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  1. #1
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    Quote Originally Posted by GearHeaded View Post
    unfortunately I don't have an exact number . theres really no way in blood work etc. to know how much Mast it takes to blunt prolactin receptors. heck even if 300mg of Mast worked great at blunting prolactin receptors, thats still at the receptor site itself and this will not be seen on blood work (prolactin may even be elevated on blood work, but doesn't matter cause its simply not doing anything at the receptor ).
    so all I can do is guess. and my guess is 250mg per week. I've also never personally or known anyone else to take Mast 'only' for the purpose of blunting prolactin. most guys generally want that benefit, but also want the androgenic and anabolic benefits as well and will run moderate to high dosages.

    keep in mind that Masteron is DHT 'derived' and therefore is not dht itself nor can it convert to DHT.. so the prostate issues are not necessarily from masteron as a 'dht' , its possibly from the androgenic effects of the Mast or the effects that Mast has on lowering SHBG and thus raising free Test , and that more available free test is whats effecting the prostate (more test avail to convert to dht as well)
    Mast makes me have to go to the bathroom but I can’t.. Cialis eliminates this.
    Not a GH type explanation, but it works for me.


    Sent from my iPhone using Tapatalk

  2. #2
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    Quote Originally Posted by charger69 View Post
    Mast makes me have to go to the bathroom but I can’t.. Cialis eliminates this.
    Not a GH type explanation, but it works for me.


    Sent from my iPhone using Tapatalk

    Can finastride also help with the pee issue when using dht derived compounds? Luckily
    I havent had any issues since im just a young punk, but i like to educate myself

  3. #3
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    Quote Originally Posted by Octaneforce View Post
    Can finastride also help with the pee issue when using dht derived compounds? Luckily
    I havent had any issues since im just a young punk, but i like to educate myself
    the answer is NO and Yes , depending on various factors.

    the answer is no -- Dht 'derived' compounds are NO longer DHT nor can they convert to DHT in the body. Also, being they are derived by something already 5-alpha reductase enzyme reduced, they can no longer be effected by that enzyme (and thus Finasteride blocking that Enzyme will do nothing).
    Finasteride is going to essentially stop 5-alpha reductase enzyme from producing (converting) Dht. Masteron as a DHT derived steroid is already 5-alpha reductase reduced by its very nature . thus Finasteride has nothing to stop or action to blunt.

    but keep in mind that a drug that is 'derived' from something means that that drug is NO LONGER that from which it was derived. example, D-bol is derived from Testosterone, so by nature when taking D-bol it can not become nor 'be' testosterone itself in the body.
    so with masteron or any other dht derivative. it cannot become or convert to DHT in the body because thats what it was derived from.. too many guys confuse a 'dht derivative' compound with being DHT itself , when it is not and no longer can be.

    ok

    now the answer is "yes'' in this regards - Masteron will lower SHBG and make more Testosterone available . this 'extra' testosterone that is now available because of using Mast, can now also be converting into DHT by the 5-aphpa reductase enzyme (which has no effect on mast but does on Test). so by taking Finasteride you will keep this "extra' available testosterone from converting to DHT and binding to areas like the prostate.

    hope that all makes some sort of sense

  4. #4
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    Quote Originally Posted by GearHeaded View Post
    the answer is NO and Yes , depending on various factors.

    the answer is no -- Dht 'derived' compounds are NO longer DHT nor can they convert to DHT in the body. Also, being they are derived by something already 5-alpha reductase enzyme reduced, they can no longer be effected by that enzyme (and thus Finasteride blocking that Enzyme will do nothing).
    Finasteride is going to essentially stop 5-alpha reductase enzyme from producing (converting) Dht. Masteron as a DHT derived steroid is already 5-alpha reductase reduced by its very nature . thus Finasteride has nothing to stop or action to blunt.

    but keep in mind that a drug that is 'derived' from something means that that drug is NO LONGER that from which it was derived. example, D-bol is derived from Testosterone, so by nature when taking D-bol it can not become nor 'be' testosterone itself in the body.
    so with masteron or any other dht derivative. it cannot become or convert to DHT in the body because thats what it was derived from.. too many guys confuse a 'dht derivative' compound with being DHT itself , when it is not and no longer can be.

