PRIVORON-For a healthier and recuperated hormone panel?

[oscar1990]

Hi there bros, in the past I have had chronic appendicitis which was undiagnosed for quite a long period of time. I was mis diagnosed with several conditions (lol).

Now, 2.5 years later.. I have found that my body has not re-adjusted itself and fully recovered. I do believe my hormones were on a havoc whilst being ill thus suppressing my HPTA quite dramatically.

It has been 4 months after surgery and nothing has changed. My physical strength, stamina and resilience has declined and is continuing to do so. My body has increase in BF%, lowered sex drive, no wood, lowered muscle density and size, and finally poor sleep quality! My quality of life is slowly but steadily also plummeting.

I do want to run a Privoron cycle as I do believe it will help regulate my hormone ratios with little damage done to the HPTA. I will also be adding GHRP-6 to Privoron.

I have heard alot of bro's running Privoron PCT and reaping all the benefits from it.

What is your advice for me as a young 22 year old running Proviron and what should I expect...? Will it assist in recuperation?

Cheers

[oscar1990]

In addition, do not tell me to go to a doctor as I have had an undiagnosed appendicitis, yes an undiagnosed appendicitis. what the **** are the chances of a doctor diagnosing a hormonal problem and will probably just brush it off as 'normal' lol

[oscar1990]

waiting patiently... sweet

[lovbyts]

Well over on this side of the map it's midnight so it may take a while for someone to respond who has experience with Privoron. Sorry I dont.

[oscar1990]

Cheers lovbyts

[oscar1990]

bump

[lovbyts]

Only 4:30 am lol

[oscar1990]

u.s or canada?

[kelkel]

Proviron will be a total waste of time and money, IMHO, unless your looking to maybe suppress your SHBG level in conjunction with other anabolics. Read this:



"Here is a post cut strait from the keyboard of a leading doctor (Endocrinologist) that treats hypogonadal men, his name is Dr. Michael Scally:


''Dianabol (methandione, methandrostenolone , metandienone, and a host of other names) suppresses the HPTA. The use of dianabol in the hope that it will provide HPTA normalization is misguided. More details, later, can be provided, if requested.

However, a brief note on proviron . What evidence is there that proviron lacks androgenic activity. The literature presents this by the absence of proviron to influence significantly infertility, erythropoiesis, lipids, and sex hormones. Except for the obsessive compulsive that needs to take a substance, thus replacing an AAS with adverse HPTA effects with one that does not, proviron is a worthless AAS, useful for nothing. Proviron will not support or provide any basis for the return of HPTA function.

The quoted abstract from the study by Varma and Patel really does not give one any information. [Varma TR, Patel RH. The effect of mesterolone on sperm count, on serum follicle stimulating hormone, luteinizing hormone, plasma testosterone and outcome in idiopathic oligospermic men. Int J Gynaecol Obstet 1988;26:121-8.] The study is poor from the abstract alone. Please note that the statement, "Mesterolone had no depressing effect on low or normal serum FSH and LH levels but had depressing effect on 25% if the levels were elevated," refers unidentified group. The groups in the study include, "One hundred ten patients . . . had normal serum FSH, LH and plasma testosterone, 85 patients . . . had low serum FSH, LH and low plasma testosterone." Nowhere is there a group with elevated levels. Nonetheless, the cited effect is a "depressing effect" not stated as significant. Knowing the fluctuation in gonadotropin levels on testing even at a P<0.05 would not be meaningful. But it does go to the point that proviron has no adverse effect on the HPTA.

Mesterolone is useless for infertility. A year after the Varma study, 1989, the World Health Organization published a study demonstrating, "[n]o significant changes semen quality during the course of the study, apart from an increase in sperm concentration 3 months after the start of treatment. The increase was greatest among the placebo treated group, but did not differ significantly between treatment groups." [Mesterolone and idiopathic male infertility: a double-blind study. World Health Organization Task Force on the Diagnosis and Treatment of Infertility. Int J Androl 1989;12:254-64.]

In 1991, a study concludes, "Because similar semen improvement also occurred in the placebo controls, our findings cast doubt on the possible usefulness of high-dose Mesterolone treatment of idiopathic male infertility." [Gerris J, Comhaire F, Hellemans P, Peeters K, Schoonjans F. Placebo-controlled trial of high-dose Mesterolone treatment of idiopathic male infertility. Fertil Steril 1991;55:603-7.]

These confirm an earlier study from 1983. [Wang C, Chan CW, Wong KK, Yeung KK. Comparison of the effectiveness of placebo, clomiphene citrate, mesterolone, pentoxifylline, and testosterone rebound therapy for the treatment of idiopathic oligospermia. Fertil Steril 1983;40:358-65.] Treatment with the mesterolone (100 mg/day) therapy did not result in a significant increase in the mean sperm concentration or pregnancy in the partners.

