Q's Log

[Quester]

TRT
Wednesday eve/Sunday morn:
Test-C 80mg, Adex .25mg, HCG 500IU,
Everything is in range and test levels are around 900.



Supplements
Injectible Glutathione: .3mL eod and .5mL the day after drinking;
Melatonin 1mg, 2 hr before bed;
Vit Pill: Vit-D 800IU, B6 10 mg, Calcium 500mg, Magnesium 80mg, Zinc 10mg, Copper 1mg, Copper 1mg, Manganese 1mg, Boron 1mg, Daily;
Probiotic Daily;
Aspirin 162 mg before bed;

[Quester]

Supplementary

[Quester]

and I don't know yet.

But, question: how do you post multiple pics , and wider rather than tall?

[Obs]

and I don't know yet.

But, question: how do you post multiple pics , and wider rather than tall?

If you use tapatalk i got no idea.
I just keep uploading directly from my computer after I have edited them

[Quester]

And, thanks to everyone!

[tarmyg]

Injectible Glutathione: .3mL eod and .5mL the day after drinking

Interesting. Would not a higher dose of NAC suffice? Curious how you decided to use this compound.

[Obs]

Interesting. Would not a higher dose of NAC suffice? Curious how you decided to use this compound.

He is cutting out the middle man.
NAC is just a precursor to glutathione.

I recently had a discussion with a gastroenterologist on the subject.

The brightest thing he said the entire time was "We use acetylcystine to treat acetaminophen overdose."

I was proud of him. Really wanted to give him a gold star.

[Quester]

I lost 6 lbs. this month with lots of room for improvement in all areas.

[Quester]

Also, the belly is staying in more from the increased tone and smaller meals. One diet goal is to learn to increase caloric content while maintaining small and healthy meals.

[Quester]

Hi Tarmyg,
Its for the reason OBS said.
The articles below are full text through PubMed
https://www.sciencedirect.com/scienc...464?via%3Dihub
"...Glutathione (GSH) is a tripeptide of glutamate, cysteine and glycine that contains an unusual γ-peptide bond between glutamate and cysteine (Fig. 1). Such a bond prevents GSH from being hydrolyzed by most peptidases. GSH is less easily oxidized than its precursors, cysteine and γ-glutamylcysteine."
https://onlinelibrary.wiley.com/doi/...1002/tox.22017
"These data suggested the tissues with low GSH concentration are highly vulnerable to MCLR toxicity and GSH was critical for the detoxification in MCLR-induced hepatotoxicity in vivo."
GSH = Glutathione and MCLR = microcystin-leucine-arginine ... (MCLR)-induced hepatotoxicity
https://www.nutritionjrnl.com/articl...123-X/fulltext
Highlights

• •
  The consumption of alcoholic beverages injures the liver through concurrent tissue oxidation and suppression of reduced glutathione synthesis, activity, and recycling.
• •
  The acute consumption of any ethanol will increase the level of hepatic oxidative stress.
• •
  Ethanol potentiates the hepatic oxidative stress–steatosis–hepatitis–fibrosis–cirrhosis–carc inoma transformative continuum.
• •
  Even an acute single episode of ethanol consumption initiates metabolic pathways that achieve ultimate expression as hepatic fibrosis and cirrhosis.
• •
  In the absence of abstinence, increasing hepatic antioxidant capacity may reduce the severity of ethanol-associated hepatic injury and its consequences.

[Obs]

Hi Tarmyg,
Its for the reason OBS said.
The articles below are full text through PubMed
https://www.sciencedirect.com/scienc...464?via%3Dihub
"...Glutathione (GSH) is a tripeptide of glutamate, cysteine and glycine that contains an unusual γ-peptide bond between glutamate and cysteine (Fig. 1). Such a bond prevents GSH from being hydrolyzed by most peptidases. GSH is less easily oxidized than its precursors, cysteine and γ-glutamylcysteine."
https://onlinelibrary.wiley.com/doi/...1002/tox.22017
"These data suggested the tissues with low GSH concentration are highly vulnerable to MCLR toxicity and GSH was critical for the detoxification in MCLR-induced hepatotoxicity in vivo."
GSH = Glutathione and MCLR = microcystin-leucine-arginine ... (MCLR)-induced hepatotoxicity
https://www.nutritionjrnl.com/articl...123-X/fulltext
Highlights

• •
  The consumption of alcoholic beverages injures the liver through concurrent tissue oxidation and suppression of reduced glutathione synthesis, activity, and recycling.
• •
  The acute consumption of any ethanol will increase the level of hepatic oxidative stress.
• •
  Ethanol potentiates the hepatic oxidative stress–steatosis–hepatitis–fibrosis–cirrhosis–carc inoma transformative continuum.
• •
  Even an acute single episode of ethanol consumption initiates metabolic pathways that achieve ultimate expression as hepatic fibrosis and cirrhosis.
• •
  In the absence of abstinence, increasing hepatic antioxidant capacity may reduce the severity of ethanol-associated hepatic injury and its consequences.

Thats a great post.
I never could explain it like that because I am dumb.

[Quester]

Thats a great post.
I never could explain it like that because I am dumb.

No your not, but I could easily fill a whole youtube video of tree cutting fails and a few sequels if I wasn't using all my brain power while simply hanging a birdhouse or shooting at squirrels.