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Thread: debate on proviron for pct

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  1. #1
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    Quote Originally Posted by Mafiusu View Post
    hes got nothing
    Sounds like he needs to do more reading before giving advice?????

  2. #2
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    Quote Originally Posted by Mafiusu View Post
    hes got nothing
    What I suggested was adding 10mg dbol in the aim and posted a link to others that have done so. It worked for them, but its hardly a controlled enviroment.

    So with respect to PCT and a controlled group you are right there is nothing.

  3. #3
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    im just confused to exactly what he means by the no legal study's post.

  4. #4
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    I think he is confused as well!! Perhaps he should explain.

  5. #5
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    Not gonna say anything more than i cover this particular topic in my sticky.

  6. #6
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    Quote Originally Posted by WARMachine View Post
    Not gonna say anything more than i cover this particular topic in my sticky.
    war can you give me the link to your sticky

  7. #7
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    You can google HCG and read about the increase. I like this one:

    "As a result, his free testosterone doubled from 86 to 157."

    http://www.physioage.com/faq/SuccessStories.php


    As for Adex studies they are out there also:

    http://www.************.com/forum/an...ne-134791.html

    Study Shows That Arimidex Boosts Testosterone



    Estrogen suppression in males: metabolic effects.
    J Clin Endocrinol Metab 2000 Jul;85(7):2370-7 (ISSN: 0021-972X)
    Mauras N; O'Brien KO; Klein KO; Hayes V [email protected].

    We have shown that testosterone (T) deficiency per se is associated with
    marked catabolic effects on protein, calcium metabolism, and body
    composition in men independent of changes in gh - growth hormone (somatropin) - or insulin-like growth
    factor I production. It is not clear,,however, whether estrogens have a
    major role in whole body anabolism in males. We investigated the metabolic
    effects of selective estrogen suppression in the male using a potent
    aromatase inhibitor, Arimidex (Anastrozole). First, a dose-response study of
    12 males (mean age, 16.1 +/- 0.3 yr) was conducted, and blood withdrawn at
    baseline and after 10 days of oral Arimidex given as two different doses
    (either 0.5 or 1 mg) in random order with a 14-day washout in between. A
    sensitive estradiol (E2) assay showed an approximately 50% decrease in E2
    concentrations with either of the two doses; hence, a 1-mg dose was selected
    for other studies. Subsequently, eight males (aged 15-22 yr; four adults and
    four late pubertal) had isotopic infusions of [(13)C]leucine and
    (42)Ca/(44)Ca, indirect calorimetry, dual energy x-ray absorptiometry,
    isokinetic dynamometry, and growth factors measurements performed
    before and after 10 weeks of daily doses of Arimidex. Contrary to the effects of T
    withdrawal, there were no significant changes in body composition (body mass
    index, fat mass, and fat-free mass) after estrogen suppression or in rates
    of protein synthesis or degradation; carbohydrate, lipid, or protein
    oxidation; muscle strength; calcium kinetics; or bone growth factors
    concentrations. However, E2 concentrations decreased 48% (P = 0.006), with
    no significant change in mean and peak gh - growth hormone (somatropin) - concentrations, but with an 18%
    decrease in plasma insulin-like growth factor I concentrations. There was a
    58% increase in serum T (P = 0.0001), sex hormone-binding globulin did not
    change, whereas lh - leutenizing hormone - and FSH - follicle stimulating hormone - concentrations increased (P < 0.02, both). Serum
    bone markers, osteocalcin and bone alkaline phosphatase concentrations, and
    rates of bone calcium deposition and resorption did not change. In
    conclusion, these data suggest that in the male 1) estrogens do not
    contribute significantly to the changes in body composition and protein
    synthesis observed with changing androgen levels; 2) estrogen is a main
    regulator of the gonadal-pituitary feedback for the gonadotropin axis; and
    3) this level of aromatase inhibition does not negatively impact either
    kinetically measured rates of bone calcium turnover or indirect markers of
    bone calcium turnover, at least in the short term. Further studies will
    provide valuable information on whether timed aromatase inhibition can be
    useful in increasing the height potential of pubertal boys with profound
    growth retardation without the confounding negative effects of gonadal
    androgen suppression.

    hGH:

    I would go pull the studies but there is no point it public knowledge...

    nolv.... public knowledge it is needed for hcg estro sides...

    I didnt include IGF-1 or slin to the HRT mix but the advanced user can do that.

  8. #8
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    <<<<<

    Note that all the studies in the prior post are not on our sub group.

    Like I stated before there are studies on other sub groups, on hypogonatics, sure there are studies on older males, and there are studies on HIV/AIDES patients.

  9. #9
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    I've used Proviron successfully at 25-50mg/ED during PCT.

    It boosts labido and reduces circulating SHBG.

    As for Dbol bridging, never done it. But can 10mg thats active for 4-5 hours only, really aid in keeping ones gains post cycle? Whilst also compromising endogenous T production somewhat? Doesnt seem worth it on paper IMHO. But then, I'll probably give it a go anyway...

  10. #10
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    Correct me if i'm wrong but don't we have a PCT SECTION???

  11. #11
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    Quote Originally Posted by c-Z View Post
    Correct me if i'm wrong but don't we have a PCT SECTION???
    what happened to ab mr c-z

  12. #12
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    The only good thing that has come of this thread is the realization or acknowledgment (by some) that no theory in recreational AAS usage is 100% certifiable. It's not like some university has an entire section devoted to studies on the juicing athlete.. 99% of info we have garnered are abstracted and derived from studies with a whole set of different objectives and an entirely different set of test subjects.

    back to proviron. I have used it successfully for so many PCTs and cant imagine a PCT without it. Those of you who say "mesterolone is steroid and should be avoided cuz all steroids shut you down" are just making blanket statements based on oversimplification IMHO Unless you guyz can actually say/prove that proviron was the actual cause you did not recover from a cycle, then your point is moot.

    no comment on the dbol, never tried it so i wouldn't know

    cheers

  13. #13
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  14. #14
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    thanks swifto..that ll help bro

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