Quote Originally Posted by MMA
great post! but are you sure this isn't a typo?




i thought it was the other way around.
the way ive come to understand it, testosterone blocks the cortisol receptor and therefore cortisols action. The body responds by upping the production of cortisol and receptor sites , thinking it needs to do so. When AAS is discontinued, the receptor sites are freed up, and the high level of cortisol is free to hit the receptor, contributing to PC crash. This is what ive read, im no scientist. Maybe you were referring to AAS ability to block cortisol receptors, not reduce cortisol in the blood? Anyone can chime in here...