Things the medical companies doesnt want us to know

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http://www.orthomed.com/polio.htm

vitamin c must be the most overlooked thing. yes we all supplement a bit with it. But consider the regular pig produces 10 grams a day!! of it while we think we can manage on a couple of hundrad mg's. Im gonna experiment with 15-20grams of vitamin c now daily while I have viral tonsilitis to se if it goes away quickly. I have had it for a week now without any signs of improving(went to the doc today he said Il just have to wait).

On orthomed.com a guy seems to think vitamin c in massive doses can be the cure for many viral disease so I guess it wont hurt to try.

here is a article on how they cured polio with it(before the vaccin way back in 1949).

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The Treatment of Poliomyelitis and Other Virus Diseases with Vitamin C

Fred R. Klenner, M.D., Reidsville, North Carolina

IN A PREVIOUS REPORT dealing with the antagonistic properties of ascorbic acid to the virus of atypical pneumonia, mention was made of the fact that other types of virus infections had responded favorably to vitamin C. This paper is to present these findings as well as the results of subsequent studies on the virus of poliomyelitis, the viruses causing measles, mumps, chickenpox, herpes zoster, herpes simplex and influenza. Further studies with the virus of atypical pneumonia will also be discussed.

These observations of the action of ascorbic acid on virus diseases were made independently of any knowledge of previous studies using vitamin C on virus pathology, except for the negative report of Sabin after treating Rhesus monkeys experimentally infected with the poliomyelitis virus. A review of the literature in preparation of this paper, however, presented an almost unbelievable record of such studies. The years of labor in animal experimentation, the cost in human effort and in "grants," and the volumes written, make it difficult to understand how so many investigators could have failed in comprehending the one thing that would have given positive results a decade ago. This one thing was the size of the dose of vitamin C employed and the frequency of its administration. In all fairness it must be said that Jungeblut noted on several occasions that he attributed his failure of results to the possibility that the strength of his injectable "C" was inadequate. It was he who unequivocally said that ''vitamin C can truthfully be designated as the antitoxic and antiviral vitamin."

In developing this paper it was felt that, since all virus infections were more or less akin, only one of this family would be considered in detail. Poliomyelitis, because of its prevalence and the seriousness of the problem it presents, was chosen as the disease to be so treated.

Poliomyelitis is in most instances an acute febrile disease of sudden onset, with symptoms of a systemic infection which either abruptly abort or develop to hyperesthesia, asymmetry of reflexes and flaccid paralysis or palsies of muscle groups. It affects individuals of all ages, but mainly children, as do more common childhood diseases to which class it most likely belongs. Only slight contact between the carrier of the virus and the susceptible person suffices in some cases for the transfer of the causative organism. In this respect and also in that the virus can be demonstrated in the nasal washings as early as six days before onset of symptoms, poliomyelitis resembles measles. We never have an epidemic of poliomyelitis preceding an epidemic of measles; the opposite is frequently true. This grouping of the virus organisms is too often repeated not .to carry some significance. For example, atypical pneumonia and influenza are caused by closely allied viruses; so are chickenpox, herpes zoster and herpes simplex; so are measles, mumps and poliomyelitis. The incubation period depends on the mode of entry. In experimental animals. Fraser and others showed that the average was 6.6 days with intracerebral inoculation and ten days when the intravenous route was used. Howitt mentions that the virus reaches the nervous system sooner after intranasal than after intravenous instillations. Transmission (Brodie, 1934) is by means of droplets from the mucous membrane of the upper respiratory tract. Infection by means of raw milk, human feces and house flies is highly improbable.

The research of Flexner, Dark and Amoss in 1914 proved that poliomyelitis is a disease of the entire nervous system, that the sensory ganglia are the seats of early and profound histological changes. The disease is significant mainly for the paralysis produced through injury to the motor neurons of the spinal cord and brain. This is caused by a special affinity of the virus for a certain type of nerve tissue. Experiments show the cerebral cortex to be the most unsatisfactory site for growth, that large amounts of the virus placed in this area are apt to disappear in a short time. Observations in monkeys and in man show that the anterior horn cells, particularly those of the lumbar cord, are the most favorable sites for proliferation of the virus.

In all clinically ill patients the virus eventually travels in the course of its invasion by several channels. The virus can make a direct assault through the olfactory bulb, to the brain, medulla and spinal cord. The virus can enter the blood stream directly or through the lymph channels. Following damage to the natural protective barrier, the choroid plexus, it can make its way to the central nervous system, or it can be excreted back onto the nasal mucous membrane where it will pick up the direct route of the olfactory bulb.

Clark, Turner and Reynolds (1926, 1927, 1929) concluded that the virus chiefly travels by the direct route to the brain. Lennette and Hudson ( 1935) confirmed this theory and reported their studies indicating that human infection is chiefly through the nasopharynx. Brodi and others showed that by section of the olfactory tracts in monkeys infection by the direct route was prevented. It is of more than mere academic interest that while the nasal mucosa of the monkey contains branches of the 5th and 7th cranial nerves and that in addition, since the virus can readily gravitate from the nasopharynx to the tonsil bed with its nerve supply, if the olfactory tracts are cut no infection will occur. The most likely explanation is that the olfactory is non-medullated, the neurons lie in the nasal mucosa and are thus exposed to the virus. The sciatic nerve (Brodi) will transport the virus only when it has been injured, suggesting that lack of myelin may render the healthy olfactory nerve vulnerable to the virus.

The most important of the secondary routes of infection is by the excretion of the virus from the blood stream onto the nasal mucosa. Lennette and Hudson (1934, 1935) demonstrated in monkeys that by sectioning the olfactory tracts and then inoculating by the intravenous route with the virus of poliomyelitis, they could prevent infection.

This would fit in with the work of Jungeblut and others that the spread of the virus through the central nervous system is along nerve tracts, rather than by means of the cerebrospinal fluid, the infection to become manifest when the first cell group is reached, and by relays of fibers, reaches the mid-brain. Here numerous fiber-paths run in all directions and the virus is carried by both motor and sensory axons, causing disease at many levels of the brain and cord.

Since there is always a period of septicemia in the first few days of poliomyelitis, it might be that this is the all-important route and that the virus is grown on a living tissue, the blood, and then is deposited out on the surface of the olfactory bulb. From this we conclude that the time to destroy the virus is during this incubation period which varies more with virulence and power of multiplication than with size of initial dose.

The second flanking maneuver of importance is through the choroid plexus. It is the function of the choroid plexus and the pial lymphatic vessels to exclude the virus present in the blood from the nervous system. Once these protective structures are injured, however, the exclusion ceases and infection can follow readily. Changes in the structure or function of the meningeal choroid plexus complex, too slight to be detected in the cerebrospinal fluid or as morphological alterations, materially diminish its protective power. Flexner and Amoss injected large doses of the virus intravenously, then tested the cerebrospinal fluid and found no virus after the first 48 hours; virus in small amounts at the end of 72 hours; after 96 hours evidence of free access to this system. The virus was still present 19 days later when paralysis was beginning.

Poliomyelitis in man is always more severe if exercise is taken at time of the infection. Here one must consider the factor of filtration of the virus through the choroid plexus as being increased due to the elevation of the vascular bed pressure. Also, that, by the acceleration of the blood flow caused by greater oxygen demand in physical effort, a marked increase in the percentage of the virus deposited on the nasal mucosa would result.