    ok

    now the answer is "yes'' in this regards - Masteron will lower SHBG and make more Testosterone available . this 'extra' testosterone that is now available because of using Mast, can now also be converting into DHT by the 5-aphpa reductase enzyme (which has no effect on mast but does on Test). so by taking Finasteride you will keep this "extra' available testosterone from converting to DHT and binding to areas like the prostate.

    hope that all makes some sort of sense
    How are you not an endocronologist?

    So basically dht derived compounds are not directly what causes dht sides. The issue arises because of the increased free test being available, and that test now has a chance to convert to dht by the alphafunka humpty dumpty 5 enzyme.
    Right?
    I feel like not many people know this in the steroid community.

  5. #5
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    Quote Originally Posted by Octaneforce View Post
    How are you not an endocronologist?

    So basically dht derived compounds are not directly what causes dht sides. The issue arises because of the increased free test being available, and that test now has a chance to convert to dht by the alphafunka humpty dumpty 5 enzyme.
    Right?
    I feel like not many people know this in the steroid community.
    He was in his last year of med school (weightlifting scholarship)

    Tested positive...

    He became bitter.
    He began his own series of human experiments in a makeshift lab that he made in the ruins of an underground fallout shelter, that had been forgotten in the forest near the university.

    Once in a while there is report of a strange muscular man, running in a bikini in the woods near the university.

    The locals have goven a name to this beastly scientist....

    GEARHEADED

  6. #6
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    Quote Originally Posted by Obs View Post
    He was in his last year of med school (weightlifting scholarship)

    Tested positive...

    He became bitter.
    He began his own series of human experiments in a makeshift lab that he made in the ruins of an underground fallout shelter, that had been forgotten in the forest near the university.

    Once in a while there is report of a strange muscular man, running in a bikini in the woods near the university.

    The locals have goven a name to this beastly scientist....

    GEARHEADED
    Thats actually a great idea for american horror story

  7. #7
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    Quote Originally Posted by Octaneforce View Post
    How are you not an endocronologist?
    well maybe I'll take the title "Steroidologist" , even though endocronologist definitely pays more

    Quote Originally Posted by Octaneforce View Post
    So basically dht derived compounds are not directly what causes dht sides. The issue arises because of the increased free test being available, and that test now has a chance to convert to dht by the alphafunka humpty dumpty 5 enzyme.
    Right?
    I feel like not many people know this in the steroid community.
    in one aspect, yes. because dht derived steroids are no longer dht and cannot covert to dht in the body. but indirectly through other mechanisms of action they can cause dht 'like' side effects.

    one thing to remember with DHT derived compounds and why its important to realize that they are NOT Dht itself, is that is how they are anabolic in muscle.
    DHT itself has no ability to be anabolic on muscle tissue. This is due to the enzyme in muscle tissue, 3-hydroxysteroid-dehydrogenase, which binds to DHT and deactivates it making it unable to activate and bind to muscle tissue whatsoever.

    so if Masteon for example acted like or was dht in the body, it could have no anabolic effect on muscle tissue. but being its been altered it can now be 100% anabolic in muscle tissue (unlike dht itself).
    think about that .. not even testosterone is 100% mg per mg anabolic in muscle tissue (because up to 20% of test gets converted to DHT). whereas mg per mg a dht derived steroid does not convert to DHT at all and is 100% able to bind and be anabolic in muscle tissue.

    just pointing that out as one big benefit to dht derived compounds. and also pointing that out as a way to show that dht derived compounds are NOT actual DHT , if they were then they couldn't be anabolic to muscle tissue.
    - I'm over emphasizing this because a lot of guys will talk about running a "dht" like winstrol, var, masteron, etc.. and use the terms dht and the compound interchangeably, when in fact they are not near the same things at all
    Last edited by GearHeaded; 11-10-2018 at 11:15 PM.

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