AI's and SERMs are two radically different groups of pharmaceuticals with their own individual pharmacokinetics/dynamics. Both interact with our endogenous oestrogenic compounds, but do so in very different ways. AI (aromatase inhibitor) are a class of drugs that possess inhibitory activities on the aromatase' function which includes it's role in being the catalyst for oestrogenic hormones (testosterone>estradiol:5a-androstanedione>estrone this will also aromatize any anabolic/androgenic steroid that possess a methylat
I was under the presumption that anastozole was relatively minor in it's alterations to your HDL/LDL levels and what alterations it did produce were unfavorable. Tamoxifin lowers your LDLs in a beneficial direction, but if you are concerned about your lipid numbers and TG/TC then anabolic/androgenic steroids in supraphysiological dosages isn't aiding in the journey either.
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