We must agree with Fairbrother and Hurst that too little consideration has been given to the pathology of the nervous system and in particular to the drainage of the tissue fluids. These men confirmed the earlier work of Schroder, who stressed that the normal flow of these fluids is along the perivascular spaces from the center of the cord outward, and that any inflammatory exudate occupying these spaces must be swept into the pial meshes; further that meningeal infiltration may seem nothing more than a drainage of cells from the interior of the cord. Fairbrother and Hurst found that meningeal infiltration does not occur in monkeys until the perivascular infiltration beginning in the deeper vessels reaches the surface.

The presence of the filterable microorganism or virus of poliomyelitis upon the mucous membrane of the nose and throat does not necessarily lead to infection. It may give rise to a class of healthy carriers who are themselves immune. Amoss and Taylor found a secretion of the mucous membrane capable of neutralizing or inactivating the virus, this property absent altogether from the secretions of some persons, in those of others present at one time and not at another. It is probable that in actively immune animals the passage of the neutralizing substance from the blood into the cerebrospinal fluid would continue as long as the inflammation present in the meninges rendered the structures easily permeable to the protein constituents of the blood. This secretion X could not have the properties of a true antibody. The virus of poliomyelitis is intracellular from the time it invades the terminal cells of the olfactory system until the end of the disease, except when crossing the synaptic junctions between cells. This explains why the virus cannot be neutralized by antibodies in the serum. Further protection is afforded the virus by the functional barrier between the circulating blood and the central nervous system.

Since immunization against poliomyelitis comparable to that against other bacterial diseases is still a matter of the future, it suggested itself that some antibiotic could be found that would destroy this scourge while in the phase of blood-stream invasion. Sabin's negative report on the value of ascorbic acid on the poliomyelitis virus stopped Jungeblut's work, but we were cognizant of its dramatic effect on the virus causing atypical pneumonia, and so kept up hope. These results were so consistently positive that we did not hesitate to try its effectiveness against all type of virus infections. The frequent administration of massive doses of vitamin C was so encouraging in the early days of the 1948 epidemic of poliomyelitis that a review of the literature was begun. Heaslip, in the Australian Journal of Experimental Biology & Medicine reported a mean urinary output of vitamin C under a load test of 19.9 per cent in 60 poliomyelitis cases, as contrasted with a mean figure of 44.3 per cent in 45 healthy contacts. This was suggestive of some relationship between the degree of vitamin C saturation and the infectious and non-infectious state. He was also able to show a correlation between the severity of the attack and the level of urinary excretion of the vitamin. This would indicate that a deficiency of vitamin C in the diet predisposed to infection and to severity of attack. Sabin reported no appreciable difference in infectivity of poliomyelitis in monkeys with much or no vitamin C in the diet. Many others, however, have reported that a "deficient vitamin C nutrition increases susceptibility to infection," and many others that animals dying from the effects of the poliomyelitis virus show a reduction of vitamin C in the tissues. Heaslip found a definite relationship between the severity of the infection and the level of vitamin C nutrition. It is consistent with accepted physiological action of vitamin C to expect and anti-edema effect in any given affected area. It is worthy of note that bacterial toxins can cause losses of from 50 to 85 per cent of the vitamin C normally contained in the adrenals. Jungeblut's investigations seemed to justify the conclusion that vitamin C was the "antibiotic" that would destroy the virus organism. He stated that the prophylactic and therapeutic administration of synthetic or natural vitamin C had given evidence of having distinct therapeutic properties in experimental poliomyelitis, and that the proper injection dose was directly proportional to the speed of the infection and the stage at which the process had arrived. Jungeblut stated in 1937 that the parental administration of natural vitamin C during its incubation period of poliomyelitis in monkeys is always followed by a distinct change in the severity of the disease; that after the fifth day of the disease distinctly larger doses are required. He realized, at that early date, that for a fast progressing infection such as results from the R. M. V. strain, very large doses—400 mg. crystalline C maximum in a 24-hour period—of vitamin C would be required; for the Aycock virus with its slower infection potential small amounts of the vitamin would suffice. Even with almost infinitesimal amounts—100 mg. ascorbic acid for each 24-hour period—he was able to demonstrate that the non-paralytic survivors in one series was six times as great as in the controls. In our work we shall speak of six, ten and 20 thousand mg. in a similar time period.

Harde et al. reported that diphtheria toxin is inactivated by vitamin C in vitro and to a lesser extent in vivo. I have confirmed this finding, indeed extended it. Diphtheria can be cured in man by the administration of massive frequent doses of hexuronic acid (vitamin C) given intravenously and/or intramuscularly. To the synthetic drug, by mouth, there is little response, even when 1000 to 2000 mg. is used every two hours. This cure in diphtheria is brought about in half the time required to remove the membrane and give negative smears by antitoxin. This membrane is removed by lysis when "C" is given, rather than by sloughing as results with the use of the antitoxin. An advantage of this form of therapy is that the danger of serum reaction is eliminated. The only disadvantage of the ascorbic acid therapy is the inconvenience of the multiple injections. This concept of the action of vitamin C against certain toxins has led to treating other diseases producing exotoxins. For years it has been our knowledge that vitamin C in 500 to 1000 mg. doses injected I. M. would cure bacillary dysentery of the Shiga type. Children having 10 to 15 bloody stools per day have cleared in 48 hours under this schedule while at the same time reverting to normal feedings. This dual action of vitamin C against certain toxins and the virus organism becomes more intelligible with the work of Kligler, Warburg and others who believed that the detoxification effected by hexuronic acid is brought about by a direct combination of the vitamin with the toxin or virus, this followed by oxidation of the new compound which destroys both the virus or toxin and the vitamin. Borsook et al. decided that the main chemical action of ascorbic acid is as a powerful reducing agent, and the virus causing poliomyelitis is known to be susceptible to the oxidizing action of various agents. It is in point here to remark that vitamin C is an integral part of the oxidation-reduction system of the body, thus playing a definite part in natural resistance.

In the poliomyelitis epidemic in North Carolina in 1948, 60 cases of this disease came under our care. These patients presented all or almost all of these signs and symptoms: Fever of 101 to 104.6°, headache, pain at the back of the eyes, conjunctivitis, scarlet throat; pain between the shoulders, the back of the neck, one or more extremity, the lumbar back; nausea, vomiting and constipation. In I5 of these cases the diagnosis was confirmed by lumbar puncture; the cell count ranging from 33 to 125. Eight had been in contact with a proven case; two of this group received spinal taps. Examination of the spinal fluid was not carried out in others for the reasons: (1) Flexner and Amoss had warned that "simple lumbar puncture attended with even very slight hemorrhage opens the way for the passage of the virus from the blood into the central nervous system and thus promotes infection." (2) A patient presenting all or almost all of the above signs and symptoms during an epidemic of poliomyelitis must be considered infected with this virus. (3) Routine lumbar puncture would have made it obligatory to report each case as diagnosed to the health authorities. This would have deprived myself of valuable clinical material and the patients of most valuable therapy, since they would have been removed to a receiving center in a nearby town.

The treatment employed was vitamin C in massive doses. It was given like any other antibiotic every two to four hours. The initial dose was 1000 to 2000 mg., depending on age. Children up to four years received the injections intramuscularly. Since laboratory facilitates for whole blood and urine determinations of the concentration of vitamin C were not available, the temperature curve was adopted as the guide for additional medication. The rectal temperature was recorded every two hours. No temperature response after the second hour was taken to indicate the second 1000 or 2000 mg. If there was a drop in fever after two hours, two more hours was allowed before the second dose. This schedule was followed for 24 hours. After this time the fever was consistently down, so the drug was given 1000 to 2000 mg. every six hours for the next 48 hours. All patients were clinically well after 72 hours. After three patients had a relapse the drug was continued for at least 48 hours longer—1000 to 2000 mg. every eight to 12 hours. Where spinal taps were performed, it was the rule to find a reversion of the fluid to normal after the second day of treatment.

