I vowed to never use DNP.. but I can concur with the PROP and T3.. I always have at least 400mgs at any given point flowing through my system..

See it's the fact it's like a BETA-ANTAGONIST or something of that nature that causes the reaction with one of the medications I am on.. it sucks.. I guess the double nature of the two medicines can cause a cardiac infarction (heart attack) ... so that's a negative NO-NO.. haha..

I am looking to switch meds so I will be able to use these compounds in the future.. they are all what bronchial dilators or something.. here let me post the interaction with the med...

MONITOR CLOSELY: Beta-2 adrenergic agonists can cause dose-related prolongation of the QT interval and potassium loss. Theoretically, coadministration with other agents that can prolong the QT interval may result in elevated risk of ventricular arrhythmias, including ventricular tachycardia and torsade de pointes, because of additive arrhythmogenic potential related to their effects on cardiac conduction. In general, the risk of an individual agent or a combination of agents causing ventricular arrhythmia in association with QT prolongation is largely unpredictable but may be increased by certain underlying risk factors such as congenital long QT syndrome, cardiac disease, and electrolyte disturbances (e.g., hypokalemia, hypomagnesemia). In addition, the extent of drug-induced QT prolongation is dependent on the particular drug(s) involved and dosage(s) of the drug(s). Clinically significant prolongation of QT interval and hypokalemia occur infrequently when beta-2 adrenergic agonists are inhaled at normally recommended dosages. However, these effects may be more common when the drugs are administered systemically or when recommended dosages are exceeded.

MANAGEMENT: Caution is advised if beta-2 adrenergic agonists are used in combination with other drugs that prolong the QT interval, including class IA and III antiarrhythmic agents, certain neuroleptic agents, phenothiazines, tricyclic antidepressants, quinolones, ketolide and macrolide antibiotics, and cisapride. It may be appropriate to monitor ECG and serum electrolytes during chronic systemic use or high-dose therapy. Patients should be advised to seek medical attention if they experience symptoms that could indicate the occurrence of torsades de pointes such as dizziness, palpitations, or syncope.

It's a MAJOR RED FLAG HIGH RISK INTERACTION when run through the drug interaction checker...

So that is why I haven't even attempted to use it until this med is LONG cleared my system...

T3 - Has absolutely ZERO interactions with any of the medications I am on.. except my TESTOSTERONE .. haha go figure.. this is what it says for that..

MONITOR: Androgens may induce reversible clinical hyperthyroidism in patients receiving thyroid hormone replacement therapy. The proposed mechanism is androgen-induced decrease in T4 binding globulin resulting in decreased serum T4, increased T3 uptake resin and free T4, and decreased TSH levels.

MANAGEMENT: Clinical and laboratory monitoring of thyroid function may be necessary, as may a 25% to 50% reduction in thyroid hormone dosage.


So out of all this.. what do you suggest.. get the doc to get me off the medication causing the MAJOR interaction to the albuterol... (which would be the same thing as clenbuterol I imagine, because CLEN isn't on the list of meds to check on the interaction checker)...

Ahhh I just wanna a fat burning aid.. just for my precontest cut !!