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  1. #1
    ACE5HIGH's Avatar
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    Everyday Estrogen Control?

    I always seem to be in great shape durring PCT (I lose some strength but thats to be expected) Always look great though.
    However as soon as I start tapering off my AI's (L-dex or whatever) I rapidly seem to loose the "hardness" I once had (more then a normal amount)

    I seem to get extra fatty deposits in certain areas (around sides of my chest and nips, love handles etc..) where as my diet does not change and Im always relatively lean (9-10% bf) I only struggle with fat in these certain areas!!??

    Could this be caused from naturaly high estrogen levels if so should I seek out HRT? When im on some kind of AI the problem is almost non existent!
    I would love to run some l-dex or aromistan all the time if I could but am affraid of long term sides and the problem then getting worse...

    What can I do?
    Last edited by ACE5HIGH; 08-11-2008 at 09:33 PM.

  2. #2
    Kratos's Avatar
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    get a blood test and find out

  3. #3
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    do a longer pct next time, youre stopping while estro is still high

  4. #4
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    Quote Originally Posted by one8nine View Post
    do a longer pct next time, youre stopping while estro is still high
    if he is fully recovered but still on an AI his estrogen would be low? and then by coming off of the AI its returning estrogen to a normal level bringing along some extra water/fat that was kept off by lowered estro levels. MHO

  5. #5
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    Quote Originally Posted by peachfuzz View Post
    if he is fully recovered but still on an AI his estrogen would be low? and then by coming off of the AI its returning estrogen to a normal level bringing along some extra water/fat that was kept off by lowered estro levels. MHO
    well two parts to pct
    hpta function increase
    get rid od extra estradoils from cycle.

    even if fully recovered, he could have high estradoil levels in the blood.
    another reason i run letro on cycle and finish with hcg ..
    hpta gets a head start and low estro to begin with

  6. #6
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    Quote Originally Posted by one8nine View Post
    well two parts to pct
    hpta function increase
    get rid od extra estradoils from cycle.

    even if fully recovered, he could have high estradoil levels in the blood.
    another reason i run letro on cycle and finish with hcg ..
    hpta gets a head start and low estro to begin with
    but high estradiol levels would hinder HPTA increase. thus he would not be fully recovered.

    .....I think.

    im just thinking of it as if he never did a cycle and hopped on an AI. some bloat would go down and body composition may change. but coming back off the AI estrogen levels would return to normal and body composition would change adversly once again.

  7. #7
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    Quote Originally Posted by peachfuzz View Post
    but high estradiol levels would hinder HPTA increase. thus he would not be fully recovered.

    .....I think.

    im just thinking of it as if he never did a cycle and hopped on an AI. some bloat would go down and body composition may change. but coming back off the AI estrogen levels would return to normal and body composition would change adversly once again.
    no i dont think think estradoils play into the gnrh loop..

    what youre saying makes sense i understand, very possible
    but if im right he can do a longer pct and fix it
    if youre right, well that just sucks ha ha

  8. #8
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    phate im too tired to respond and i see you in here. post up some tid bits will you. estradiol is a huge factor in recovery.

  9. #9
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    OVERALL VIEW OF NEGATIVE FEEDBACK INHIBITION BY TESTOSTERONE




    The majority of bodybuilders, athletes and educated laypersons are at least somewhat familiar with the fundamentals of feedback inhibition of the Hypothalamic-Pituitary-Testicular-Axis by testosterone and its metabolites estradiol and DHT. The process is summarized in the figure below.







    Figure 1. Schematized notion of postulated feedback actions of testosterone in the normal male GnRH-LH-gonadal axis. In situ denotes feedback mediated by putative central (hypothalamic-pituitary) aromatization of testosterone to estradiol, rather than by peripheral blood estradiol levels. Other nonaromatizable androgens act like DHT on the hypothalamus to suppress the action of GnRH (Gonadotropin Releasing Hormone) secreting neurons as well as acting on the pituitary to suppress LH. Gonadotropes are LH/FSH secreting pituitary cells. The lower block illustrates testosterone and estradiol secreted from the testes. Adapted from Schnorr JA, Bray MJ, Veldhuis JD. J Clin Endocrinol Metab. 2001 Jun; 86(6):2600-6.