For patients treated in the home the dose schedule was 2000 mg. by needle every six hours, supplemented by 1000 to 2000 mg. every two hours by mouth. The tablet was crushed and dissolved in fruit juice. All of the natural "C" in fruit juice is taken up by the body; this made us expect catalytic action from this medium. Ruin, 20 mg., was used with vitamin C by mouth in a few cases, instead of the fruit juice. Hawley and others have shown that vitamin C taken by mouth will show its peak of excretion in the urine in from four to six hours. Intravenous administration produces this peak in from one to three hours. By this route however, the concentration in the blood is raised so suddenly that a transitory overflow into the urine results before the tissues are saturated. Some authorities suggest that the subcutaneous method is the most conservative in terms of vitamin C loss but this factor is overwhelmingly neutralized by the factor of pain inflicted.

Two patients in this series of 60 regurgitated fluid through the nose. This was interpreted as representing the dangerous bulbar type. For a patient in this category postural drainage, oxygen administration, in some cases tracheotomy, needs to be instituted, until the vitamin C has had sufficient time to work—in our experience 36 hours. Failure to recognize this factor might sacrifice the chance of recovery. With these precautions taken, every patient of this series recovered uneventfully within three to five days.

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In the treatment of other types of virus infections the same "fluid" dose schedule was adopted. In herpes zoster 2000 to 3000 mg. of vitamin C was given every 12 hours, this supplemented by 3 000 mg. in fruit juice by mouth every two hours. Eight cases were treated in this series, all of adults. Seven experienced cessation of pain within two hours of the first injection and remained so without the use of any other analgesic medication. Seven of these cases showed drying of the vesicles within 24 hours and were clear of lesions within 72 hours. They received from five to seven injections. One patient; a diabetic, stated that she was always conscious of an uncomfortable feeling, but that it was not an actual pain. Although nine-tenths of the vesicles cleared in the usual 72-hour period, she was given 14 injections, the last seven of only 1000 mg. This extra therapy was given because of a small ulceration, an inch in diameter, secondarily infected by rupture of the vesicles by a corset stave prior to the first visit. Vitamin C apparently had no effect on this lesion, which was healed in two weeks under compound tincture of benzoin locally and penicillin and sulfadiazine by mouth. (The patient objected to taking penicillin by needle.) One of the patients, a man of 65, came to the office doubled up with abdominal pain and with a history of having taken opiates for the preceding 36 hours. He gave the impression of having an acute surgical condition. A massive array of vesicles extended from the dorsal nerve roots to the umbilicus, a hand's breadth wide. He was given 3000 mg. of vitamin C intravenously and directed to return to the office in four to five hours. It was difficult to convince him that his abdominal pain was the result of his having "shingles." He returned in four hours completely free of pain. He was given an additional 2000 mg. of vitamin C, and following the schedule given above he recovered completely in three days.

In herpes simplex it is important to continue the treatment for at least 72 hours. We have seen "fever blisters" that appeared healed after two injections recur when therapy was discontinued after 24 hours. Vitamin C in a strength of 1000 mg. per 10 c.c. of buffered solution gave no response when applied locally. This was true no matter how often the applications were made. In several cases 10 mg. of riboflavin by mouth t.i.d. in conjunction with the vitamin C injections appeared to cause faster healing.

Chickenpox gave equally good response, the vesicles responding in the same manner as did those of herpes. These vesicles were crusted after the first 24 hours, and the patient well in three to four cays. We interpreted this similarity of response in these three diseases to suggest that the viruses responsible were closely related to one another.

Many cases of influenza were treated with vitamin C. The size of the dose and the number of Injections required were in direct proportion to the fever curve and to the duration of the illness. Forcing of fruit juice was always recommended, because of the frequency and ease of reinfection during certain periods of the year.

The response of virus encephalitis to ascorbic acid therapy was dramatic. Six cases of virus encephalitis were treated and cured with vitamin C injections. Two cases were associated with virus pneumonia; one followed chickenpox, one mumps, one measles and one a combination of measles and mumps. In the case that followed the measles-mumps complex, definite evidence was found to confirm the belief that massive, frequent injections are necessary in treating virus infections with vitamin C. This lad of eight years was first seen with a temperature of 104°. He was lethargic, very irritable when molested. His mother said he had gradually developed his present clinical picture over the preceding four or five days. His first symptom was anorexia which became complete 36 hours before his first examination. He next complained of a generalized headache, later be became stuporous. Although very athletic and active, he voluntarily took to his bed. He was given 2000 mg. of vitamin C intravenously and allowed to return home because there were no available hospital accommodations. His mother was asked to make an hourly memorandum of his conduct until his visit set for the following day. Seen 18 hours after the initial injection of vitamin C, the memorandum revealed a quick response to the antibiotic—after two hours he asked for food and ate a hearty supper, then played about the house as usual and then, for .several hours, he appeared to have completely recovered. Six hours following the initial injection, he began to revert to the condition of his first visit. When seen the second time temperature was 101.6°, he was sleepy but he would respond to questions. The rude irritability shown prior to the first injection was strikingly absent. A second injection of 2000 mg. vitamin C was given intravenously and 1000 mg. of "C" prescribed every two hours by mouth. The next day he was fever and symptom-free. As a precautionary measure a third 2000 mg. was given with direction to continue the drug by mouth for at least 48 hours. He has remained well since. A lad of 12 years had generalized headache a week after having mumps, this followed by malaise, and in 12 hours a lethargic state and a fever of 105°. Admitted to hospital he was given 2000 mg. of vitamin C then, and 1000 mg. every two hours. Following the third injection he was sitting up in bed, laughing, talking, begging for food and completely without pain. He was discharged 24 hours following admission clinically well. Since relapses do occur if the drug is discontinued too soon, he was given 2000 mg. of vitamin C every 12 hours for two additional days.

The use of vitamin C in measles proved to be a medical curiosity. During an epidemic vitamin C was used prophylactically and all those who received as much as 1000 mg. every six hours, by vein or muscle, were protected from the virus. Given by mouth, 1000 mg. in fruit juice every two hours was not protective unless it was given around the clock. It was further found that 1000 mg. by mouth, four to six times each day, would modify the attack; with the appearance of Koplik's spots and fever, if the administration was increased to 12 doses each 24 hours, all signs and symptoms would disappear in 48 hours. If the drug was discontinued or reduced to three or four doses each 24 hours following the disappearance of Koplik's spots, within another 48-hour period the fever, the conjunctivitis and Koplik's spots would be back.

It was our privilege to observe this picture over and over in two little volunteer girls for 30 days. These "research helpers" were my own little daughters. The measles virus was eventually destroyed in this instance by continuing 12,000 mg. by mouth each 24 hours for four days. We interpreted this result to indicate that on withdrawing the drug with the cessation of signs and symptoms, a small quantity of the virus remained, which after another incubation period produced anew the first stage of measles; when the drug was continued beyond the clearing stage the virus was destroyed in toto. No case of post-measles bronchopneumonia was seen. The "measles-cough" of measles bronchitis was over with after three or four 1000 mg. injections of "C" at 6-hour intervals. This was true even when other medications well above the calculated dose range for cough had had no effect. Whenever a patient presented a mixed-virus infection, such as receding mumps and developing measles, it was found that double the calculated dose of vitamin C was necessary to obtain the usual results.

