Made trip to see Dr. Crisler

[Lemonada8]

Ive never heard of the SubQ test is aromatized at a lower rate, i would actually think the oppsite due to thats where alot of adipose tissue is and that is a large source of aromatization. Very interesting.

Can someone link some info that backs up that claim?

I am also a fan of more frequent injections, according to the book i have on testosterone, the hydrolysis of the ester chain is very quick but the release of the testosterone injection depot is the main limiting factor. (Testosterone:action, deficiency, substuition. AMAZING book btw, Nieschlag really knows his stuff)
"the rate of hydrolysis again dependson the structure of the acid chain but this process is much faster than release from the injection depot"

I agree with swifto on the ED/EOD HCG usage, there are 2 LH spikes with the 2nd being just as large or larger than the first. by going ED/EOD with it, you * theoretically* would have a lower 2nd spike b/c the next admin of Hcg wouldnt allow it.
perhaps, by going ED/EOD with the HCG, the LH receptors on the testes to increase (assuming that they didnt properly develop b/c its primary), but this is a double edged sword b/c after long duration those receptors start to not be activated but following the diminished androgen production the natural feedback kicks in and more androgen is produced. *that would support the E3D use of HCG in normal males.

To the OP:
have you always had these issues? did you ever do a cycle? where increased androgens would lead to premature 'aging' of the leydig cells? Did you try gettin the injections closer together than 2 weeks, like E10days? Did you have your thyroid checked? did you get blood work values done of the common/useful things (Test, free test, SHBG, LH, FSH, TSH). Do you have testicular atrophy? how was your 'puberty'.. Normal, late, early?

[Ibreakspawns]

Ive never heard of the SubQ test is aromatized at a lower rate, i would actually think the oppsite due to thats where alot of adipose tissue is and that is a large source of aromatization. Very interesting.

Can someone link some info that backs up that claim?

I am also a fan of more frequent injections, according to the book i have on testosterone, the hydrolysis of the ester chain is very quick but the release of the testosterone injection depot is the main limiting factor. (Testosterone:action, deficiency, substuition. AMAZING book btw, Nieschlag really knows his stuff)
"the rate of hydrolysis again dependson the structure of the acid chain but this process is much faster than release from the injection depot"

I agree with swifto on the ED/EOD HCG usage, there are 2 LH spikes with the 2nd being just as large or larger than the first. by going ED/EOD with it, you * theoretically* would have a lower 2nd spike b/c the next admin of Hcg wouldnt allow it.
perhaps, by going ED/EOD with the HCG, the LH receptors on the testes to increase (assuming that they didnt properly develop b/c its primary), but this is a double edged sword b/c after long duration those receptors start to not be activated but following the diminished androgen production the natural feedback kicks in and more androgen is produced. *that would support the E3D use of HCG in normal males.

To the OP:
have you always had these issues? did you ever do a cycle? where increased androgens would lead to premature 'aging' of the leydig cells? Did you try gettin the injections closer together than 2 weeks, like E10days? Did you have your thyroid checked? did you get blood work values done of the common/useful things (Test, free test, SHBG, LH, FSH, TSH). Do you have testicular atrophy? how was your 'puberty'.. Normal, late, early?

I will answer all of these when I get home! Which is in about two hours. But, to be brief, puberty was late, I take Synthroid, never cycled.

[Ibreakspawns]

Edited