Has anyone ever taken Anavar?? If so what was the results. All that I have been reading it shows to have the less sides and easier on the liver. How well does it cut up and can you use it with Test Cyp?? Thanks for the info guys!
Has anyone ever taken Anavar?? If so what was the results. All that I have been reading it shows to have the less sides and easier on the liver. How well does it cut up and can you use it with Test Cyp?? Thanks for the info guys!
makes u stronger
leans you out, veins popping
yes with cyp,
increases your cholesterol, sometimes x30%
kills appetite..
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Anavar is a mild oral. I ran an anavar only cycle for 6 weeks and gained 8lbs of lean muscle. Because it is 17a, I wouldnt run it longer than 8 weeks and yes it it can be ran with test cyp
whats the dosage and how many times a week do you take it?? Would 4 weeks be enough?
4 weeks is short IMO, you may see some strength gains but id run it for at least 6 weeks. I keep the dosing at 60mg ED (20mg 3x/day) anything more and the pumps are debilitating.
damn that just seems like alot. What side do you see on Anavar if any?? Also would you forsee any problems running it 6 weeks with test?
Did you guys run PCT after the Anavar?
iv run it with test before. i did test 1-12
anavar 8-12
the strength gains were unreal. but like someone else mentioned the pumps were dibilitating. i struggled to drive home after the gym my arms hurt that much at times!!! i used it at 50mg a day. in future i think it would be best run at 40mg for a longer duration - 6or7weeks
i ran it at 20mgs a day during pct for 4 weeks the went to 40mgs a day for another 3 weeks. loved it after what I read about it I was really suprised to see how well it worked! also take them through out the day not all at once.
Ummm you do realize that that is anavar is supressive to your endogenous test production and during PCT you trying to restore natural production.... so by taking 20mg then upping to 30mg your not doing anything for helping your natural test production, only inhibiting it.
I know the that 10mgs can be taken saftly during pct, and I do know it is not the best idea to raise the dose but I did. I ran it during pct to try and keep as much as possible.
Doses as low as 2.5mg have been shown to cause suppression. Anavar can not be included in a proper post cycle. It is the exact opposite of what you are trying to accomplish.
Live and learn I guess.
Effect of low dose oxandrolone and testosterone treatment on the pituitary-testicular and GH axes in boys with constitutional delay of growth and puberty.
Crowne EC, Wallace WH, Moore C, Mitchell R, Robertson WH, Holly JM, Shalet SM.
Department of Endocrinology, Christie Hospital Trust, Manchester, UK.
OBJECTIVE: To investigate the effect of low dose oxandrolone and testosterone on the pituitary-testicular and GH-IGF-I axes. DESIGN: Prospective double-blind placebo-controlled trial. PATIENTS: Sixteen boys with constitutional delay of growth and puberty (CDGP) with testicular volumes 4-6 ml were randomized to 3 months treatment: Group 1 (n = 5), daily placebo: Group 2 (n = 5), 2.5 mg oxandrolone daily or Group 3 (n = 6), 50 mg testosterone monthly intramuscular injections with assessment (growth, pubertal development and overnight hormone profiles) at 0, 3, 6 and 12 months. MAIN OUTCOME MEASURES: LH and GH profiles (15-minute samples) were analysed by peak detection (Pulsar), Fourier transformation and autocorrelation. Testosterone levels were measured hourly and insulin, SHBG, IGF-I, and IGFBP-3 levels at 0800 h. Statistical analysis was by multivariate analysis of variance for repeated measures. RESULTS: LH and testosterone parameters increased significantly with time in all 16 (LH AUC, P < 0.001; peak amplitude, P = 0.02; number of peaks, P = 0.02; testosterone AUC, P = 0.02; morning testosterone, P = 0.002). In Group 2, however, LH and testosterone parameters decreased at 3 months followed by a rebound increase at 6 and 12 months. SHBG levels were markedly reduced at 3 months (P = 0.006) and a wider range of dominant GH frequencies was present although GH AUC was not increased until 6 months, with an increase in GH pulse frequency but not amplitude. IGF-I levels were increased at both 3 and 12 months. In Group 3, pituitary-testicular suppression was not apparent, but GH levels increased with an increase in GH amplitude at 3 and 12 months. CONCLUSION: Oxandrolone transiently suppressed the pituitary-testicular axis and altered GH pulsatility. Testosterone increased GH via amplitude modulation.
here is another one. kind of long.
http://jcem.endojournals.org/cgi/content/full/84/8/2705
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