Of mumps, 33 cases were treated with ascorbic acid. When vitamin C was given at the peak of the infection the fever was gone within 24 hours, the pain within 36 hours, the swelling in 48 to 72 hours. Two cases were complicated with orchitis. A young man of 23 years developed bilateral orchitis one Friday morning, by seven o'clock that night he was in severe pain, had a fever of 105" and was nursing testicles the size of tennis balls. Vitamin C was started at this time—1000 mg. every two hours, intravenously. The pain began to subside following the first injection and ceased in 12 hours. There was no fever after 36 hours. The patient was out of bed feeling his old self after 60 hours. He had received 25,000 mg. of "C" in this 60-hour period. An experiment involving three cousins: One, a boy of seven, had the old routine of bed rest, aspirin, and warm camphor oil applications and iodex to the swollen glands. This child had a rough time for a week. A second boy, aged 11, was allowed to develop mumps to the point of maximum swelling without any therapy, then given vitamin C, 1000 mg. intramuscularly, every two to four hours. This lad was entirely well in 48 hours. To the third patient, a girl of 9, vitamin C was given on the up curve when the swellings were 60 per cent of the expected, and the temperature recorded at 102.3°. The dose was 1000 mg. of vitamin C given intravenously every four hours. This child was well and remained so from the third day of treatment.

Further studies on virus pneumonia showed that the clinical response was better when vitamin C was given to these patients according to the dose schedule outlined for poliomyelitis. Where pneumonitis was demonstrated, the clearing of the chest film was parallel with the clinical recovery. In cases of consolidation of entire lobes the x-ray clearing lagged days behind the clinical response. In these cases 1000 mg. of "C" should be given every 12 hours for at least a week after the patient is apparently well. There was no change in the results as given in a previous paper; the patients were well in the third day of treatment.

In using vitamin C as an antibiotic no factor of toxicity need be considered. To confirm this observation 200 consecutive hospital patients were given ascorbic acid, 500 to 1000 mg. every four to six hours, for five to ten days. One volunteer received 100,000 mg. in a 12-day period. It must be remembered that 90 per cent of these patients did not have a virus infection to assist in destroying the vitamin. In no instance did examination of the blood or urine indicate any toxic reaction, and at no time were there any clinical manifestations of a reaction to the drug. When vitamin C was given by mouth one per cent of these patients vomited shortly after taking the drug. In half of these cases the vomiting was controlled by increasing the carbohydrate content of the mixture. This reaction was not interpreted as representing a toxic manifestation; rather it was thought to be due to a hypersensitive gastric mucosa. The dose was reduced from 1000 to 100 mg. in young children showing this complex; vomiting occurred as before. However, in these same patients administration of massive, frequent doses of vitamin C by needle affected a cure of the infection without causing vomiting.

From a review of the literature one can safely state that in all instances of experimental work with ascorbic acid on the virus organism the amount of virus used was beyond the range of the administered dose of this vitamin. No one would expect to relieve kidney colic with a five-grain aspirin tablet; by the same logic we cannot hope to destroy the virus organism with doses of vitamin C of 10 to 400 mg. The results which we have reported in virus diseases using vitamin C as the antibiotic may seem fantastic. These results, however, are no different from the results we see when administering the sulfa, or the mold-derived drugs against many other kinds of infections. In these latter instances we expect and usually get 48- to 72-hour cures; it is laying no claim to miracle-working then, when we say that many virus infections can be cleared within a similar time limit

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curing west nile virus with massive doses of vitamin c

http://www.boulderweekly.com/archive...overstory.html

Battle of the bite
Boulder County versus the mosquito
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by Amy Brouillette ([email protected])



With mosquito season here and several human West Nile cases already confirmed in the West, Boulder residents can expect a feverish public campaign against the virus that last year reached epidemic proportions in Colorado, the state that claimed the most reported cases in the nation.

This year Boulder County Health Department hired a marketing firm, Boulder-based Stratecom, to beef up its 2004 West Nile prevention campaign meant to raise public awareness around mosquito-prevention methods. Taking from its health reserve funds, the department spent $50,000 on the campaign, reviving last year's slogan, "One Bite. One Life Changed Forever."

It represents how health officials here and nationwide are upping the battle against the virus-spreading insect. Learning from last year's West Nile outbreak, which appeared in 46 states and was linked to 9,389 illnesses and 246 deaths, local, state and federal health departments are amplifying their prevention messages with renewed public campaigns-and, in Boulder's case, by widening the scope of who's at risk. Boulder County health officials this year are saying 97 percent of all residents are at risk of contracting the virus.

Fighting the bugs

The virus, for which there is currently no human vaccination or accepted cure, is spread to birds, humans and horses through the bite of an infected mosquito-primarily from the sturdy, efficient Culex tarsalis variety here in Colorado and the West. It attacks the central nervous system and carries symptoms ranging from imperceptible to mild to severe and can cause encephalitis, meningitis and even death.

Last year, Boulder County was the third most infected county in the state with 430 infections and seven deaths-only Larimer and Weld counties fared worse. Officials suspect the total number of unreported cases in Boulder County actually climbed into the thousands. Last year's epidemic also came with a hefty price tag, costing county residents $2.3 million in medical expenses, alternative care and lost wages, according to health department estimates.

In early May this year, city officials responded to the pending West Nile season by approving a controversial though eco-minded countywide mosquito control plan using the fog pesticide permethrin. The pesticide is only to be used on a need-to basis. Placating concerned residents, the city has likewise taken an environmentally sounder approach with a non-pesticide mosquito plan that uses larva-killing bacteria to kill mosquitoes in their infancy. This is backed by a revved-up public-education campaign urging residents to protect themselves with mosquito repellent, to avoid the outdoors at dawn and dusk and to eliminate standing water (the mosquito's breeding grounds) on their property.

Such integrative measures represent how, here and nationwide, the battle against West Nile is being fought. For their part, Boulder health officials have taken the traditional route, underscoring personal protection through the use of DEET-based (N, N-diethyl- methyl-meta-toluamide) repellant. The agency cites a July 2004 study in the New England Journal of Medicine, which concludes DEET as the superior product to soy-based repellants. A product containing 28.8 percent DEET, according the study, provides an average of five hours of protection from mosquito bites, compared to 4.7 percent DEET or 2 percent soybean oil, which provides roughly one and half to two hours of protection. The study also claims no correlation between the concentration of DEET used and risk of toxic effects.

"We see DEET as the gold standard in repellants," said Heath Harmon, Boulder County Health Department epidemiologist. "The level of protection is really based on the duration of protection, with different percentages offering different protection. With soy-based repellants, you'll get some protection, just not for the same duration."

The health department's self-protection message and strong endorsement of repellants, DEET or soy-based, was inspired by a Boulder County Health Department survey of Boulder residents, which found that although most knew West Nile was carried by mosquitoes, few did anything to protect themselves. This behavior is consistent with a nationwide survey conducted by the CDC last year that found that fewer than half of respondents used repellant. The study, presented at the Fifth National Conference on West Nile Virus in the United States in Denver last February, revealed a general misperception regarding who is at risk for contracting the illness. It even cited a general "mistrust of media hype" as a reason for the public's aversion to basic self-protection.

"Raising public awareness around repellant use has been a real challenge," said Harmon, who is heading a city-sponsored study of last year's West Nile cases. Harmon says he has spent the past year analyzing data collected from last year's bird, mosquito and horse samples in order to help county officials better prepare the public for the 2004 season. Although last year's research did not include infected humans, Harmon and his colleagues did collect that information from county health providers.

"The goal of the study is to better define risk and to determine who is at risk for developing long-lasting conditions as a result from the illness," he says.

