Test-E for only 8 weeks?

[bruary17]

Hey OP, I am considering my first cycle a little further down the line. What you are running is pretty much what I am thinking. Test E for 10 weeks, at a low dose. Are you def gonna run it at 300 mg? Or do you think you might go for 400mg/week? Just curious, I want to play it safe, but at the same time I don't want to waste my cycle by under dosing.

Also, are you planning on running an AI on cycle? What's planned for PCT? Thanks man, just curious.

Def going to stay at 300mg. I don't want any sides. And I don't want any DHT conversion. I may run deca instead of enth actually. Just want to do my HW on itm first. It aromatization rate and DHT conversion are practically non existent from what I hear. For PCT, Nolva and clomid will be on hand.

[Bonaparte]

Def going to stay at 300mg. I don't want any sides. And I don't want any DHT conversion. I may run deca instead of enth actually. Just want to do my HW on itm first. It aromatization rate and DHT conversion are practically non existent from what I hear. For PCT, Nolva and clomid will be on hand.

Well, you heard wrong. Test is test and it all converts the same amount into estrogen or DHT for any given blood concentration level. That last part is the key and the only difference between different esters of the same compound, since the ester will determine the release rate and pattern, along with the amount of actual hormone per mg of product injected (longer esters weigh more, so they contain less hormone per mg).

[bruary17]

I was talking about the Deca when refering to the DHT/aromatization.

[Swifto]

I was talking about the Deca when refering to the DHT/aromatization.

Deca only is a terrible first cycle.

Stick with what you have - 10 weeks of TE at 300mg/wk.

If you dont want DHT conversion, you may be better with a shorter ester than Enanthate. Long and short estered Testosterone's will all convert to estrogen (aromotase) to varied degrees.

If you want no conversion to DHT/E, then go with something like Tbol 40-60mg/ED for 6 weeks.

[Swifto]

Well, you heard wrong. Test is test and it all converts the same amount into estrogen or DHT for any given blood concentration level. That last part is the key and the only difference between different esters of the same compound, since the ester will determine the release rate and pattern, along with the amount of actual hormone per mg of product injected (longer esters weigh more, so they contain less hormone per mg).

Twice you have stated this. You ignored me at OLM when I stated this is NOT the case.

Longer ester's (Enanthate for example) convert to DHT and estrogen more heavily at the same dosage than short esters (Propionate for example).

Unless you have anything in the contrary?

The ester does effect the rate of conversion to DHT, estrogen, HPTA shutdown and how anabolic a specific compound is.

http://forums.steroid.com/showthread...icle-on-Esters...

I dont like Anthony, but this is good reading.

[Bonaparte]

Twice you have stated this. You ignored me at OLM when I stated this is NOT the case.

Longer ester's (Enanthate for example) convert to DHT and estrogen more heavily at the same dosage than short esters (Propionate for example).

Unless you have anything in the contrary?

The ester does effect the rate of conversion to DHT, estrogen, HPTA shutdown and how anabolic a specific compound is.

http://forums.steroid.com/showthread...icle-on-Esters...

I dont like Anthony, but this is good reading.

Actually, I must have missed that.
And I have read that article before (though it was like 2 years ago), but I dismissed it as more of Anthony Roberts' unfounded conjecture. And I really don't feel like reading all that shit again, but didn't AR claim that long esters convert more readily to estrogen, while short esters convert more readily to DHT?
And either way, what could actually account for the difference, since the hormone is inactive while still attached to an ester?

[Swifto]

Actually, I must have missed that.
And I have read that article before (though it was like 2 years ago), but I dismissed it as more of Anthony Roberts' unfounded conjecture. And I really don't feel like reading all that shit again, but didn't AR claim that long esters convert more readily to estrogen, while short esters convert more readily to DHT?
And either way, what could actually account for the difference, since the hormone is inactive while still attached to an ester?

Read the article.

[Bonaparte]

Read the article.

I actually went ahead and read it anyway a few hours ago. It was a bit different than the one I was thinking of.

But isn't it very possible that the findings in these studies he cites were skewed because of the infrequency of the shots? It seems that they are just based on what an individual shot of test will do (or multiple infrequent shots that allow for a return to baseline in between), and not a steady level like we would have in a cycle with frequent injections. I only took the time to look up the monkey study (source #2), but the shortest ester used was Enanthate, which doesn't really tell us much about Prop vs Enanthate. I'm still far from convinced, as it still just seems like a lot of conjecture, since he can't really provide a scientific basis for these findings.
Whatever, maybe I'm just being cranky and contrary because its midnight here and I have to teach a class in 8 hrs, but I'm awaiting your thoughts on the matter (and not trying to pick a fight with you that I'll surely lose) lol.

[Swifto]

I actually went ahead and read it anyway a few hours ago. It was a bit different than the one I was thinking of.

But isn't it very possible that the findings in these studies he cites were skewed because of the infrequency of the shots? It seems that they are just based on what an individual shot of test will do (or multiple infrequent shots that allow for a return to baseline in between), and not a steady level like we would have in a cycle with frequent injections. I only took the time to look up the monkey study (source #2), but the shortest ester used was Enanthate, which doesn't really tell us much about Prop vs Enanthate. I'm still far from convinced, as it still just seems like a lot of conjecture, since he can't really provide a scientific basis for these findings.
Whatever, maybe I'm just being cranky and contrary because its midnight here and I have to teach a class in 8 hrs, but I'm awaiting your thoughts on the matter (and not trying to pick a fight with you that I'll surely lose) lol.

No fighting here bro. I'm being blunt as I'm just about to put my dog down... :-(

They did compare Prop and Enanthate. I'm going to come back to this tomorrow.