, and column density.
In some mammals, including humans, the growth plate is resorbed at the time of sexual maturation. This process, epiphyseal fusion, terminates longitudinal bone growth. Estrogen is pivotal for epiphyseal fusion in both young men and women. This key role for estrogen was confirmed only recently with the recognition of two genetic disorders, estrogen deficiency due to mutations in the aromatase gene and estrogen resistance due to mutations in the estrogen receptor-a gene. In both conditions, the growth plate fails to fuse and growth persists into adulthood. Conversely, premature estrogen exposure, e.g., precocious puberty, leads to premature epiphyseal fusion.
The mechanism by which estrogen promotes epiphyseal fusion is not known. Previous reports suggest that estrogen accelerates growth plate ossification by stimulating vascular and bone cell invasion of the growth plate cartilage, causing ossification to advance beyond the hypertrophic zone into the proliferative and resting zones. This proposed mechanism of estrogen action would be expected to induce epiphyseal fusion promptly, a prediction that does not match clinical experience. Prompt fusion occurs only in estrogen-deficient adults treated with estrogen. In children, epiphyseal fusion occurs only after years of estrogen exposure. The accelerated ossification hypothesis does not readily explain this delayed action.
Take care of you're body, don't abuse it.
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