A growing threat

Health officials began to worry when the Culex tarsalis mosquito began appearing in the city's 11 mosquito traps in record-high numbers early in June 2003, says Harmon. Last year's environmental conditions, a wet spring followed by a dry summer, were ideal for mosquito production and helped set the stage for what Harmon calls the "perfect season" in Colorado. "This species is a very efficient vector that just so happens to thrive in Colorado's warm, dry climate," he says.

Heavy snowfall in March led to more standing water in the region than usual, areas that then became breeding grounds for the mosquito. The increase in the number of virus-bearing mosquitoes was coupled with what Harmon calls the "second-year factor," a common epidemiological reaction that makes both birds and humans highly susceptible to the virus in its second year.

Harmon's study revealed something else.

"We'd always said that about 80 percent would have no symptoms at all, 20 percent would have flu-like symptoms that last three to seven days, and less than 1 percent would develop complications from what we now call Œneuro-invasive disease'," says Harmon. "While those proportions have not changed, we found instead that what those 20 percent experienced can hardly be considered Œmild'. One out of every five people who get bit by that infected mosquito is going to develop significant, debilitating, possibly long-lasting complications."

Boulder's health officials are not alone in their battle to understand the epidemiology of West Nile. Medical researchers and public health officials nationwide have scrambled to understand the virus, which has moved steadily westward with the vigorous Culex tarsalis mosquito since the first human case was reported in New York in 1999.

That same year, Bethesda, Md.-based National Institute of Health began researching vaccines in anticipation of West Nile virus becoming established here in the U.S.

"We initially underestimated its potential," says NIH program officer in virology Dr. James Meegan, who oversees NIH-funded clinical trials and vaccination studies across the country. Meegan reports several encouraging vaccine therapies underway, the most promising of which comes from a Boston-based lab. Still, he reports any vaccination, once approved, is several years away.

"The challenge with West Nile is that it is a virus, which cannot be treated with antibiotics," says Dr. Nelson Gantz, chief epidemiologist at Boulder Community Hospital. Gantz ran clinical trials last year using a drug made by Oregon-based pharmaceutical company, AVI BioPharma Inc. After success in treating penguins in the Milwaukee Zoo, the drug was tested on its first human subjects in Boulder with positive results, says Gantz. Although last season's trials have ended, he is currently lobbying the FDA for funding in order to continue the trials this year.

A mega-dose of relief

Meanwhile, as mainstream medicos grapple with funding and FDA-approvals for the perfect West Nile pill, those suffering from prolonged effects of the illness have sought respite in medical alternatives. Boulder resident Jack Butler, 68, contracted West Nile last summer while in his backyard. After a week of typical symptoms-persistent headaches, low-grade fever, confusion-Butler went to Boulder Community and tested positive for West Nile.

Rather than accept the conventional approach that addresses the illness symptomatically, chasing the illness around the body with localized treatment, Butler came across information on the effectiveness of mega-doses of vitamin C, administered intravenously, in treating a variety of viral infections. He and other West Nile patients underwent the treatment with Denver-based medical researcher and IV-C-proponent Dr. Thomas E. Levy, and today both claim having no symptoms of the virus. In Butler's case, one mega-dose of IV-C knocked out the virus in 30 hours.

The other patient, Boulder resident John Howard, 55, had chronic, prolonged effects six months after contracting the virus last July. After undergoing three consecutive sessions of IV-C, Howard claims to have no remaining symptoms. "It's almost like a miracle," he says.

A conventionally trained doctor, Dr. Levy has spent the last decade researching and conducting clinical trials using mega-doses-50 to 150 grams at a time-of vitamin C to treat infectious diseases such as viral hepatitis, viral pneumonia, influenza and Rocky Mountain Spotted Fever. His most recent book, Vitamin C, Infectious Diseases and Toxins, documents his findings and details the larger history of IV-C treatment over the past 80 years.

According to Levy, vitamin C in large doses has cured virtually every acute virus that he has treated. His research, however, makes a distinction between IV-C's success in treating "acute" versus "chronic" illness. "We've cured acute hepatitis with IV-C, for instance, but we haven't cured chronic hepatitis," says Levy. He says the evidence suggests that the effectiveness of IV-C treatment is predicated on whether or not you can get a high-enough dosage of the vitamin within the direct proximity of the virus.

"While I will not announce this is a cure for West Nile, I can say that the two cases I treated with IV-C have been successful," says Levy. He and Butler have collaborated to fund a study on a handful IV-C and West Nile patients this season and are currently looking for viable subjects.

While Butler himself has become a strong proponent of IV-C, he harbors no delusions about what he is up against in its gaining acceptance in the wider, mainstream medical community. "There's just no way a big drug company is going to spend a significant amount of money researching the benefits of vitamin C. In fact it would be a big embarrassment to them and a great boon to natural therapy if the word got out that vitamin C in mega-doses can help cure West Nile," he says.

Nor is it likely the FDA will endorse such therapy anytime soon. In the meantime, health officials maintain the public's primary means of combating the virus is through mosquito control and self-protection. Boulder County health officials say a key factor in whether or not this season will be a replay of the last will be measured in the community's response.

"We're putting out the message that it's now kind of a fact of life, that regardless of all ages, all of us are at risk, which is why we all have to protect ourselves-in addition to whatever mosquito control is in place," says Harmon.

For information on West Nile virus, visit www.bouldercountymosquito.net and www.cdc.gov. For information on Dr. Levy's clinical trials, call 303-926-1111.

Respond: [email protected]

[Kärnfysikern]

http://www.orthomed.com/auto.htm


The Ascorbate Effect in Infectious and Autoimmune Diseases
Robert F. Cathcart, M.D.
127 Second Street, #4
Los Altos, CA 94022
650-949-2822
http://www.orthomed.com

The vitamin C effects are all the usual effects of the usual small doses of vitamin C and also the effects of the moderate and usual high doses of the vitamin C. The ascorbate effect is where massive doses of vitamin C are used where we are mostly throwing away the vitamin C for the electrons carried. With massive amounts of ascorbate it is possible to neutralize the massive amounts of free radicals generated mostly by the damage to mitochondria of infectious diseases, allergies, and injuries. Under most conditions the electrons carried by free radical scavengers come from the metabolism of glucose in the mitochondria. The amount of electrons from this source, when the mitochondria are not damaged, are sufficient when we are well to neutralize the free radicals of living. However, when we are sick and especially where the mitochondria are damaged, the free radicals overwhelm the mitochondrial ability to make electrons available. In these cases vitamin C in massive amounts can be the source of the necessary numbers of electrons to eliminate most of the free radicals of diseases. The ordinary doses of vitamin C, vitamin E, beta carotene, etc. cannot suffice. Any inflammation is evidence that the free radicals have not been adequately eliminated.
In 1969, I discovered that the amount of ascorbic acid tolerated orally without loosening of stools (a benign diarrhea) was somewhat proportional to the free radical toxicity of the condition being treated. The sicker a person was, the more ascorbic acid they would tolerate orally without it causing diarrhea. In a person with an otherwise normal GI tract when they were well, would tolerate 5 to 15 grams of ascorbic acid orally in divided doses without diarrhea. With a mild cold 30 to 60 grams; with a bad cold, 100 grams; with a flu, 150 grams; and with mononucleosis, viral pneumonia, etc. 200 grams or more of ascorbic acid would be tolerated orally without diarrhea. The process of finding what dose will cause diarrhea and will eliminate the acute symptoms, I call titrating to bowel tolerance.
When at the peak of the cold it is possible to take 100 grams of ascorbate in divided doses in 24 hours, I call it a 100 Gram Cold.
Sodium ascorbate intravenously never causes diarrhea in any dose. The diarrhea of ascorbic acid taken orally is caused by a hypertonic situation in the rectum. Intravenous sodium ascorbate actually increases bowel tolerance to ascorbic acid orally if administered at the same time.