    According to the model depicted in Figure1, estradiol (E2) is produced from the aromatization of testosterone. Estradiol acts at both the pituitary and hypothalamic levels to suppress testosterone production. In the hypothalamus, testosterone is converted in situ (within the hypothalamus itself) to estradiol, suppressing the production of Gonadotropin Releasing Hormone (GnRH). GnRH normally stimulates the pituitary gland to secrete Luteinizing Hormone (LH) which in turn acts directly on the testes to stimulate testosterone production. Estradiol is also produced in situ in the pituitary gland where it acts to suppress LH synthesis. Estradiol is also produced from the aromatization of testosterone in peripheral tissues such as fat and muscle (1). It leaves these tissues and enters the circulation where it then can enter to pituitary gland to further suppress LH production in addition to the earlier described estradiol formed in situ within the pituitary. Incidentally, it was only recently recognized that skeletal muscle possesses aromatase and may contribute significantly to the pool of circulating estrogen, perhaps as much so as does adipose tissue. Ironically, the more muscular one becomes, the more estrogen the body produces.




    It is also believed that testosterone and its 5 alpha reduced metabolite DHT both act at the hypothalamic and pituitary levels to further suppress the signals for testosterone production.




    There exists an additional negative feedback mechanism on testosterone production that is seldom if ever discussed in the bodybuilding literature. Cholesterol is the basic building block for testosterone. Cholesterol enters the testicular Leydig cells and after a number of steps is ultimately converted to testosterone. As it turns out, testosterone acts to block entry of cholesterol into the Leydig cells, thus directly controlling its own production. It is this little discussed mechanism of feedback inhibition I would like to outline here.

  10. #10
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    damn its 2 45 am i dunno if you just didnt post up anything that said having leftover estradoils from cycle will reduce test production or if i was just too tired to catch it

  11. #11
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    sorry my friend. hope this helps.

    E2 is produced from the aromatization of testosterone . Estradiol acts at both the pituitary and hypothalamic levels to suppress testosterone production. In the hypothalamus, testosterone is converted in situ (within the hypothalamus itself) to estradiol, suppressing the production of Gonadotropin Releasing Hormone (GnRH).

    now off to the gym for me!

  12. #12
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    Quote Originally Posted by peachfuzz View Post
    sorry my friend. hope this helps.

    E2 is produced from the aromatization of testosterone . Estradiol acts at both the pituitary and hypothalamic levels to suppress testosterone production. In the hypothalamus, testosterone is converted in situ (within the hypothalamus itself) to estradiol, suppressing the production of Gonadotropin Releasing Hormone (GnRH).

    now off to the gym for me!
    got it i did read that i just suck at 2:45 am ish'i guess that means i should go to sleep huh

  13. #13
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    Thanks for the input from both sides!

    I may not have run a long enough PCT as suggested... Since I only really have this prob around the end of cycles...

    If this is a case of having left over estradoils from the cycle would running some nolva or Clomi be better then an AI or should I use some Aromasin ?

    Also the longer I run one of these really gonna matter in the end? wont estradoils levels return to a norm in time reguardless?

    Thanks!

  14. #14
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    Quote Originally Posted by ACE5HIGH View Post
    Thanks for the input from both sides!

    I may not have run a long enough PCT as suggested... Since I only really have this prob around the end of cycles...

    If this is a case of having left over estradoils from the cycle would running some nolva or Clomi be better then an AI or should I use some Aromasin ?

    Also the longer I run one of these really gonna matter in the end? wont estradoils levels return to a norm in time reguardless?

    Thanks!
    gyno/sides/control
    estrogen/progesterone and gyno/side effects INFO FOR NEW GUYS

    my pct
    http://forums.steroid.com/pct-post-cycle-therapy/354573-one8nines-opinion-pct-links-side-effect-control-too.html#post4111013

    ive seen people with elevated prolactin for years

  15. #15
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    Aromasin + Nolvadex for maybe another two weeks, does sound ok then?

  16. #16
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    Quote Originally Posted by ACE5HIGH View Post
    Aromasin + Nolvadex for maybe another two weeks, does sound ok then?
    try it our bro

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    aromasin is good cuz it will keep your estrogen down a long time after you stop taking it.

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