Tren Cough......WTF????

[stpete]

Yea i hear ya T-Mos. I wish i knew the answer as well and Marcus makes the most sense. But here's my issue. I only cough when i inject my quads. I taste it everytime, but never cough when i inject anywhere else. I also have shortness of breath when i hit quads, but it only lasts for maybe a minute or so. Kinda weird imo. Never felt that before with any other drug.

[Matt]

I cant believe no one asked this guy for stats..

[T-MOS]

I cant believe no one asked this guy for stats..

HAHAH

ok

5'9
330 lbs
45% bf
just started lifting last week
10 cycles
PCT? who needs that

[Matt]

HAHAH

ok

5'9
330 lbs
45% bf
just started lifting last week
10 cycles
PCT? who needs that

I love it...

[D7M]

I need to find Pheendo's take on this...hold on

[D7M]

http://forums.steroid.com/showthread.php?t=74729

It's been widely disussed of Trens fat burning properties through rises in IGF and Prostaglandins. While IGF is a fairly well known substance in the bodybuilding world today, prostaglandins are fairly unknown in terms of formation and roles in the body.
So below, a brief dicription of prostoglandins and their role in fat burning, "fina cough", and why a person going through Tren administration can experience it's fat burning effects without the dreaded "Cough"

The term prostaglandin comes from the word-Prostate. The first prostoglandins were first dicovered in semen about the mid 1930's and it was thought that prostaglandins were made from the prostate. Since this time, it has been dicovered that most prostaglandins are not even constructed in the prostate.

Prostaglandins are made by two different pathways(Cyclooxygenase and Lipoxygenase), and considering prostaglandins are a group of about 20 lipid cells, they have contrary function; responsible for stimulating as well as alleviating inflammation(Inflammation stimulation is the rapid metabolism of them expelled through the bronchials), regulate blood flow to particular organs, control ion transport across membranes, modulate synaptic transmission, induce sleep, mediate lipid release, and regulate metabolism is various tissue.

Prostaglandins are synthesized from arachidonate(Lipoxygenase which catalyze the dioxygenation of polyunsaturated fatty acids) in the cell membrane by the action of phospholipase A2. Cyclooxygenase and lipoxygenase pathways, compete with one another to form prostaglandins(as well as thromboxane or leukotriene-leukotriene being a bronchial stimulator),
In the cyclooxygenase pathway, the prostaglandins D, E and F plus thromboxane and prostacyclin are made. Thromboxanes are made in platelets and cause constriction of vascular smooth muscle and platelet aggregation
Leukotrienes are made in leukocytes and macrophages via the lipoxygenase pathway. They are potent constrictors of the bronchial airways. They are also important in inflammation and hypersensitivity reactions as they increase vascular permeability.

Being that prostaglandins from either pathway, are still fatty acids of a group, they mediate lipid release and controll tissue metabolization, so fat burning is a luxerry of either pathway of formation. It's the pathway from which they are constructed that dictates "fina cough". As prostaglandins made from the Cyclooxygenase pathway dictate muscle constriction and platlet aggregation, and the Lipoxygenase pathway dictates bronchial constriction(the main form of expulsion)


Refs:
Cackatoo Press
Columbia Encyclopedia 6th Edition
Science Daily Magazine

[TITANIUM]

Tren cough
The so called 'tren cough' or 'Fina cough' is well known amongst many tren users. Some users seem to get the cough following every injection; others never or extremely rarely will get the cough. Usually it is manifested upon injection, with a tightness in the chest, and a metallic taste in the back of the mouth, followed by an uncontrollable violent cough which can be quite frightening, as anyone who has experienced it will tell you, whether it's for the first time or not. There have been some very elaborate theories about the reasons for getting the cough from trenbolone , some of which have had mechanisms involving molecules that only trenbolone affects resulting in bronchioconstriction, etc. However, the fact remains that many users have also experienced the same cough from steroids such as equipoise and testosterone cypionate . In addition, these mechanisms that are proposed are highly unlikely to occur immediately upon injection, as that is too fast a timescale for the proposed mechanism. Thus it must be the result of something entering the blood stream and traveling to the lungs for the cough to be manifested that quickly.

This leads us onto the next theory suggested by many which is that trenbolone is produced by many UGLs, and as such is made with higher percentages of Benzyl Alcohol (BA) than pharma grade products are, and it is the alcohol that is causing the reaction. The only problem with this theory is that trenbolone is made by most UGLs with the same BA percentages as things such as testosterone propionate , and nandrolone decanoate. If it was purely the BA concentration, than we would expect to see the cough with these other products as well, which we do not. Thus, as we have eliminated the oil, solvents and carriers, it leaves us with the Trenbolone product itself as the potential culprit.

One thing that you notice about trenbolone is that it is often a golden-brown / rust colour when in oil solution. If the hormone powder is refined to greater than 99.5% purity or so, then the colour of trenbolone in solution actually gives a very light golden colour, much like other testosterone products; however, refining the hormone to this level of purity is extremely difficult. This is why there is colour variation from batch to batch with different underground labs; something as small as 0.1% purity can affect the colour of the final product.

As mentioned above when discussing kidney effects of trenbolone, the oxidised trenbolone is a rust colour – much like the colour seen of trenbolone in oil solution. What you also notice with steroids such as Equipoise and to a lesser degree, testosterone cypionate , is that these steroids too are hard to very highly refine and often a browny-rust colour, more so than products such as testosterone propionate , etc. It is very likely then that these oxidised particles get into the blood stream upon injection and this causes some sort of anaphylactic (allergic) reaction in the lungs as the particles react with the alveoli, perhaps. This seems to be confirmed by the fact that the darker the trenbolone is the more likely one is to get a cough (personal and general experience). The best way to try and avoid this is to firstly inject very slowly and not move the needle around after aspirating, and also mixing the trenbolone with another product such as test prop.

[Roshambo]

Does anyone know the exact reason for Tren Cough??

I mean I just about hacked up a lung today.

I don't understand what the physical function is causing it when injecting deep into muscle that would cause you to hack uncontrollably and ONLY with TREN??
Other compounds go in the exact same way but don't cause that cough.

Is there something in the chemical makeup of Tren (tren ace this time, but happens to me with Tren E as well) that causes an instant reaction to the lungs?

Anyone???????

Being that prostaglandins from either pathway, are still fatty acids of a group, they mediate lipid release and controll tissue metabolization, so fat burning is a luxerry of either pathway of formation. It's the pathway from which they are constructed that dictates "fina cough". As prostaglandins made from the Cyclooxygenase pathway dictate muscle constriction and platlet aggregation, and the Lipoxygenase pathway dictates bronchial constriction(the main form of expulsion).