The ascorbate effect is a threshold effect. Symptoms are usually neutralized when a dose of about 90% or more of bowel tolerance is reached with oral ascorbic acid. Intravenous sodium ascorbate is about 2 ½ times more powerful than ascorbic acid by mouth and since for all practical purposes huge doses of sodium ascorbate are non toxic, whatever dose necessary to eliminate free radical driven symptoms should be given.

The mathematical formulas that describe redox potential involve logarithms. Logarithms go low, low, low and then rapidly go high. The ascorbate effect acts at a threshold dose as would be anticipated from the logarithms in the formula when a reducing redox potential is forced into the oxidized tissues involved in the disease.

Example or the Common Cold
Most people have had the experience of feeling that they are catching a cold one evening but then wake up the next morning all well. What has happened here is that either antibodies from a previous cold wipe out the virus, or the white cells in the nose and throat destroy the virus by phagocytosis. These white cells need a little vitamin
C to perform phagocytosis. If the virus damages enough mitochondria in the nose and throat to produce enough free radicals to destroy all the vitamin C then the white cells shut down and that is when you wake up knowing you will be sick for a week or so. A condition of acute induced scurvy exists in the nose and throat.

Small doses of vitamin C taken as a maintenance dose will prevent a certain percentage of colds because this acute induced scurvy is harder to induce.; Once the free radical cascade is induced in the nose and throat small and moderate doses of vitamin will not cure the cold. However, moderate doses will prevent the spread of the acute induced scurvy into the sinuses, ears, and bronchial tubes so complications will be prevented. It is interesting to note than moderate doses by reducing the free radicals systemically will slow down the production of new antibodies; therefore, the basic, uncomplicated mild disease, unsick condition, will last a little longer than an uncomplicated untreated cold.

However, if massive doses, (usually bowel tolerance doses of ascorbic acid will suffice, but not always, sometimes intravenous sodium ascorbate is necessary) are driven into the nose and throat sufficient to neutralize the free radicals and eliminate the acute induced scurvy in the nose and throat, the white cells come out fighting mad and destroy the virus.

Humeral Immunity Reduced by Massive Amounts of Ascorbate
Massive doses of ascorbate augment cellular immunity while reducing humeral immunity. Clinically, the affinity of antibodies for their antigen is augmented by free radicals or an oxidative redox potential. I hypothesize that the single disulfide bond that holds the light chain of the antibody to the heavy chain is strengthened in an oxidative redox potential. Single disulfide bonds similarly hold all the other receptor sites of the immune system together.

I further hypothesize that the immune system receptor sites are under some normal stress and that under a reducing redox potential there is more of a tendency for the disulfide bond to break into two sulfhydryls which incapacitates the antigen bonding site. This would be a simple, neat mechanism whereby the humeral immune system would be turned off when there was no injury to the body. Any injury to cells would damage mitochondria, produce free radicals, induce an oxidative redox potential and turn on the immune system. While it appears from all the scientific work done on the immune system, its turn on is more complicated than this hypothesis, this hypothesis would explain many of the clinical effects of massive doses of ascorbate.

This hypothesis would explain why symptoms of hay fever, asthma and anaphylaxis are blocked or ameliorated by massive doses of ascorbate. It is pointed out that when a person is given penicillin or other antibiotics, they are sick and have oxidative redox potential in various parts of the body. This oxidative redox potential turns on the antibodies and the penicillin, or etc., can be recognized as a foreign body. In my experience, massive doses of ascorbate given along with penicillin prevent the anaphylactic and other allergic reactions to penicillin.

Ascorbate Treatment of Viral Hepatitis

In my experience acute viral hepatitis, A, B, C, non AB, etc., are all cured by massive amounts of ascorbate given over a few days intravenously. Chronic viral hepatitis is a different story. It is such a different story that something other that just a continuing viral infection must be going on. I think it is possible that chronic liver damage releases chemicals from the interior of the liver cells that cause an autoimmune like situation to be turned on. Chronic hepatitis, like that diagnosed as chronic hepatitis C, can be vastly ameliorated by continuing high doses of ascorbic acid by mouth, alpha lipoic acid (thank you Bert Berkson), selenium, vitamin E, silymarin, and strict restriction of sugar.



Chronic Fatigue Syndrome
I practiced medicine in Incline Village, Nevada between 1970 and 1980. There I saw many mononucleosis and bad flu cases. All responded to massive doses of ascorbate. I never saw an acute viral disease develop into chronic fatigue. Shortly after I left Incline, the chronic fatigue syndrome was identified by Dr Paul Cheney in 1983. A friend, the dentist in Incline, told me that none of my old massive vitamin C takers got chronic fatigue syndrome. I admit that this observation is not hard science but it is interesting.

Chronic fatigue syndrome is ameliorated by continuing bowel tolerance doses of ascorbic acid but these patients must be worked up for candida, parasites, food and chemical sensitivities, hypothyroidism, T4 resistence, etc., and treated appropriately. The nutritional program should include no sugar, low carbohydrates, elimination of all foods they are allergic to, chemicals, zinc, manganese, chromium, selenium, cod liver oil, vitamin E, multiple Bs, sometimes IM B12, folic acid and multiple Bs, along with the massive doses of C.

Ebola and Other Hemorrhagic Fevers, Nipah Virus and Etc.

All of these diseases produce massive amounts of free radicals. These hemorrhagic fevers are examples of probably 500 gram diseases. These diseases are so toxic, produce so many free radicals, that they rapidly produce not only a localized acute induced scurvy but a systemic induced scurvy. Shortly. collagen fibers begin to break down and bleeding is induced throughout the body. These cases must be treated with massive amount of sodium ascorbate intravenously immediately at the beginning of the disease. The rate of administration should be rapidly increased until the fever and other acute symptoms are diminished. My guess at a starting dose would be at a rate of at least 240 grams of sodium ascorbate per 24 hours. Do not be cheap. Give them vitamin E, B vitamins, zinc, manganese, chromium, selenium, EPA, DHA, etc. I have never treated a hemorrhagic fever case.

SARS is just another flu virus, possibly more toxic than most flues so give them intravenous sodium ascorbate. Probably 120 grams of sodium ascorbate intravenously per 24 hours would do it but give more according to the symptoms. I have treated at lease a thousand cases of flu and never so much as hospitalize one case.

Distemper and Kennel Fever

Although dogs are ascorbate producing animals, it is possible that a very toxic disease will overcome their ability to produce ascorbate. Wendell Belfield, DVM, of San Jose, CA has been curing dogs of distemper and kennel fever for 20 years with massive doses of sodium ascorbate intravenously. The dog just needs to be helped out for a few days with the intravenous and then he takes over himself.

Poliomyelitis

The first physician who used massive amounts of sodium ascorbate intravenously on serious viral diseases was Fred Klenner, M.D. of Reidsville, North Carolina. He published curing 60 cases of polio out of 60 cases with intravenous C. See Southern Medicine and Surgery, July 1949, p. 209. The whole article is on my website http://www.orthomed.com/polio.htm



Bacterial Infections

Bacterial infections cause symptoms, suppress the immune system, and cause allergic reactions to antibiotics by way of free radicals. While these diseases should be treated with the appropriate antibiotics, they should also be treated with massive doses of ascorbate. Massive doses of ascorbate clinically seem to broaden the spectrum of activity of antibiotics against resistant bacteria.

