The UNANSWERED QUESTIONS Thread

[KimboHalfSlice]

1. Ejaculatory volume will decrease because endogenous LH and FSH have been shutdown too. There is no LH/FSH to stimulate the leydig cells to manufacturer testosterone and steroli cells to manufacturer sperm.

2. Short esters aromotase less than long esters do. Even at the same doses. Less aromotase = less estrogen = less water retention. The ester can actually influence aromotasation and DHT conversion, even if the parent hormone is the same. Long esters also seem to suppress gonodal function more than short esters.

OK I can understand the second one, thanks for that.

But as regards the first one... I really think that the testicles play a minimal role in the production of semen. Sure, they add sperm to semen, but that makes up only a tiny volume of semen (you can confirm this by asking guys who have had a vesectomy)

I don't see why damage to the testicles should result in decreased ejaculate volume.

If lack of LH or FSH caused something like the Cowper's Gland to deteriorate, then I could understand, but I haven't heard anything about this.

[Swifto]

OK I can understand the second one, thanks for that.

But as regards the first one... I really think that the testicles play a minimal role in the production of semen. Sure, they add sperm to semen, but that makes up only a tiny volume of semen (you can confirm this by asking guys who have had a vesectomy)

I don't see why damage to the testicles should result in decreased ejaculate volume.

If lack of LH or FSH caused something like the Cowper's Gland to deteriorate, then I could understand, but I haven't heard anything about this.

If you use HCG during some sort of testosterone based cycle, your seminal volume wont decrease. Why? Because it mimics LH. Or better, mimic FSH (as its used primarily for sperm production) and use HMG.

Arginine seems to increase seminal volume too. I'm not quite sure how as I've never looked into it. But SERMs like Tore, Tamox and Clomid have the same effect and all they do is raise ganadotropins. That would suggest ganadotropins are respnsible (they are) for seminal volume and function.

Vasectomy's have nothing to do with ganadotropins (LH, FSH, T). I dont see the complication in understanding this...

Endogenous ganadotropins are shutdown when using exogenous testosterone. Its that simple.

Not everyone will have a reduction in seminal volume, but the majority will. This is a common side effect during PCT too. LH and FSH never really shut off completely, so I assume thats very individual. It is as some AAS users bounce back faster than others.