**Dianaviron: The ULTIMATE Anabolic Bridge**

[-The Ross-]

KEEP 100% OF YOUR GAINS!

Do NOT discontinue anabolic steroids ABRUPTLY!

Post-cycle regimens containing Aromatase Inhibitors and SERM's are simply not enough for the SERIOUS bodybuilder to maintain his muscular gains post-cycle. Once a bodybuilders reaches a certain point of muscular development, the continued use of a mild anabolic becomes justified..

There is a HUGE DIFFERENCE between TESTICULAR inhibtion and PITUITARY inhibition!

MAINTAINING PITUITARY FUNCTION is neccessary in order for Testosterone levels to begin increasing. No matter how much Arimidex or Clomid you take, if your PITUITARY is not responsive, neither will be your testicles. Ergo, we must first restore PITUITARY function before we can restore TESICULAR function.

We MUST discontinue all PITUITARY-inhibiting compounds(Testosterone, Nanrolone, Trenbolone) at least 6 weeks before we can even THINK about getting our testosterone levels back up. During this 6 week period AFTER those compounds have been discontinued, we begin administering compounds that are NOT inhibitory to the pituitary(even though they all supress testicular function) such as Oxandrolone, Methandrostenelone, Stanzolol, Methenelone, and Masteron , and Proviron This will allow your body to remain in an anabolic state WHILE YOUR PITUITARY BEGINS TO RECOVER.

Your body MUST remain in an anabolic state while the pituitary begins to recover.

Enter "Dianaviron", the ULTIMATE anabolic bridge.

Dianabol has only a minimal influence on the HPTA, despite being even more anabolic than testosterone. One 10mg dosage will drastically increase testosterone concentrations, and will also significantly reduce CORTISOL, which is utterly important post-cycle.

Proviron will increase FREE test levels, decrease SHBG, decrease ESTROGEN, and it binds well to the A-R providing the muscles with density and hardness. Studies have shown that Proviron can be used with NO EFFECT on the HPTA*

KEEP your gains! By combining low doses of Dianabol with Proviron in a post-cycle environment, one can not only maintain all of their hard-earned gains, but continue to make muscular gains up until their next full cycle--ALL WITHOUT affecting the HPTA!

Dianaviron should be run as follows:

Dianabol: 10-20mgs ED
Proviron: 50mgs ED

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NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)

Very Androgenic /Progestenic/Estrogenic steroids (Tren , Deca , Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
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Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone , estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS


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