Autoimmune Diseases

My experience with some autoimmune diseases, particularly lupus, is that ascorbate in massive doses is very helpful. The following is my theory as to why. This theory involves many simplifications and probably some ideas that turn out inaccurate but are a way of thinking about the problem of autoimmune diseases that explain the role of massive doses of ascorbate. It also gives the patient a theory with which to listen to their body to figure out their biochemical individuality as related to a treatment of their disease.

Any disease that has symptoms of inflammation, which are mediated by free radicals, cannot help but be benefited by eliminating those free radicals as much as possible with massive doses of ascorbate. When you use enough ascorbate, throwing away the vitamin C for the electrons carried, it is a matter of chemistry, not necessarily medicine, that the free radicals will be neutralized.

The immune system is very complex but to use the example of antibody exclusion, antibodies are made by B cells in utero and after. When a new B cell develops it takes on a random combination that determines the shape of the receptor site of the antibody it makes. These B cells try to match chemicals on the surface of cells. When an immature B cell matches something it dies. When a mature B cell matches something, it multiplies and produces antibodies of that shape. This is called antibody exclusion and is one of the reasons why antibodies do not ordinarily attack a person’s own cells. When the person is 100% well, their antibodies will not attack the person’s own cells. However, when a person is sick, making many free radicals, these free radicals increase the affinity of the antibodies for their antigen and may cause the antibodies to fit some shape which is not a perfect fit but a close fit.

One of the purposes of antibodies is to mop up dead or diseased cells. Remember that the antibodies and the B cells making them are extracellular and only have tried to fit shapes on the surfaces of cells. Antibodies could fit some of the chemicals in the interior of cells. Therefore, when a cell leaks, for whatever reason, certain chemicals from the interior of cells, certain antibodies may attack that cell.

The other thing is that certain injuries like from chemicals, etc., may alter the shape of chemicals on the surfaces of cells. This, especially in the oxidative redox potential of the injured area, may cause the cross reaction of antibodies on these changed cells.


So, now an infection, allergy, injury, chemical reaction, etc.;, may cause damage to cells and cause antibodies to attack. For example, suppose the person has a condition, like candida, EBV, HHV6, and various other stresses, that results in the release of lots of free radicals, These free radical up regulate the immune system. It is obvious that massive doses of ascorbate at this point may down regulate the immune system by eliminating free radicals in such a way as that the following may be prevented.



First step

The person may have a hidden or not so hidden allergy to something like milk. The immune system may then produce antibodies to milk that are similar in shape to the chemicals on the surfaces of the synovial lining of joints. The shape would not be exact because antibody exclusion would have prevented the formation of B cells making that shaped antibody. However, if the shape is close enough, with the increased affinity of antibodies in this oxidative redox potential situation, the antibodies will attack the synovial lining of the joints. Maybe some previous injury to the joint or some stress increases the oxidative redox potential in a particular joint and that joint becomes inflamed first.

At this early point, in this example, an absolutely milk free diet may stop all this. Massive doses of ascorbate would obviously help by reducing the oxidative redox potential. I saw a patient 3 months ago who had a diagnosis of rheumatoid arthritis by a local immunologist 10 years ago. She, on her own, discovered when she ate no red meat or milk products that the arthritis went away. She has been in total remission for 10 years. The immunologists I know were not interested in discussing the case.



Second step

With this injury to the synovial cells, they start leaking chemicals from their interior to the outside where antibodies can match them. In the possible case being discussed, the autoimmune reaction may take on a life of its own and perpetuate itself even though the person stops any milk. Antibodies build up in numbers and their affinity increases because of the increasing oxidative redox potential. Then, if other joints have not been involved before, they may become involved now because maybe some of the chemicals from the interior of the cells are on the surface in minute amounts but never before enough to cause a noticeable reaction. Now with the increasing numbers of antibodies and the increased oxidative redox potential, more joints become involved.

At this point, the case is not so easy to put into remission but it may be that massive doses of ascorbate, maybe even intravenously, plus eliminating the original problem (in this case milk) may throw the person into remission.



Other Problems
I find that most of the time other problems such as candida, and other food and chemical sensitivities, and leaky gut frequently get involved. All of these have to be treated. Antiyeast programs, no sugar, low carbohydrate diet, elimination of all things the patient is sensitive to, support with large amount of vitamins, minerals, essential fats, and amino acids are necessary. With Sjögren’s syndrome use in addition primrose oil. Bio-identical hormones, especially progesterone, can be helpful in osteoporosis.

I want to make special mention of nightshades (tomatoes, potatoes, egg plant, red, green, and yellow peppers, paprika and tobacco). Nightshades should be eliminated in everyone who has osteoarthritis of the fingers but they can be involved in other arthritis also. There is a relatively common genetic weakness in the ability to digest a toxin within the nightshades especially manifesting itself as one ages. If there is a genetic tendency to get lupus or rheumatoid arthritis, these diseases can be triggered by nightshades. I have seen this several times in lupus patients. The immunologists I know are not interested in this.

If standard medical treatments are used such as prednisone, methyltrexate, etc., massive doses of ascorbate plus other nutrients may augment their effects and reduce side reactions. It never hurts with any disease to eliminate as many of the free radicals as possible and reduce the oxidative redox potential.

Nightshades (Solanaceae species) are:



1. potato, the white potato

In some baby foods, potato starch or potato flour in

some breads, doughnuts, biscuits, candies, cookies and

in soups. Sweet potatoes are ok. Yams are risky.



2. tomato

husk tomato, or ground cherry tomato, cherry, yellow,

and plum tomatoes, European bitter sweet, tree tomato,

tomatillo, strawberry tomato.



3. green pepper

tobasco pepper, garden pepper, cayenne, cherry, red

cluster, hot, bell, sweet, pimiento, Chili, long and

red peppers.

(Black or condiment pepper is OK because it is not a

nightshade.)



4. eggplant



5. Misc., garden huckleberry, Morelle, wonderberry and

sunberry, pepino, Cape gooseberry.



6. Tobacco, belladonna, atropine and scopolamine.



Childers NF, Russo GM. The Nightshades and Health. Horticultural

Publications, Sumerset Press, Somerville, N.J., 1977. I think this

book is out of print but you might find it on an old book search

site on the internet.

MEDICAL PAPERS PUBLISHED RELATED TO VITAMIN C



1. Cathcart RF. Clinical trial of vitamin C. Letter to the Editor, Medical Tribune, June 25, 1975.

2. Cathcart RF. The method of determining proper doses of vitamin C for the treatment of diseases by titrating to bowel tolerance. The Australian Nurses Journal 9(4):9‑13, Mar 1980.

3. Cathcart RF. The method of determining proper doses of vitamin C for the treatment of disease by titrating to bowel tolerance. J Orthomolecular Psychiatry 10:125‑132, 1981.

4. Cathcart RF. Vitamin C: titrating to bowel tolerance, an* ascorbemia, and acute induced scurvy. Medical Hypotheses 7:1359‑1376, 1981.

5. Cathcart RF. C‑vitaminbehandling till tarmintolerans vid infektioner och allergi. Biologisk Medicin 3:6‑8, 1983.
6. Cathcart RF. Vitamin C: titrating to bowel tolerance, anascorbemia, and acute induced scurvy. Let's Live (Japan) 16:9, Nov 1983.
7. Cathcart, R.F. Vitamin C: the nontoxic, nonrate-limited, antioxidant free radical scavenger. Medical Hypotheses, 18:61-77, 1985.
8. Cathcart, R.F. Vitamin C in the treatment of acquired immune deficiency syndrome (AIDS). Medical Hypotheses, 14(4):423-433, Aug 1984.
9. Cathcart, R.F. The vitamin C treatment of allergy and the normally unprimed state of antibodies. Medical Hypotheses, 21(3):307-321, Nov 1986.
10. Cathcart, R.F. The Three Faces of Vitamin C. J. Orthomolecular Med. 7:4;197-200, 1993.

