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Thread: May 2018 Most Improved Competition [FINISHED: Results in post #5]

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  1. #1
    guitarzan's Avatar
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    Can e2 cause elevated hr?

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    Quote Originally Posted by guitarzan View Post
    Can e2 cause elevated hr?
    You don't say? It drove my BP up to 160/100 last year and was on Test only.

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    Quote Originally Posted by hammerheart View Post
    You don't say? It drove my BP up to 160/100 last year and was on Test only.
    It was a question not a statement. FYI.

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    Quote Originally Posted by guitarzan View Post
    Can e2 cause elevated hr?
    Appears to have the opposite effect

    Estrogen (which is increased in men who have had heart attacks) is another factor which decreases the heart’s stroke volume, and estrogen is closely associated with the physiology of the free unsaturated fatty acids.”

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    Quote Originally Posted by Capebuffalo View Post
    Appears to have the opposite effect

    Estrogen (which is increased in men who have had heart attacks) is another factor which decreases the heart’s stroke volume, and estrogen is closely associated with the physiology of the free unsaturated fatty acids.”
    Ok...

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    Quote Originally Posted by Capebuffalo View Post
    Appears to have the opposite effect

    Estrogen (which is increased in men who have had heart attacks) is another factor which decreases the heart’s stroke volume, and estrogen is closely associated with the physiology of the free unsaturated fatty acids.”
    If the volume of blood pumped is lower, the hr (heart rate) will increase to compensate.

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    Quote Originally Posted by Mr.BB View Post
    If the volume of blood pumped is lower, the hr (heart rate) will increase to compensate.
    The whole sentence was quite of an hyperbole though, rare read even in places like eroids. Men with heart attacks. As far as I reckon. I never had an heart attack.

    Guitarzan, you ever had one?

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    Quote Originally Posted by hammerheart View Post
    The whole sentence was quite of an hyperbole though, rare read even in places like eroids. Men with heart attacks. As far as I reckon. I never had an heart attack.

    Guitarzan, you ever had one?
    I have not, but my hr is elevated. I'm still on t3, but a low dose, it keeps my blood sugar from dropping when I eat. Hoping the tren and other aas's have something to do with it, should be out of my system soon

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    Quote Originally Posted by guitarzan View Post
    I have not, but my hr is elevated. I'm still on t3, but a low dose, it keeps my blood sugar from dropping when I eat. Hoping the tren and other aas's have something to do with it, should be out of my system soon
    Honestly I don't know about your situation but I monitor my vitals closely and my resting HR is usually 60-65bpm, hence is definitely not normal for me to read above that, I can also tell from water retention and distribution thereof, how I respond to coffee, etc I'm pretty acquainted my myself and the way I respond to the hormones.

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    Quote Originally Posted by hammerheart View Post
    The whole sentence was quite of an hyperbole though, rare read even in places like eroids. Men with heart attacks. As far as I reckon. I never had an heart attack.

    Guitarzan, you ever had one?
    Bit confusing to me to. Another question is did the increased estrogen was caused by the heart attacks, or the estrogen was already there promoting the heart attack?

    ... Am I making any sense? Already took my zolpidem.

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    Quote Originally Posted by Mr.BB View Post
    Bit confusing to me to. Another question is did the increased estrogen was caused by the heart attacks, or the estrogen was already there promoting the heart attack?

    ... Am I making any sense? Already took my zolpidem.
    As simply as correlation doesn't imply causation... good night.

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    Quote Originally Posted by Mr.BB View Post
    If the volume of blood pumped is lower, the hr (heart rate) will increase to compensate.
    Stroke volume (SV) is the amount of blood pumped out of the heart (left ventricle - to the body) during each contraction measured in mL/beat (millilitres per beat). Therefore cardiac output (Q), the amount of blood leaving the heart each minute, measured in L/minute, can be calculated by multiplying stroke volume by heart rate (Q = SV x HR).

    As you can see, the cardiac output (Q) will not increase just by increasing heart rate, since the stroke volume will probably decrease to compensate.

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    Quote Originally Posted by Capebuffalo View Post
    Stroke volume (SV) is the amount of blood pumped out of the heart (left ventricle - to the body) during each contraction measured in mL/beat (millilitres per beat). Therefore cardiac output (Q), the amount of blood leaving the heart each minute, measured in L/minute, can be calculated by multiplying stroke volume by heart rate (Q = SV x HR).

    As you can see, the cardiac output (Q) will not increase just by increasing heart rate, since the stroke volume will probably decrease to compensate.
    Thats a basic pump formula lol. The question was not Q increasing, it was HR increasing (to compensate Q, to use your own words heheh).

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    Shoulders are fried, managed some negatives at the cable side raises. Couldn't rack a damn empty bar into position when I was done.

    Might of have gone heavier on the DB presses though.

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    Quote Originally Posted by hammerheart View Post
    Shoulders are fried, managed some negatives at the cable side raises. Couldn't rack a damn empty bar into position when I was done.

    Might of have gone heavier on the DB presses though.
    how did you do negatives on lateral raises? I have a hard time finding a way to do negatives for most push exercises

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    Quote Originally Posted by HoldMyBeer View Post
    how did you do negatives on lateral raises? I have a hard time finding a way to do negatives for most push exercises
    Do one arm at a time. When working right arm assist on the concentric phase by using your left arm too. I usually grab the cable at the ball where it connects to the hand grip. Hope that makes sense

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