Proviron is useless in promoting erythropoiesis (formation of red blood cell elements) and bone formation (a mixed effect of testosterone through the androgen receptor and estradiol receptor), both evidence of androgenic activity. Mesterolone (100 mg/d) is ineffective in raising hemoglobin and hematocrit levels significantly from baseline in individuals with hypogonadism. The study cites that Mesterolone did not increase serum testosterone (but also did not mention that there is a decrease). [Jockenhovel F, Vogel E, Reinhardt W, Reinwein D. Effects of various modes of androgen substitution therapy on erythropoiesis. Eur J Med Res 1997;2:293-8.]

As recent as 2003, mesterolone (100 mg/d) for 6 months administered to hypogonadal males failed to significantly raise bone mineral density (BMD). Treatment with testosterone undecanoate (160 mg/d), testosterone enanthate 250 mg (every 21 days), or a single subcutaneous implantation of 1,200 mg crystalline testosterone did result in BMD increases. [Schubert M, Bullmann C, Minnemann T, Reiners C, Krone W, Jockenhovel F. Osteoporosis in male hypogonadism: responses to androgen substitution differ among men with primary and secondary hypogonadism. Horm Res 2003;60:21-8.]

Erythropoiesis and bone formation are positive aspects of androgens useful under certain clinical conditions. AAS consistently have adverse effects on lipid profiles that are generally observed as a decrease in HDL (good cholesterol). In 1999, twenty years after the study cited by MaxRep [Nikkanen V. Plasma cholesterol, triglycerides, FSH and testosterone levels of normolipemic male patients with decreased fertility treated with mesterolone. Andrologia 1979;11:33-6.] proviron was found to adversely effect the lipid profile in hypogonadal men. The study by abstract analysis is hard to detail but an adverse effect of proviron is reported. Also, the study reports on serum testosterone levels with androgen treatments. Androgen substitution led to no significant increase of serum testosterone in the proviron group, subnormal testosterone in the testosterone undecanoate group, normal testosterone in the testosterone enanthate group, and high-normal testosterone in the crystalline testosterone group. The message is proviron did not affect the HPTA. [Jockenhovel F, Bullmann C, Schubert M, et al. Influence of various modes of androgen substitution on serum lipids and lipoproteins in hypogonadal men. Metabolism 1999;48:590-6.] The same author reports that proviron administration has no effect on serum FSH or testosterone. [Nikkanen V. The effects of mesterolone on the male accessory sex organs, on spermiogram, plasma testosterone and FSH. Andrologia 1978;10:299-306.]

I have said too much already. A further review of proviron literature will not change the use of proviron as an AAS for either anabolic or androgenic effects. Bottom line: Proviron is of no use for anything.

Thank you.''

Dr. Michael Scally

******

Hope that helps a bit....

[oscar1990]

Cheers Kelkel,

Im looking to solely free up some test without damaging the HPTA. I do not want to use privoron with any AAS as I am not ready for AAS.

There is alot of controversial journals/abstracts/experience with privoron, many are suggesting it works great for libido/drying up/leaning down. Another group suggesting it doesn't do much and will present it self with many symptoms.

[kelkel]

Add Vit D3 to your daily protocol. Everyone is low and D3 will reduce your SHBG level thus allowing more free Test. Test your current level. Maybe start with 5k IU per day...

http://www.ncbi.nlm.nih.gov/pubmed/18400738

http://www.ncbi.nlm.nih.gov/pubmed/20050857

[oscar1990]

Human growth hormone and prolactin are
evolutionarily related, and SHBG levels are
similarly low in acromegaly and patients receiving growth hormone treatments. The SHBG level
is likely to be abnormal in male hypogonadism,
androgen insensitivity, thyroid disorders, diabetes
mellitus, and liver disease.
5

http://www.medical.siemens.com/sieme...ts/zb170-b.pdf

Kelkel, would you say that I am on my way to andropause at a very early age due to my high SHBG? Does this mean I will need to seek HRT?

My SHBG is within range but rising for some unknown reason?

What would you think based on prima facie?

[oscar1990]

you dudes are all asleep, bad timing

[kelkel]

Oscar can you post up recent BW with ranges. It would help alot. High shbg can be caused by many things such as thyroid, insulin or estrogen to name a few. The quickest way to reduce shbg is to elevate your test level.

http://www.ncbi.nlm.nih.gov/pubmed/2080856

[oscar1990]

Kelkel, ill post my most recent blood work (from 3-4 months ago). In addition, I will get my GP to run some tests which include: TT, SHBG, LH, FSH, E2 Sensitive Assay, DHEA, DHT (I dont think they test DHT in AUS, will have to check), and TSH. Will post results hopefully by Wednesday/Thursday.

[kelkel]

Nice. DHEA-S is what you want.