[ire]

Interesting indeed. Thanks for posting this johan. Let us know your feedback on results.

[Kärnfysikern]

I will. just drank 2grams lol and another 2 will follow in my post workout shake and had 2 grams when waking upp. Im glad I found a place where they sell vitamin c cheapluy

[Kärnfysikern]

the big question is if the creator of www.orthomed.com is a nutjob or not. The page doesnt realy make one think he is serious. He sounds like the typical conspiracy theorist. I would like to get the polio thing confirmed somehow but dont know how ??

[***xxx***]

well a pig is a pig johan, we re pretty similar to the pig but not the same. and non natural vitamine c is know to produce arteriosclerosis. let s see if I can find those articles...

[63190]

2 grams and then 2 grams after your workout? Dude, maybe you just need to slack off for a week and rest to recover. :headscratch: Just my two cents, Johan.

[ire]

Ive never read that can cause arteriosclerosis, interesting and at what dose. Is it true that vitamin C is the only vitamin repeatedly proven to increase the human lifespan when taken in doses that exceed dietary levels?

[63190]

Is it true that vitamin C is the only vitamin repeatedly proven to increase the human lifespan when taken in doses that exceed dietary levels?

It is an antioxidant. But I just thought that meant it prevented rust. j/k

[Jantzen4k]

interesting johan


is there a cliff's notes version of your redic long posts???

[Prime]

I know there is a downside to taking to much vit C ill see i if i can find out what it was.

[Kärnfysikern]

2 grams and then 2 grams after your workout? Dude, maybe you just need to slack off for a week and rest to recover. :headscratch: Just my two cents, Johan.

well even the DOC said I can workout or do whatever I want it wont hinder me recovering from the disease.

[Kärnfysikern]

I know there is a downside to taking to much vit C ill see i if i can find out what it was.

it can cause copper depletion and raise iron levels to high. But its with longtime high levels. These articels are about short duration MEGA dosages of vitamin c. Read the articles I posted its a LONG read but very interesting.

[perfectbeast2001]

I know there is a downside to taking to much vit C ill see i if i can find out what it was.

you get the squirts. Good luck running to the toilet mate!!

[Kärnfysikern]

yeah that is true. But if you read the articles you would se that depending on the disease the tollerance to vitamin c increases ALOT. The worse the disease are the more vitamin c can be used without dihareea. As much as 200grams for polio patiens.

[Kärnfysikern]

well a pig is a pig johan, we re pretty similar to the pig but not the same. and non natural vitamine c is know to produce arteriosclerosis. let s see if I can find those articles...

the fact still remains that every animal except monkys and humans have a high production of vitamin c. To assume we only need 1/20 of what a animal need does sound far fetched to me

Hope you can find the article. What is arteriosclerosis btw?

[Kärnfysikern]

from one of the articles

In using vitamin C as an antibiotic no factor of toxicity need be considered. To confirm this observation 200 consecutive hospital patients were given ascorbic acid, 500 to 1000 mg. every four to six hours, for five to ten days. One volunteer received 100,000 mg. in a 12-day period. It must be remembered that 90 per cent of these patients did not have a virus infection to assist in destroying the vitamin. In no instance did examination of the blood or urine indicate any toxic reaction, and at no time were there any clinical manifestations of a reaction to the drug. When vitamin C was given by mouth one per cent of these patients vomited shortly after taking the drug. In half of these cases the vomiting was controlled by increasing the carbohydrate content of the mixture. This reaction was not interpreted as representing a toxic manifestation; rather it was thought to be due to a hypersensitive gastric mucosa. The dose was reduced from 1000 to 100 mg. in young children showing this complex; vomiting occurred as before. However, in these same patients administration of massive, frequent doses of vitamin C by needle affected a cure of the infection without causing vomiting.

[*Narkissos*]

interesting....now it begs to query if the creator of said site is a nutjob..or a visionary

[IronFreakX]

and those animals are not exposed to smoke,stress bad food chemicals etc....
im not a doctor or nething but i think higher doses are required megadoses for a shorttime sound good to me

p.s ne1 one gonna dig up articles to prove me wrong etc.... go ahead im not gonna debate this and not gonna change my mind

johan thnx for the read bro

[Kärnfysikern]

interesting....now it begs to query if the creator of said site is a nutjob..or a visionary

Yeah confirming that polio study and finding other doctors that aggre with him is a must. Gonna dig around.

[HammerCurler]

I just had my last final exam today, i'm not about to read all of that aha

[Kärnfysikern]

here is the explaination to why more vitamin c(ascorbate) can be tolerated in sic individuals

In many cases the amount of ascorbic acid requisite for this function can be taken orally. Diarrhea (really just a softening of the stools) is the usual limiting factor. This is because as ascorbate destroys free radicals, the free radicals destroy ascorbate. And, of the ascorbate, what does not reach the rectum, does not cause diarrhea. This process is why the sicker you are, the more ascorbic acid will be tolerated orally without it producing this diarrhea. I have named this process of determining the effective dose of oral ascorbic acid titrating to bowel tolerance.

[Kärnfysikern]

lol here is some reading to do

http://www.seanet.com/~alexs/ascorbate/

god **** thats a wealth of information

[Kärnfysikern]

another doc that has cured polio with vitamin c

http://www.seanet.com/~alexs/ascorba...-n11-p1160.htm

[Kärnfysikern]

My "experiment" results. On thursday I crammed down 15g of vitamin c spaced out every 2-3 hours. All my symptoms of the viral tonsiliis disapered after 8 hours. On friday I keept on with 3 grams of vitamin c every 2-3 hours until 7 a clock when I went to a girls place. Around 2 at night I got back the tonsilitis symptoms. Om saturday when I got home(7 a clock in the evning) I started craming down vitamin c again and they where gone before I feel asleep and today I havent feelt anything and Im still cramming down vitamin c. So it defenetly works against the virus I wish I could afford 30 grams a day so I could get healthy in 2 days or so.

[rambo]

Well, here's the thing. I bet of that 15 grams of vit C, you may have absorbed like 1. The body can only take in so much vitamin C, or any amount before it becomes toxic or pissed out- one or the other. I think this guy is a conspiracy theorist, although I do know that vitamin cocktails can prevent the onset of a cold from continuing- there's a difference. All the journals that were cited are non-accredited, none are from worthy sources.

But if it floats your boat, have at it.

[Kärnfysikern]

rambo do you realy think the absorbation is that low. Are you sure the polio study is false? I have seen it cited on many sites and many not from nutcases like the one on orthomed.com

Where would I search if I want to find any real documentation on it`? Does pubmed have articles that old? Any kind of medical library??

[rambo]

rambo do you realy think the absorbation is that low. Are you sure the polio study is false? I have seen it cited on many sites and many not from nutcases like the one on orthomed.com

Where would I search if I want to find any real documentation on it`? Does pubmed have articles that old? Any kind of medical library??

Pubmed has articles back to '55. You can get access through your university for free. If you wanted to do background work, you can start checking on absorption rates of different vitamins as a function of time. You can do that online, or at any medical or science library.

[Kärnfysikern]

Il do some reading on that.

But wouldnt iv injections kind of mess that upp? Can absorbation rates be effected by various conditions?

[63190]

well even the DOC said I can workout or do whatever I want it wont hinder me recovering from the disease.

My docotor suck. They always tell me to rest when I'm sick.