Risks Involved with Cycling at 21?

[CsB]

Really? yea i guess being in medical school is considered bro science. but you prolly know more than me.

lol sure doc, you said it wont cause problems straight away but 10-15 years down the line? because of steroids? quite a big time frame isn't it considering other life factors that can play a more adverse role than steroids? "looks like you have no natural test, you also have cardio problems herp derp you took steroids 15 years ago right!?"

[Eazy20]

lol sure doc, you said it wont cause problems straight away but 10-15 years down the line? because of steroids? quite a big time frame isn't it considering other life factors that can play a more adverse role than steroids? "looks like you have no natural test, you also have cardio problems herp derp you took steroids 15 years ago right!?"

In terms of non-cardiac morbidity, AAS use is associated with hypogonadism, testicular atrophy, impaired spermatogenesis, baldness, acne, gynaecomastia, and psychiatric disturbance. Such drugs also have toxic effects on metabolic profile and hepatic structure and function,10 as well as potentially promoting neoplastic growth.10 Indeed, Parsinnen reported the 12 year mortality to be 12.9% among 62 male powerlifters suspected of AAS use, compared to 3.1% in a control population.16
LVH is an independent risk factor for cardiovascular mortality and (through whatever mechanism) one might anticipate an excess cardiovascular mortality among AAS users in whom LVH occurs. In addition, the recognised association of AAS use with hypertension and dislipidaemia (raised low density lipoprotein and reduced high density lipoprotein cholesterol, and raised triglycerides),10 as well as influences on coagulation and platelet aggregation,10 might increase such risk. While it is debatable whether ASS use is indeed associated with an increased risk of premature cardiovascular death, 38% of the deaths in Parssinen’s powerlifting group were attributed to “myocardial infarction”,16 while several case reports have attributed myocardial infarction in athletes to ASS abuse.
In some cases, infarction has occurred without evident coronary thrombosis or atherosclerosis, leading to the hypothesis that ASS may induce coronary vasospasm in susceptible individuals.10 Similarly there are several case reports of increased thromboembolic risk.10 In a recent postmortem series of 34 AAS abusers aged 20–45 years (comprising 12 homicides, 11 suicides, 12 “accidental” deaths, and two of indeterminate cause), 12 of the deceased showed cardiac pathology. Findings included hypertrophy (7 cases), myocardial or endocardial fibrosis (5), cardiac steatosis (1), myocardial coagulation necrosis (2), and coronary atheroma (4). Cardiac changes were adjudged to have contributed to death by poisoning in two cases.17 However mediated, such a morbid burden is likely to rise with time.