Originally Posted by
BrokenBricks
To answer your question you cant do math to know what your test level will be following administration of test. But this study im going to post should give you a rough idea. In it they gave different doses of test E to men over 20 weeks and measured their serum test. However they also administered a GnRH agonist for the purpose of supressing the patienrs natural test. This was required becuase precicely speaking your own production is *not* shut down on a cycle, at least not every cycle. It just depends on the amount you do.
Do tren an whether you start with 200ng/dl of natural test or 1000, you will be in the same boat, becuase that anabolic is so efficient at stimulating the negative feedback loop that regulates your own test production. But if a guys natural test levels are 1000ng/dl what should be clear is that his feedback loop is in equilibrium at that high level of test and will not respond to extra test in the same way as a man with 200ng/dl of natural test. If both men take a low dose of test, say 400mg/week, that amount is so far beyond what the 200ng mans feedback loop is comfortable with that he will shut down what he does make. The 400mg/week is not so far beyond the 1000ng/dl mans natural equilibrium and his body wont respond the same way. Will his natural test drop? No doubt, but if it falls by 50% he is still making more that the other guys natural. Take a different anabolic that is a potent feedback loop stimulator, or take enough test itself and anyone will be shut down. But to be precice, being shut down is a gradual process that is dependant on amount and type of exogenous androgen and not an either or type phenomenon.
Something that might help is to think about cycles where you did a mild test cycle vs the time you did a big cycle or one with tren in it. Were your balls equaly small during each? Mine arent. They maintain more of their volume on low dose cycles.
Anyway, I talk to much. Here is what you want to see.
Testosterone dose-response relationships in healthy young men.
"Testosterone increases muscle mass and strength and regulates other physiological processes, but we do not know whether testosterone effects are dose dependent and whether dose requirements for maintaining various androgen-dependent processes are similar. To determine the effects of graded doses of testosterone on body composition, muscle size, strength, power, sexual and cognitive functions, prostate-specific antigen (PSA), plasma lipids, hemoglobin, and insulin-like growth factor I (IGF-I) levels, 61 eugonadal men, 18-35 yr, were randomized to one of five groups to receive monthly injections of a long-acting gonadotropin-releasing hormone (GnRH) agonist, to suppress endogenous testosterone secretion, and weekly injections of 25, 50, 125, 300, or 600 mg of testosterone enanthate for 20 wk. Energy and protein intakes were standardized. The administration of the GnRH agonist plus graded doses of testosterone resulted in mean nadir testosterone concentrations of 253, 306, 542, 1,345, and 2,370 ng/dl at the 25-, 50-, 125-, 300-, and 600-mg doses, respectively. Fat-free mass increased dose dependently in men receiving 125, 300, or 600 mg of testosterone weekly (change +3.4, 5.2, and 7.9 kg, respectively). The changes in fat-free mass were highly dependent on testosterone dose (P = 0.0001) and correlated with log testosterone concentrations (r = 0.73, P = 0.0001). Changes in leg press strength, leg power, thigh and quadriceps muscle volumes, hemoglobin, and IGF-I were positively correlated with testosterone concentrations, whereas changes in fat mass and plasma high-density lipoprotein (HDL) cholesterol were negatively correlated. Sexual function, visual-spatial cognition and mood, and PSA levels did not change significantly at any dose. We conclude that changes in circulating testosterone concentrations, induced by GnRH agonist and testosterone administration, are associated with testosterone dose- and concentration-dependent changes in fat-free mass, muscle size, strength and power, fat mass, hemoglobin, HDL cholesterol, and IGF-I levels, in conformity with a single linear dose-response relationship. However, different androgen-dependent processes have different testosterone dose-response relationships."
Keeping in mind that those numbers were in the presence of a chemical given specifically to shut the person down completely. So the low doses would give higher total test concentration in your body in you or I becuase we are not shutting down our natural production, wheras the higher doses results are more in line with what our total would be becuase the will more effectively reduce our natural test, mimicing the conditions of this experiment.
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. I know its asking alot seeing how its been three years but if you can remember when you got your bloodwork done back in 2007 because if you got the work done in the mourning when you test levels are the highest it could result with different test levels.
