A couple of notes to add for your consideration:
1) There is debate about long term adverse sexual effects of finasteride. Some call it post-finasteride syndrome. Dr. Crysler, as well as another world famous TRT specialist I used to go to, are two of their proponents. The theory (mostly through clinical observation) is that finasteride crosses the blood-brain barrier and permanently affects 5AR enzyme activity in the brain. The brain, unlike many other tissues in the body, requires the conversion of T to DHT for sexual function. So, even if T levels are normal (or even high), DHT within the brain tissues is low and so is libido. I've never used finasteride, so I've not been following the issue and I don't know if they've come up with effective treatments. FYI: there was a class action lawsuit in Florida a couple years ago against the manufacturer of finasteride for this reason. I have no idea where it is in the court system.
2) Per previous notes, LH is highly pulsatile (I happened to do my Master's thesis on the subject). Your level may or may not be indicative of a Low LH and secondary hypogonadalism. Multiple tests are necessary for this diagnosis. Make absolutely sure that the blood is drawn as early as possible in the morning, as LH pulses, and overall baseline levels, are higher at night than in the day time. LH pulses tends to be secreted more after REM sleep.
3) Your T and Free T are within range, but at the lower end of the range. It could be indicative of a T secretion problem, particularly at your age. Your SHBG levels also are toward the lower end of the range, which probably accounts for Free T being in range with Total T at the lower end of the range. just an observation.
4) I don't see any E2 labs, but then again, I would expect E2 to be low since T is low (E2 is made from T as it's precursor molecule). Low E2 is definitely linked to ED. If you are taking any T enhancing supplements, make sure they do not have any E2 inhibitors or blockers, they may worsen the problem (e.g. chrysin).
5) I'm not a huge proponent of long-acting T esters like Nebido (Undecanoate ester). You end up having to inject larger volumes than T-cyp or T-eth to get the same amount of molecular T. The longer the ester side chain, the less the amount of T per mg of the drug. I don't know the exact amount for Nebido, but T-prop is 83.7% T, T-eth is 72.0% T, and T-cyp is 68.2% T-Und is 61% T. Also, though the manufacturer claims very long half lives, the actual half life (like all of the esters) varies from person to person. So going to a very long injection schedule may work for some guys, but others may not fit the mold and have a return of symptoms much sooner that the scheduled next injection. The other issue is the very large volumes of oil you have to inject with Nebido makes the injection process uncomfortable at best. I much prefer to inject small amounts (0.20 to 0.25 mL) of shorter acting esters on a more frequent schedule (every 3 days) using a 28G insulin syringe.
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